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4. ADAMTS9 and ADAMTS20 are differentially affected by loss of B3GLCT in mouse model of Peters plus syndrome. Holdener BC; Percival CJ; Grady RC; Cameron DC; Berardinelli SJ; Zhang A; Neupane S; Takeuchi M; Jimenez-Vega JC; Uddin SMZ; Komatsu DE; Honkanen R; Dubail J; Apte SS; Sato T; Narimatsu H; McClain SA; Haltiwanger RS Hum Mol Genet; 2019 Dec; 28(24):4053-4066. PubMed ID: 31600785 [TBL] [Abstract][Full Text] [Related]
5. Peters plus syndrome and Chorioretinal findings associated with B3GLCT gene mutation - a case report. Wang YE; Ramirez DA; Chang TC; Berrocal A BMC Ophthalmol; 2020 Mar; 20(1):118. PubMed ID: 32204707 [TBL] [Abstract][Full Text] [Related]
6. Contribution of a Novel Totoń-Żurańska J; Kapusta P; Rybak-Krzyszkowska M; Lorenc K; Machlowska J; Skalniak A; Filipek E; Pawlik D; Wołkow PP Int J Mol Sci; 2019 Nov; 20(23):. PubMed ID: 31795264 [TBL] [Abstract][Full Text] [Related]
7. Novel B3GALTL mutations in classic Peters plus syndrome and lack of mutations in a large cohort of patients with similar phenotypes. Weh E; Reis LM; Tyler RC; Bick D; Rhead WJ; Wallace S; McGregor TL; Dills SK; Chao MC; Murray JC; Semina EV Clin Genet; 2014 Aug; 86(2):142-8. PubMed ID: 23889335 [TBL] [Abstract][Full Text] [Related]
8. Hydrocephalus in mouse B3glct mutants is likely caused by defects in multiple B3GLCT substrates in ependymal cells and subcommissural organ. Neupane S; Goto J; Berardinelli SJ; Ito A; Haltiwanger RS; Holdener BC Glycobiology; 2021 Sep; 31(8):988-1004. PubMed ID: 33909046 [TBL] [Abstract][Full Text] [Related]
9. First functional analysis of a novel splicing mutation in the B3GALTL gene by an ex vivo approach in Tunisian patients with typical Peters plus syndrome. Ben Mahmoud A; Siala O; Mansour RB; Driss F; Baklouti-Gargouri S; Mkaouar-Rebai E; Belguith N; Fakhfakh F Gene; 2013 Dec; 532(1):13-7. PubMed ID: 23954224 [TBL] [Abstract][Full Text] [Related]
12. Peters Plus syndrome: a recognizable clinical entity. Demir GÜ; Lafcı NG; Doğan ÖA; Şimşek-Kiper PÖ; Utine GE Turk J Pediatr; 2020; 62(1):136-140. PubMed ID: 32253880 [TBL] [Abstract][Full Text] [Related]
13. Two Tunisian patients with Peters plus syndrome harbouring a novel splice site mutation in the B3GALTL gene that modulates the mRNA secondary structure. Siala O; Belguith N; Kammoun H; Kammoun B; Hmida N; Chabchoub I; Hchicha M; Fakhfakh F Gene; 2012 Oct; 507(1):68-73. PubMed ID: 22759511 [TBL] [Abstract][Full Text] [Related]
14. Analyzing the Effects of O-Fucosylation on Secretion of ADAMTS Proteins Using Cell-Based Assays. Berardinelli SJ; Haltiwanger RS Methods Mol Biol; 2020; 2043():25-43. PubMed ID: 31463900 [TBL] [Abstract][Full Text] [Related]
15. Impaired ADAMTS9 secretion: A potential mechanism for eye defects in Peters Plus Syndrome. Dubail J; Vasudevan D; Wang LW; Earp SE; Jenkins MW; Haltiwanger RS; Apte SS Sci Rep; 2016 Sep; 6():33974. PubMed ID: 27687499 [TBL] [Abstract][Full Text] [Related]
16. Ocular Phenotype of Peters-Plus Syndrome. Shah PR; Chauhan B; Chu CT; Kofler J; Nischal KK Cornea; 2022 Feb; 41(2):219-223. PubMed ID: 34629439 [TBL] [Abstract][Full Text] [Related]
17. Loss of the AMD-associated B3GLCT gene affects glycosylation of TSP1 without impairing secretion in retinal pigment epithelial cells. Lauwen S; Baerenfaenger M; Ruigrok S; de Jong EK; Wessels HJCT; den Hollander AI; Lefeber DJ Exp Eye Res; 2021 Dec; 213():108798. PubMed ID: 34695439 [TBL] [Abstract][Full Text] [Related]
18. Hydrocephalus, agenesis of the corpus callosum, and cleft lip/palate represent frequent associations in fetuses with Peters' plus syndrome and B3GALTL mutations. Fetal PPS phenotypes, expanded by Dandy Walker cyst and encephalocele. Schoner K; Kohlhase J; Müller AM; Schramm T; Plassmann M; Schmitz R; Neesen J; Wieacker P; Rehder H Prenat Diagn; 2013 Jan; 33(1):75-80. PubMed ID: 23161355 [TBL] [Abstract][Full Text] [Related]
19. Genomic, cDNA and embryonic expression analysis of zebrafish IRF6, the gene mutated in the human oral clefting disorders Van der Woude and popliteal pterygium syndromes. Ben J; Jabs EW; Chong SS Gene Expr Patterns; 2005 Jun; 5(5):629-38. PubMed ID: 15939375 [TBL] [Abstract][Full Text] [Related]
20. Peters'-plus syndrome is a congenital disorder of glycosylation caused by a defect in the beta1,3-glucosyltransferase that modifies thrombospondin type 1 repeats. Heinonen TY; Maki M Ann Med; 2009; 41(1):2-10. PubMed ID: 18720094 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]