These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

537 related articles for article (PubMed ID: 28614711)

  • 1. DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP Specific.
    Goodell DJ; Zaegel V; Coultrap SJ; Hell JW; Bayer KU
    Cell Rep; 2017 Jun; 19(11):2231-2243. PubMed ID: 28614711
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode.
    Larsen ME; Buonarati OR; Qian H; Hell JW; Bayer KU
    J Biol Chem; 2023 Jun; 299(6):104706. PubMed ID: 37061000
    [TBL] [Abstract][Full Text] [Related]  

  • 3. CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity.
    Cook SG; Rumian NL; Bayer KU
    J Biol Chem; 2022 Sep; 298(9):102299. PubMed ID: 35872016
    [TBL] [Abstract][Full Text] [Related]  

  • 4. CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac Arrest.
    Buonarati OR; Cook SG; Goodell DJ; Chalmers NE; Rumian NL; Tullis JE; Restrepo S; Coultrap SJ; Quillinan N; Herson PS; Bayer KU
    Cell Rep; 2020 Jan; 30(1):1-8.e4. PubMed ID: 31914378
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength.
    Barcomb K; Hell JW; Benke TA; Bayer KU
    J Biol Chem; 2016 Jul; 291(31):16082-9. PubMed ID: 27246855
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Distinct synaptic pools of DAPK1 differentially regulate activity-dependent synaptic CaMKII accumulation.
    Tullis JE; Bayer KU
    iScience; 2023 May; 26(5):106723. PubMed ID: 37216104
    [TBL] [Abstract][Full Text] [Related]  

  • 7. CaMKII autophosphorylation is the only enzymatic event required for synaptic memory.
    Chen X; Cai Q; Zhou J; Pleasure SJ; Schulman H; Zhang M; Nicoll RA
    Proc Natl Acad Sci U S A; 2024 Jun; 121(26):e2402783121. PubMed ID: 38889145
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Autonomous CaMKII mediates both LTP and LTD using a mechanism for differential substrate site selection.
    Coultrap SJ; Freund RK; O'Leary H; Sanderson JL; Roche KW; Dell'Acqua ML; Bayer KU
    Cell Rep; 2014 Feb; 6(3):431-7. PubMed ID: 24485660
    [TBL] [Abstract][Full Text] [Related]  

  • 9. CaMKII regulates the depalmitoylation and synaptic removal of the scaffold protein AKAP79/150 to mediate structural long-term depression.
    Woolfrey KM; O'Leary H; Goodell DJ; Robertson HR; Horne EA; Coultrap SJ; Dell'Acqua ML; Bayer KU
    J Biol Chem; 2018 Feb; 293(5):1551-1567. PubMed ID: 29196604
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Nucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2B.
    O'Leary H; Liu WH; Rorabaugh JM; Coultrap SJ; Bayer KU
    J Biol Chem; 2011 Sep; 286(36):31272-81. PubMed ID: 21768120
    [TBL] [Abstract][Full Text] [Related]  

  • 11. CaMKII-mediated phosphorylation of GluN2B regulates recombinant NMDA receptor currents in a chloride-dependent manner.
    Tavalin SJ; Colbran RJ
    Mol Cell Neurosci; 2017 Mar; 79():45-52. PubMed ID: 27998718
    [TBL] [Abstract][Full Text] [Related]  

  • 12. CaMKII binding to GluN2B is differentially affected by macromolecular crowding reagents.
    Goodell DJ; Eliseeva TA; Coultrap SJ; Bayer KU
    PLoS One; 2014; 9(5):e96522. PubMed ID: 24796865
    [TBL] [Abstract][Full Text] [Related]  

  • 13. CaMKII regulation in information processing and storage.
    Coultrap SJ; Bayer KU
    Trends Neurosci; 2012 Oct; 35(10):607-18. PubMed ID: 22717267
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Modelling the dynamics of CaMKII-NMDAR complex related to memory formation in synapses: the possible roles of threonine 286 autophosphorylation of CaMKII in long term potentiation.
    He Y; Kulasiri D; Samarasinghe S
    J Theor Biol; 2015 Jan; 365():403-19. PubMed ID: 25446714
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Differential stimulus-dependent synaptic recruitment of CaMKIIα by intracellular determinants of GluN2B.
    She K; Rose JK; Craig AM
    Mol Cell Neurosci; 2012 Nov; 51(3-4):68-78. PubMed ID: 22902837
    [TBL] [Abstract][Full Text] [Related]  

  • 16. On the mechanism of synaptic depression induced by CaMKIIN, an endogenous inhibitor of CaMKII.
    Gouet C; Aburto B; Vergara C; Sanhueza M
    PLoS One; 2012; 7(11):e49293. PubMed ID: 23145145
    [TBL] [Abstract][Full Text] [Related]  

  • 17. A Modeling and Analysis Study Reveals That CaMKII in Synaptic Plasticity Is a Dominant Affecter in CaM Systems in a T286 Phosphorylation-Dependent Manner.
    Stevens-Bullmore H; Kulasiri D; Samarasinghe S
    Molecules; 2022 Sep; 27(18):. PubMed ID: 36144710
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Role of inhibitory autophosphorylation of calcium/calmodulin-dependent kinase II (αCAMKII) in persistent (>24 h) hippocampal LTP and in LTD facilitated by novel object-place learning and recognition in mice.
    Goh JJ; Manahan-Vaughan D
    Behav Brain Res; 2015 May; 285():79-88. PubMed ID: 24480420
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Altered GluN2B NMDA receptor function and synaptic plasticity during early pathology in the PS2APP mouse model of Alzheimer's disease.
    Hanson JE; Pare JF; Deng L; Smith Y; Zhou Q
    Neurobiol Dis; 2015 Feb; 74():254-62. PubMed ID: 25484285
    [TBL] [Abstract][Full Text] [Related]  

  • 20. GluN2A and GluN2B subunit-containing NMDA receptors in hippocampal plasticity.
    Shipton OA; Paulsen O
    Philos Trans R Soc Lond B Biol Sci; 2014 Jan; 369(1633):20130163. PubMed ID: 24298164
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 27.