BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

233 related articles for article (PubMed ID: 28733489)

  • 1. Interaction of misfolded proteins and mitochondria in neurodegenerative disorders.
    Abramov AY; Berezhnov AV; Fedotova EI; Zinchenko VP; Dolgacheva LP
    Biochem Soc Trans; 2017 Aug; 45(4):1025-1033. PubMed ID: 28733489
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Interaction of Oxidative Stress and Misfolded Proteins in the Mechanism of Neurodegeneration.
    Abramov AY; Potapova EV; Dremin VV; Dunaev AV
    Life (Basel); 2020 Jun; 10(7):. PubMed ID: 32629809
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Alpha-synuclein and beta-amyloid - different targets, same players: calcium, free radicals and mitochondria in the mechanism of neurodegeneration.
    Angelova PR; Abramov AY
    Biochem Biophys Res Commun; 2017 Feb; 483(4):1110-1115. PubMed ID: 27470584
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Mitochondrial dysfunction and neurodegenerative proteinopathies: mechanisms and prospects for therapeutic intervention.
    Briston T; Hicks AR
    Biochem Soc Trans; 2018 Aug; 46(4):829-842. PubMed ID: 29986938
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Role of protein aggregation in mitochondrial dysfunction and neurodegeneration in Alzheimer's and Parkinson's diseases.
    Hashimoto M; Rockenstein E; Crews L; Masliah E
    Neuromolecular Med; 2003; 4(1-2):21-36. PubMed ID: 14528050
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Mitochondrial membrane permeabilisation by amyloid aggregates and protection by polyphenols.
    Camilleri A; Zarb C; Caruana M; Ostermeier U; Ghio S; Högen T; Schmidt F; Giese A; Vassallo N
    Biochim Biophys Acta; 2013 Nov; 1828(11):2532-43. PubMed ID: 23817009
    [TBL] [Abstract][Full Text] [Related]  

  • 7. The Role of Extracellular Matrix Components in the Spreading of Pathological Protein Aggregates.
    Moretto E; Stuart S; Surana S; Vargas JNS; Schiavo G
    Front Cell Neurosci; 2022; 16():844211. PubMed ID: 35573838
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Mitochondrial Dynamics and Proteins Related to Neurodegenerative Diseases.
    Alexiou A; Nizami B; Khan FI; Soursou G; Vairaktarakis C; Chatzichronis S; Tsiamis V; Manztavinos V; Yarla NS; Md Ashraf G
    Curr Protein Pept Sci; 2018; 19(9):850-857. PubMed ID: 28799502
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Retinol (Vitamin A) Increases α-Synuclein, β-Amyloid Peptide, Tau Phosphorylation and RAGE Content in Human SH-SY5Y Neuronal Cell Line.
    Kunzler A; Kolling EA; da Silva JD; Gasparotto J; de Bittencourt Pasquali MA; Moreira JCF; Gelain DP
    Neurochem Res; 2017 Oct; 42(10):2788-2797. PubMed ID: 28497345
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Is alpha-synuclein pathology a target for treatment of neurodegenerative disorders?
    Windisch M; Wolf HJ; Hutter-Paier B; Hofmeister A; Wronski R
    Curr Alzheimer Res; 2007 Sep; 4(4):446-57. PubMed ID: 17908049
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Monomeric Alpha-Synuclein Exerts a Physiological Role on Brain ATP Synthase.
    Ludtmann MH; Angelova PR; Ninkina NN; Gandhi S; Buchman VL; Abramov AY
    J Neurosci; 2016 Oct; 36(41):10510-10521. PubMed ID: 27733604
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Mitochondrial Ca
    Naia L; Ferreira IL; Ferreiro E; Rego AC
    Biochem Biophys Res Commun; 2017 Feb; 483(4):1069-1077. PubMed ID: 27485547
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Pathogenic mechanisms of prion protein, amyloid-β and α-synuclein misfolding: the prion concept and neurotoxicity of protein oligomers.
    Ugalde CL; Finkelstein DI; Lawson VA; Hill AF
    J Neurochem; 2016 Oct; 139(2):162-180. PubMed ID: 27529376
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Heat shock proteins as suppressors of accumulation of toxic prefibrillar intermediates and misfolded proteins in neurodegenerative diseases.
    Arawaka S; Machiya Y; Kato T
    Curr Pharm Biotechnol; 2010 Feb; 11(2):158-66. PubMed ID: 20170473
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Influence of physical exercise on β-amyloid, α-synuclein and tau accumulation: an in vitro model of oxidative stress in human red blood cells.
    Iofrida C; Daniele S; Pietrobono D; Fusi J; Galetta F; Trincavelli ML; Bonuccelli U; Franzoni F; Martini C
    Arch Ital Biol; 2017 Jul; 155(1-2):33-42. PubMed ID: 28715596
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Development or disease: duality of the mitochondrial permeability transition pore.
    Pérez MJ; Quintanilla RA
    Dev Biol; 2017 Jun; 426(1):1-7. PubMed ID: 28457864
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Insights into mitochondrial dysfunction: aging, amyloid-β, and tau-A deleterious trio.
    Schmitt K; Grimm A; Kazmierczak A; Strosznajder JB; Götz J; Eckert A
    Antioxid Redox Signal; 2012 Jun; 16(12):1456-66. PubMed ID: 22117646
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Antioxidant modulation in restoring mitochondrial function in neurodegeneration.
    Pahrudin Arrozi A; Wan Ngah WZ; Mohd Yusof YA; Ahmad Damanhuri MH; Makpol S
    Int J Neurosci; 2017 Mar; 127(3):218-235. PubMed ID: 27074540
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Mitochondria, metabolic disturbances, oxidative stress and the kynurenine system, with focus on neurodegenerative disorders.
    Sas K; Robotka H; Toldi J; Vécsei L
    J Neurol Sci; 2007 Jun; 257(1-2):221-39. PubMed ID: 17462670
    [TBL] [Abstract][Full Text] [Related]  

  • 20. alpha-synuclein aggregation: a link between mitochondrial defects and Parkinson's disease?
    Lee SJ
    Antioxid Redox Signal; 2003 Jun; 5(3):337-48. PubMed ID: 12880487
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 12.