340 related articles for article (PubMed ID: 28482072)
1. Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.
Chen X; Zhong J; Dong D; Liu G; Yang P
Toxicol Sci; 2017 Aug; 158(2):275-285. PubMed ID: 28482072
[TBL] [Abstract][Full Text] [Related]
2. Silenced CHOP protects pancreatic B-cell function by targeting peroxisome proliferator-activated receptor-γ coactivator-1α through nuclear factor-κB signaling pathway in diabetes mellitus.
Hu H; Peng L; Jiang H; Shen H; Zhou P; Gao Y
J Cell Biochem; 2019 Aug; 120(8):12595-12603. PubMed ID: 30848505
[TBL] [Abstract][Full Text] [Related]
3. Superoxide dismutase 2 overexpression alleviates maternal diabetes-induced neural tube defects, restores mitochondrial function and suppresses cellular stress in diabetic embryopathy.
Zhong J; Xu C; Gabbay-Benziv R; Lin X; Yang P
Free Radic Biol Med; 2016 Jul; 96():234-44. PubMed ID: 27130031
[TBL] [Abstract][Full Text] [Related]
4. Deficiency of the oxidative stress-responsive kinase p70S6K1 restores autophagy and ameliorates neural tube defects in diabetic embryopathy.
Cao S; Shen WB; Reece EA; Yang P
Am J Obstet Gynecol; 2020 Nov; 223(5):753.e1-753.e14. PubMed ID: 32416155
[TBL] [Abstract][Full Text] [Related]
5. Peroxisome proliferator-activated receptor γ coactivator-1α (PGC-1α) overexpression alleviates endoplasmic reticulum stress after acute kidney injury.
Pan H; Hu Z; Shao Z; Ning Y
Ren Fail; 2022 Dec; 44(1):358-367. PubMed ID: 35225153
[TBL] [Abstract][Full Text] [Related]
6. Punicalagin exerts protective effect against high glucose-induced cellular stress and neural tube defects.
Zhong J; Reece EA; Yang P
Biochem Biophys Res Commun; 2015 Nov; 467(2):179-84. PubMed ID: 26453010
[TBL] [Abstract][Full Text] [Related]
7. Dominant negative FADD dissipates the proapoptotic signalosome of the unfolded protein response in diabetic embryopathy.
Wang F; Weng H; Quon MJ; Yu J; Wang JY; Hueber AO; Yang P
Am J Physiol Endocrinol Metab; 2015 Nov; 309(10):E861-73. PubMed ID: 26419589
[TBL] [Abstract][Full Text] [Related]
8. Superoxide dismutase 1 overexpression in mice abolishes maternal diabetes-induced endoplasmic reticulum stress in diabetic embryopathy.
Wang F; Reece EA; Yang P
Am J Obstet Gynecol; 2013 Oct; 209(4):345.e1-7. PubMed ID: 23791840
[TBL] [Abstract][Full Text] [Related]
9. The increased activity of a transcription factor inhibits autophagy in diabetic embryopathy.
Xu C; Chen X; Reece EA; Lu W; Yang P
Am J Obstet Gynecol; 2019 Jan; 220(1):108.e1-108.e12. PubMed ID: 30312583
[TBL] [Abstract][Full Text] [Related]
10. Endoplasmic reticulum stress-induced apoptosis contributes to articular cartilage degeneration via C/EBP homologous protein.
Uehara Y; Hirose J; Yamabe S; Okamoto N; Okada T; Oyadomari S; Mizuta H
Osteoarthritis Cartilage; 2014 Jul; 22(7):1007-17. PubMed ID: 24795271
[TBL] [Abstract][Full Text] [Related]
11. Protein kinase C-alpha suppresses autophagy and induces neural tube defects via miR-129-2 in diabetic pregnancy.
Wang F; Xu C; Reece EA; Li X; Wu Y; Harman C; Yu J; Dong D; Wang C; Yang P; Zhong J; Yang P
Nat Commun; 2017 May; 8():15182. PubMed ID: 28474670
[TBL] [Abstract][Full Text] [Related]
12. Endoplasmic reticulum stress links hepatitis C virus RNA replication to wild-type PGC-1α/liver-specific PGC-1α upregulation.
Yao W; Cai H; Li X; Li T; Hu L; Peng T
J Virol; 2014 Aug; 88(15):8361-74. PubMed ID: 24829353
[TBL] [Abstract][Full Text] [Related]
13. Endoplasmic reticulum stress contributes to NMDA-induced pancreatic β-cell dysfunction in a CHOP-dependent manner.
Huang XT; Liu W; Zhou Y; Sun M; Sun CC; Zhang CY; Tang SY
Life Sci; 2019 Sep; 232():116612. PubMed ID: 31260687
[TBL] [Abstract][Full Text] [Related]
14. The Nrf2 Activator Vinylsulfone Reduces High Glucose-Induced Neural Tube Defects by Suppressing Cellular Stress and Apoptosis.
Dong D; Reece EA; Yang P
Reprod Sci; 2016 Aug; 23(8):993-1000. PubMed ID: 26802109
[TBL] [Abstract][Full Text] [Related]
15. Recombinant adiponectin peptide promotes neuronal survival after intracerebral haemorrhage by suppressing mitochondrial and ATF4-CHOP apoptosis pathways in diabetic mice via Smad3 signalling inhibition.
Wu X; Luo J; Liu H; Cui W; Guo W; Zhao L; Guo H; Bai H; Guo K; Feng D; Qu Y
Cell Prolif; 2020 Feb; 53(2):e12759. PubMed ID: 31922310
[TBL] [Abstract][Full Text] [Related]
16. CCAAT/enhancer binding protein-beta is a transcriptional regulator of peroxisome-proliferator-activated receptor-gamma coactivator-1alpha in the regenerating liver.
Wang H; Peiris TH; Mowery A; Le Lay J; Gao Y; Greenbaum LE
Mol Endocrinol; 2008 Jul; 22(7):1596-605. PubMed ID: 18467525
[TBL] [Abstract][Full Text] [Related]
17. High glucose-induced oxidative stress represses sirtuin deacetylase expression and increases histone acetylation leading to neural tube defects.
Yu J; Wu Y; Yang P
J Neurochem; 2016 May; 137(3):371-83. PubMed ID: 26896748
[TBL] [Abstract][Full Text] [Related]
18. Effects of maternal diabetes and fetal sex on human placenta mitochondrial biogenesis.
Jiang S; Teague AM; Tryggestad JB; Aston CE; Lyons T; Chernausek SD
Placenta; 2017 Sep; 57():26-32. PubMed ID: 28864016
[TBL] [Abstract][Full Text] [Related]
19. IDH1, a CHOP and C/EBPβ-responsive gene under ER stress, sensitizes human melanoma cells to hypoxia-induced apoptosis.
Yang X; Du T; Wang X; Zhang Y; Hu W; Du X; Miao L; Han C
Cancer Lett; 2015 Sep; 365(2):201-10. PubMed ID: 26049021
[TBL] [Abstract][Full Text] [Related]
20. Endoplasmic reticulum stress downregulates PGC-1α in skeletal muscle through ATF4 and an mTOR-mediated reduction of CRTC2.
Montori-Grau M; Aguilar-Recarte D; Zarei M; Pizarro-Delgado J; Palomer X; Vázquez-Carrera M
Cell Commun Signal; 2022 Apr; 20(1):53. PubMed ID: 35428325
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
