BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

646 related articles for article (PubMed ID: 28598725)

  • 1. A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humans.
    Chernoff N; Hill DJ; Diggs DL; Faison BD; Francis BM; Lang JR; Larue MM; Le TT; Loftin KA; Lugo JN; Schmid JE; Winnik WM
    J Toxicol Environ Health B Crit Rev; 2017; 20(4):1-47. PubMed ID: 28598725
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Beyond Guam: the cyanobacteria/BMAA hypothesis of the cause of ALS and other neurodegenerative diseases.
    Bradley WG; Mash DC
    Amyotroph Lateral Scler; 2009; 10 Suppl 2():7-20. PubMed ID: 19929726
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Biomagnification of cyanobacterial neurotoxins and neurodegenerative disease among the Chamorro people of Guam.
    Cox PA; Banack SA; Murch SJ
    Proc Natl Acad Sci U S A; 2003 Nov; 100(23):13380-3. PubMed ID: 14612559
    [TBL] [Abstract][Full Text] [Related]  

  • 4. A mechanism for slow release of biomagnified cyanobacterial neurotoxins and neurodegenerative disease in Guam.
    Murch SJ; Cox PA; Banack SA
    Proc Natl Acad Sci U S A; 2004 Aug; 101(33):12228-31. PubMed ID: 15295100
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Return of the cycad hypothesis - does the amyotrophic lateral sclerosis/parkinsonism dementia complex (ALS/PDC) of Guam have new implications for global health?
    Ince PG; Codd GA
    Neuropathol Appl Neurobiol; 2005 Aug; 31(4):345-53. PubMed ID: 16008818
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Biomagnification of cycad neurotoxins in flying foxes: implications for ALS-PDC in Guam.
    Banack SA; Cox PA
    Neurology; 2003 Aug; 61(3):387-9. PubMed ID: 12913204
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Creating a Simian Model of Guam ALS/PDC Which Reflects Chamorro Lifetime BMAA Exposures.
    Banack SA; Cox PA
    Neurotox Res; 2018 Jan; 33(1):24-32. PubMed ID: 28478528
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Animal models of BMAA neurotoxicity: a critical review.
    Karamyan VT; Speth RC
    Life Sci; 2008 Jan; 82(5-6):233-46. PubMed ID: 18191417
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The cyanobacteria derived toxin Beta-N-methylamino-L-alanine and amyotrophic lateral sclerosis.
    Banack SA; Caller TA; Stommel EW
    Toxins (Basel); 2010 Dec; 2(12):2837-50. PubMed ID: 22069578
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Parkinsonism and motor neuron disorders: Lessons from Western Pacific ALS/PDC.
    Spencer PS
    J Neurol Sci; 2022 Feb; 433():120021. PubMed ID: 34635325
    [TBL] [Abstract][Full Text] [Related]  

  • 11. BMAA and Neurodegenerative Illness.
    Cox PA; Kostrzewa RM; Guillemin GJ
    Neurotox Res; 2018 Jan; 33(1):178-183. PubMed ID: 28540663
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Occurrence of beta-methylamino-l-alanine (BMAA) in ALS/PDC patients from Guam.
    Murch SJ; Cox PA; Banack SA; Steele JC; Sacks OW
    Acta Neurol Scand; 2004 Oct; 110(4):267-9. PubMed ID: 15355492
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm.
    Papapetropoulos S
    Neurochem Int; 2007 Jun; 50(7-8):998-1003. PubMed ID: 17296249
    [TBL] [Abstract][Full Text] [Related]  

  • 14. 2-Amino-3-(methylamino)-propanoic acid (BMAA) in cycad flour: an unlikely cause of amyotrophic lateral sclerosis and parkinsonism-dementia of Guam.
    Duncan MW; Steele JC; Kopin IJ; Markey SP
    Neurology; 1990 May; 40(5):767-72. PubMed ID: 2330104
    [TBL] [Abstract][Full Text] [Related]  

  • 15. The cycad neurotoxic amino acid, beta-N-methylamino-L-alanine (BMAA), elevates intracellular calcium levels in dissociated rat brain cells.
    Brownson DM; Mabry TJ; Leslie SW
    J Ethnopharmacol; 2002 Oct; 82(2-3):159-67. PubMed ID: 12241991
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Commentary on: Return of the cycad hypothesis--does the amyotrophic lateral sclerosis/Parkinsonism dementia complex (ALS/PDC) of Guam have new implications for global health?
    Cox PA; Banack S; Murch S; Sacks O
    Neuropathol Appl Neurobiol; 2006 Dec; 32(6):679-82. PubMed ID: 17083482
    [No Abstract]   [Full Text] [Related]  

  • 17. Does α-amino-β-methylaminopropionic acid (BMAA) play a role in neurodegeneration?
    Chiu AS; Gehringer MM; Welch JH; Neilan BA
    Int J Environ Res Public Health; 2011 Sep; 8(9):3728-46. PubMed ID: 22016712
    [TBL] [Abstract][Full Text] [Related]  

  • 18. beta-N-methylamino-l-alanine induces oxidative stress and glutamate release through action on system Xc(-).
    Liu X; Rush T; Zapata J; Lobner D
    Exp Neurol; 2009 Jun; 217(2):429-33. PubMed ID: 19374900
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Content of the neurotoxins cycasin (methylazoxymethanol beta-D-glucoside) and BMAA (beta-N-methylamino-L-alanine) in cycad flour prepared by Guam Chamorros.
    Kisby GE; Ellison M; Spencer PS
    Neurology; 1992 Jul; 42(7):1336-40. PubMed ID: 1620343
    [TBL] [Abstract][Full Text] [Related]  

  • 20. BMAA selectively injures motor neurons via AMPA/kainate receptor activation.
    Rao SD; Banack SA; Cox PA; Weiss JH
    Exp Neurol; 2006 Sep; 201(1):244-52. PubMed ID: 16764863
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 33.