252 related articles for article (PubMed ID: 28500787)
1. Corticosterone primes the neuroinflammatory response to Gulf War Illness-relevant organophosphates independently of acetylcholinesterase inhibition.
Locker AR; Michalovicz LT; Kelly KA; Miller JV; Miller DB; O'Callaghan JP
J Neurochem; 2017 Aug; 142(3):444-455. PubMed ID: 28500787
[TBL] [Abstract][Full Text] [Related]
2. Corticosterone primes the neuroinflammatory response to DFP in mice: potential animal model of Gulf War Illness.
O'Callaghan JP; Kelly KA; Locker AR; Miller DB; Lasley SM
J Neurochem; 2015 Jun; 133(5):708-21. PubMed ID: 25753028
[TBL] [Abstract][Full Text] [Related]
3. The Neuroinflammatory Phenotype in a Mouse Model of Gulf War Illness is Unrelated to Brain Regional Levels of Acetylcholine as Measured by Quantitative HILIC-UPLC-MS/MS.
Miller JV; LeBouf RF; Kelly KA; Michalovicz LT; Ranpara A; Locker AR; Miller DB; O'Callaghan JP
Toxicol Sci; 2018 Oct; 165(2):302-313. PubMed ID: 29846716
[TBL] [Abstract][Full Text] [Related]
4. Corticosterone and pyridostigmine/DEET exposure attenuate peripheral cytokine expression: Supporting a dominant role for neuroinflammation in a mouse model of Gulf War Illness.
Michalovicz LT; Locker AR; Kelly KA; Miller JV; Barnes Z; Fletcher MA; Miller DB; Klimas NG; Morris M; Lasley SM; O'Callaghan JP
Neurotoxicology; 2019 Jan; 70():26-32. PubMed ID: 30339781
[TBL] [Abstract][Full Text] [Related]
5. Epigenetic impacts of stress priming of the neuroinflammatory response to sarin surrogate in mice: a model of Gulf War illness.
Ashbrook DG; Hing B; Michalovicz LT; Kelly KA; Miller JV; de Vega WC; Miller DB; Broderick G; O'Callaghan JP; McGowan PO
J Neuroinflammation; 2018 Mar; 15(1):86. PubMed ID: 29549885
[TBL] [Abstract][Full Text] [Related]
6. Corticosterone potentiates DFP-induced neuroinflammation and affects high-order diffusion imaging in a rat model of Gulf War Illness.
Koo BB; Michalovicz LT; Calderazzo S; Kelly KA; Sullivan K; Killiany RJ; O'Callaghan JP
Brain Behav Immun; 2018 Jan; 67():42-46. PubMed ID: 28782715
[TBL] [Abstract][Full Text] [Related]
7. The β-adrenergic receptor blocker and anti-inflammatory drug propranolol mitigates brain cytokine expression in a long-term model of Gulf War Illness.
Michalovicz LT; Kelly KA; Miller DB; Sullivan K; O'Callaghan JP
Life Sci; 2021 Nov; 285():119962. PubMed ID: 34563566
[TBL] [Abstract][Full Text] [Related]
8. Differential phosphoprotein signaling in the cortex in mouse models of Gulf War Illness using corticosterone and acetylcholinesterase inhibitors.
Penatzer JA; Miller JV; Prince N; Shaw M; Lynch C; Newman M; Hobbs GR; Boyd JW
Heliyon; 2021 Jul; 7(7):e07552. PubMed ID: 34307952
[TBL] [Abstract][Full Text] [Related]
9. Genome-wide transcriptome architecture in a mouse model of Gulf War Illness.
Xu F; Ashbrook DG; Gao J; Starlard-Davenport A; Zhao W; Miller DB; O'Callaghan JP; Williams RW; Jones BC; Lu L
Brain Behav Immun; 2020 Oct; 89():209-223. PubMed ID: 32574576
[TBL] [Abstract][Full Text] [Related]
10. Neurochemical and neuroinflammatory perturbations in two Gulf War Illness models: Modulation by the immunotherapeutic LNFPIII.
