222 related articles for article (PubMed ID: 28760883)
1. Cutting Edge: NKG2D Signaling Enhances NK Cell Responses but Alone Is Insufficient To Drive Expansion during Mouse Cytomegalovirus Infection.
Nabekura T; Gotthardt D; Niizuma K; Trsan T; Jenus T; Jonjic S; Lanier LL
J Immunol; 2017 Sep; 199(5):1567-1571. PubMed ID: 28760883
[TBL] [Abstract][Full Text] [Related]
2. Ly49H signaling through DAP10 is essential for optimal natural killer cell responses to mouse cytomegalovirus infection.
Orr MT; Sun JC; Hesslein DG; Arase H; Phillips JH; Takai T; Lanier LL
J Exp Med; 2009 Apr; 206(4):807-17. PubMed ID: 19332875
[TBL] [Abstract][Full Text] [Related]
3. Activation receptor-induced tolerance of mature NK cells in vivo requires signaling through the receptor and is reversible.
Bolanos FD; Tripathy SK
J Immunol; 2011 Mar; 186(5):2765-71. PubMed ID: 21263069
[TBL] [Abstract][Full Text] [Related]
4. Ly49H engagement compensates for the absence of type I interferon signaling in stimulating NK cell proliferation during murine cytomegalovirus infection.
Geurs TL; Zhao YM; Hill EB; French AR
J Immunol; 2009 Nov; 183(9):5830-6. PubMed ID: 19828630
[TBL] [Abstract][Full Text] [Related]
5. Altered NK cell development and enhanced NK cell-mediated resistance to mouse cytomegalovirus in NKG2D-deficient mice.
Zafirova B; Mandarić S; Antulov R; Krmpotić A; Jonsson H; Yokoyama WM; Jonjić S; Polić B
Immunity; 2009 Aug; 31(2):270-82. PubMed ID: 19631564
[TBL] [Abstract][Full Text] [Related]
6. Natural Killer Cell Sensing of Infected Cells Compensates for MyD88 Deficiency but Not IFN-I Activity in Resistance to Mouse Cytomegalovirus.
Cocita C; Guiton R; Bessou G; Chasson L; Boyron M; Crozat K; Dalod M
PLoS Pathog; 2015 May; 11(5):e1004897. PubMed ID: 25954804
[TBL] [Abstract][Full Text] [Related]
7. Cmv1-independent antiviral role of NK cells revealed in murine cytomegalovirus-infected New Zealand White mice.
Rodriguez M; Sabastian P; Clark P; Brown MG
J Immunol; 2004 Nov; 173(10):6312-8. PubMed ID: 15528370
[TBL] [Abstract][Full Text] [Related]
8. DAP10 associates with Ly49 receptors but contributes minimally to their expression and function in vivo.
Tassi I; Le Friec G; Gilfillan S; Takai T; Yokoyama WM; Colonna M
Eur J Immunol; 2009 Apr; 39(4):1129-35. PubMed ID: 19247984
[TBL] [Abstract][Full Text] [Related]
9. Ly49h-deficient C57BL/6 mice: a new mouse cytomegalovirus-susceptible model remains resistant to unrelated pathogens controlled by the NK gene complex.
Fodil-Cornu N; Lee SH; Belanger S; Makrigiannis AP; Biron CA; Buller RM; Vidal SM
J Immunol; 2008 Nov; 181(9):6394-405. PubMed ID: 18941230
[TBL] [Abstract][Full Text] [Related]
10. Ly49C Impairs NK Cell Memory in Mouse Cytomegalovirus Infection.
Forbes CA; Scalzo AA; Degli-Esposti MA; Coudert JD
J Immunol; 2016 Jul; 197(1):128-40. PubMed ID: 27233959
[TBL] [Abstract][Full Text] [Related]
11. NKG2D stimulation of CD8
Kavazović I; Lenartić M; Jelenčić V; Jurković S; Lemmermann NAW; Jonjić S; Polić B; Wensveen FM
Eur J Immunol; 2017 Jul; 47(7):1123-1135. PubMed ID: 28378389
[TBL] [Abstract][Full Text] [Related]
12. Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection.
Min-Oo G; Lanier LL
J Exp Med; 2014 Dec; 211(13):2669-80. PubMed ID: 25422494
[TBL] [Abstract][Full Text] [Related]
13. T cells gene-engineered with DAP12 mediate effector function in an NKG2D-dependent and major histocompatibility complex-independent manner.
Teng MW; Kershaw MH; Hayakawa Y; Cerutti L; Jane SM; Darcy PK; Smyth MJ
J Biol Chem; 2005 Nov; 280(46):38235-41. PubMed ID: 16169855
[TBL] [Abstract][Full Text] [Related]
14. Type I IFN promotes NK cell expansion during viral infection by protecting NK cells against fratricide.
Madera S; Rapp M; Firth MA; Beilke JN; Lanier LL; Sun JC
J Exp Med; 2016 Feb; 213(2):225-33. PubMed ID: 26755706
[TBL] [Abstract][Full Text] [Related]
15. NKG2D Signaling: The Immune Subversive Side of HDAC3.
Folgueras AR; Gonzalez S; López-Soto A
Trends Immunol; 2017 Mar; 38(3):151-153. PubMed ID: 28089218
[TBL] [Abstract][Full Text] [Related]
16. Viral infection transiently reverses activation receptor-mediated NK cell hyporesponsiveness in an MHC class I-independent mechanism.
Mazumdar B; Bolanos FD; Tripathy SK
Eur J Immunol; 2013 May; 43(5):1345-55. PubMed ID: 23440763
[TBL] [Abstract][Full Text] [Related]
17. Expansion and Protection by a Virus-Specific NK Cell Subset Lacking Expression of the Inhibitory NKR-P1B Receptor during Murine Cytomegalovirus Infection.
Rahim MM; Wight A; Mahmoud AB; Aguilar OA; Lee SH; Vidal SM; Carlyle JR; Makrigiannis AP
J Immunol; 2016 Sep; 197(6):2325-37. PubMed ID: 27511735
[TBL] [Abstract][Full Text] [Related]
18. Dual Requirement of Cytokine and Activation Receptor Triggering for Cytotoxic Control of Murine Cytomegalovirus by NK Cells.
Parikh BA; Piersma SJ; Pak-Wittel MA; Yang L; Schreiber RD; Yokoyama WM
PLoS Pathog; 2015 Dec; 11(12):e1005323. PubMed ID: 26720279
[TBL] [Abstract][Full Text] [Related]
19. Continuous engagement of a self-specific activation receptor induces NK cell tolerance.
Tripathy SK; Keyel PA; Yang L; Pingel JT; Cheng TP; Schneeberger A; Yokoyama WM
J Exp Med; 2008 Aug; 205(8):1829-41. PubMed ID: 18606857
[TBL] [Abstract][Full Text] [Related]
20. Activating Receptors for Self-MHC Class I Enhance Effector Functions and Memory Differentiation of NK Cells during Mouse Cytomegalovirus Infection.
Nabekura T; Lanier LL
Immunity; 2016 Jul; 45(1):74-82. PubMed ID: 27438766
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
