These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
77 related articles for article (PubMed ID: 2846353)
1. Phosphoinositide breakdown and superoxide anion release in formyl-peptide-stimulated human alveolar macrophages. Comparison between quiescent and activated cells. Damon M; Vial H; Crastes de Paulet A; Godard P FEBS Lett; 1988 Nov; 239(2):169-73. PubMed ID: 2846353 [TBL] [Abstract][Full Text] [Related]
2. Calcium regulation of phosphatidyl inositol turnover in macrophage activation by formyl peptides. Holian A; Stickle DF J Cell Physiol; 1985 Apr; 123(1):39-45. PubMed ID: 2982888 [TBL] [Abstract][Full Text] [Related]
3. Leukotriene B4 stimulation of phosphatidylinositol turnover in macrophages and inhibition by pertussis toxin. Holian A FEBS Lett; 1986 May; 201(1):15-9. PubMed ID: 3011502 [TBL] [Abstract][Full Text] [Related]
4. Chemotactic peptide activation of human neutrophils and HL-60 cells. Pertussis toxin reveals correlation between inositol trisphosphate generation, calcium ion transients, and cellular activation. Krause KH; Schlegel W; Wollheim CB; Andersson T; Waldvogel FA; Lew PD J Clin Invest; 1985 Oct; 76(4):1348-54. PubMed ID: 3877077 [TBL] [Abstract][Full Text] [Related]
5. Interactions of formylmethionyl-leucyl-phenylalanine, adenosine, and phosphodiesterase inhibitors in human monocytes. Effects on superoxide release, inositol phosphates and cAMP. Elliott KR; Leonard EJ FEBS Lett; 1989 Aug; 254(1-2):94-8. PubMed ID: 2550281 [TBL] [Abstract][Full Text] [Related]
6. Inhibition of macrophage activation by isoquinolinesulfonamides, phenothiazines, and a napthalenesulfonamide. Holian A; Jordan MK; Nguyen HV; Devenyi ZJ J Cell Physiol; 1988 Oct; 137(1):45-54. PubMed ID: 2844836 [TBL] [Abstract][Full Text] [Related]
8. Enhancement of chemotactic peptide-induced activation of phosphoinositide 3-kinase by granulocyte-macrophage colony-stimulating factor and its relation to the cytokine-mediated priming of neutrophil superoxide-anion production. Kodama T; Hazeki K; Hazeki O; Okada T; Ui M Biochem J; 1999 Jan; 337 ( Pt 2)(Pt 2):201-9. PubMed ID: 9882616 [TBL] [Abstract][Full Text] [Related]
9. [Formation of cell membrane signal ATP by activated neutrophils and macrophages: connection with superoxide production and formyl peptide receptor status]. Globa AG; Zaĭtseva NV; Tepliakov VG; Karelin AA Vopr Med Khim; 1992; 38(3):13-8. PubMed ID: 1329348 [TBL] [Abstract][Full Text] [Related]
10. N-formyl-methionyl-leucyl-phenylalanine induced accumulation of inositol phosphates indicates the presence of oligopeptide chemoattractant receptors on circulating human lymphocytes. Schubert T; Müller WE FEBS Lett; 1989 Oct; 257(1):174-6. PubMed ID: 2553489 [TBL] [Abstract][Full Text] [Related]
11. Receptor-mediated O2- release by alveolar macrophages and peripheral blood monocytes from smokers and nonsmokers. Priming and triggering effects of monomeric IgG, concanavalin A, N-formyl-methionyl-leucyl-phenylalanine, phorbol myristate acetate, and cytochalasin D. Nakashima H; Ando M; Sugimoto M; Suga M; Soda K; Araki S Am Rev Respir Dis; 1987 Aug; 136(2):310-5. PubMed ID: 3039878 [TBL] [Abstract][Full Text] [Related]
12. Possible physiological role of guanosine triphosphate and inositol 1,4,5-trisphosphate in Ca2+ release in macrophages stimulated with chemotactic peptide. Kimura Y; Hirata M; Hamachi T; Koga T Biochem J; 1988 Jan; 249(2):531-6. PubMed ID: 3257693 [TBL] [Abstract][Full Text] [Related]
14. Requirement of Ca2+ for the production and degradation of inositol 1,4,5-trisphosphate in macrophages. Kukita M; Hirata M; Koga T Biochim Biophys Acta; 1986 Jan; 885(1):121-8. PubMed ID: 3002487 [TBL] [Abstract][Full Text] [Related]
15. Interleukin-one induced inositol phospholipid breakdown in murine macrophages: possible mechanism of receptor activation. Wijelath ES; Kardasz AM; Drummond R; Watson J Biochem Biophys Res Commun; 1988 Apr; 152(1):392-7. PubMed ID: 2965872 [TBL] [Abstract][Full Text] [Related]
16. Human alveolar macrophages release a factor that inhibits phagocyte function. Sibille Y; Merrill WW; Naegel GP; Care SB; Cooper JA; Reynolds HY Am J Respir Cell Mol Biol; 1989 Nov; 1(5):407-16. PubMed ID: 2561589 [TBL] [Abstract][Full Text] [Related]
17. Complete dissociation between the activation of phosphoinositide turnover and of NADPH oxidase by formyl-methionyl-leucyl-phenylalanine in human neutrophils depleted of Ca2+ and primed by subthreshold doses of phorbol 12,myristate 13,acetate. Grzeskowiak M; Della Bianca V; Cassatella MA; Rossi F Biochem Biophys Res Commun; 1986 Mar; 135(3):785-94. PubMed ID: 3008746 [TBL] [Abstract][Full Text] [Related]
18. Polyphosphoinositides are the major source of inositol phosphates in carbamoylcholine-stimulated SK-N-SH neuroblastoma cells. Fisher SK; Heacock AM; Seguin EB; Agranoff BW Mol Pharmacol; 1990 Jul; 38(1):54-63. PubMed ID: 2164631 [TBL] [Abstract][Full Text] [Related]
19. Effect of extracellular calcium on superoxide release by rat alveolar macrophages. Forman HJ; Nelson J J Appl Physiol Respir Environ Exerc Physiol; 1983 May; 54(5):1249-53. PubMed ID: 6305896 [TBL] [Abstract][Full Text] [Related]
20. Aroclor 1242 stimulates the production of inositol phosphates in polymorphonuclear neutrophils. Tithof PK; Contreras ML; Ganey PE Toxicol Appl Pharmacol; 1995 Mar; 131(1):136-43. PubMed ID: 7878669 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]