Carpenter JM; Gordon HE; Ludwig HD; Wagner JJ; Harn DA; Norberg T; Filipov NM
Neurotoxicology; 2020 Mar; 77():40-50. PubMed ID: 31866310
[TBL] [Abstract][Full Text] [Related]
11. Acetylcholinesterase inhibitor exposures as an initiating factor in the development of Gulf War Illness, a chronic neuroimmune disorder in deployed veterans.
Michalovicz LT; Kelly KA; Sullivan K; O'Callaghan JP
Neuropharmacology; 2020 Jul; 171():108073. PubMed ID: 32247728
[TBL] [Abstract][Full Text] [Related]
12. Gastrointestinal neuroimmune disruption in a mouse model of Gulf War illness.
Hernandez S; Fried DE; Grubišić V; McClain JL; Gulbransen BD
FASEB J; 2019 May; 33(5):6168-6184. PubMed ID: 30789759
[TBL] [Abstract][Full Text] [Related]
13. Fingolimod mitigates memory loss in a mouse model of Gulf War Illness amid decreasing the activation of microglia, protein kinase R, and NFκB.
Carreras I; Jung Y; Lopez-Benitez J; Tognoni CM; Dedeoglu A
Neurotoxicology; 2023 May; 96():197-206. PubMed ID: 37160207
[TBL] [Abstract][Full Text] [Related]
14. Oligodendrocyte involvement in Gulf War Illness.
Belgrad J; Dutta DJ; Bromley-Coolidge S; Kelly KA; Michalovicz LT; Sullivan KA; O'Callaghan JP; Fields RD
Glia; 2019 Nov; 67(11):2107-2124. PubMed ID: 31339622
[TBL] [Abstract][Full Text] [Related]
15. Delayed cognitive impairments in a rat model of Gulf War Illness are stimulus-dependent.
Burzynski HE; Ayala KE; Frick MA; Dufala HA; Woodruff JL; Macht VA; Eberl BR; Hollis F; McQuail JA; Grillo CA; Fadel JR; Reagan LP
Brain Behav Immun; 2023 Oct; 113():248-258. PubMed ID: 37437820
[TBL] [Abstract][Full Text] [Related]
16. Exposure to an organophosphate pesticide, individually or in combination with other Gulf War agents, impairs synaptic integrity and neuronal differentiation, and is accompanied by subtle microvascular injury in a mouse model of Gulf War agent exposure.
Ojo JO; Abdullah L; Evans J; Reed JM; Montague H; Mullan MJ; Crawford FC
Neuropathology; 2014 Apr; 34(2):109-27. PubMed ID: 24118348
[TBL] [Abstract][Full Text] [Related]
17. A role for neuroimmune signaling in a rat model of Gulf War Illness-related pain.
Lacagnina MJ; Li J; Lorca S; Rice KC; Sullivan K; O'Callaghan JP; Grace PM
Brain Behav Immun; 2021 Jan; 91():418-428. PubMed ID: 33127584
[TBL] [Abstract][Full Text] [Related]
18. Pyridostigmine bromide exposure creates chronic, underlying neuroimmune disruption in the gastrointestinal tract and brain that alters responses to palmitoylethanolamide in a mouse model of Gulf War Illness.
Hernandez S; Morales-Soto W; Grubišić V; Fried D; Gulbransen BD
Neuropharmacology; 2020 Nov; 179():108264. PubMed ID: 32758565
[TBL] [Abstract][Full Text] [Related]
19. Pathophysiology in a model of Gulf War Illness: Contributions of pyridostigmine bromide and stress.
Macht VA; Woodruff JL; Grillo CA; Wood CS; Wilson MA; Reagan LP
Psychoneuroendocrinology; 2018 Oct; 96():195-202. PubMed ID: 30041099
[TBL] [Abstract][Full Text] [Related]
20. Gulf War toxicant-induced reductions in dendritic arbors and spine densities of dentate granule cells are improved by treatment with a Nrf2 activator.
Murray KE; Ratliff WA; Delic V; Citron BA
Brain Res; 2024 Jan; 1823():148682. PubMed ID: 37989436
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]