BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

191 related articles for article (PubMed ID: 28964968)

  • 1. Dephosphorylation is the mechanism of fibroblast growth factor inhibition of guanylyl cyclase-B.
    Robinson JW; Egbert JR; Davydova J; Schmidt H; Jaffe LA; Potter LR
    Cell Signal; 2017 Dec; 40():222-229. PubMed ID: 28964968
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Prevention of guanylyl cyclase-B dephosphorylation rescues achondroplastic dwarfism.
    Wagner BM; Robinson JW; Lin YW; Lee YC; Kaci N; Legeai-Mallet L; Potter LR
    JCI Insight; 2021 May; 6(9):. PubMed ID: 33784257
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Dephosphorylation of the NPR2 guanylyl cyclase contributes to inhibition of bone growth by fibroblast growth factor.
    Shuhaibar LC; Robinson JW; Vigone G; Shuhaibar NP; Egbert JR; Baena V; Uliasz TF; Kaback D; Yee SP; Feil R; Fisher MC; Dealy CN; Potter LR; Jaffe LA
    Elife; 2017 Dec; 6():. PubMed ID: 29199951
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Phosphatase inhibition by LB-100 enhances BMN-111 stimulation of bone growth.
    Shuhaibar LC; Kaci N; Egbert JR; Horville T; Loisay L; Vigone G; Uliasz TF; Dambroise E; Swingle MR; Honkanen RE; Biosse Duplan M; Jaffe LA; Legeai-Mallet L
    JCI Insight; 2021 May; 6(9):. PubMed ID: 33986191
    [TBL] [Abstract][Full Text] [Related]  

  • 5. A loss-of-function mutation in natriuretic peptide receptor 2 (Npr2) gene is responsible for disproportionate dwarfism in cn/cn mouse.
    Tsuji T; Kunieda T
    J Biol Chem; 2005 Apr; 280(14):14288-92. PubMed ID: 15722353
    [TBL] [Abstract][Full Text] [Related]  

  • 6. A human skeletal overgrowth mutation increases maximal velocity and blocks desensitization of guanylyl cyclase-B.
    Robinson JW; Dickey DM; Miura K; Michigami T; Ozono K; Potter LR
    Bone; 2013 Oct; 56(2):375-82. PubMed ID: 23827346
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Catalytically Active Guanylyl Cyclase B Requires Endoplasmic Reticulum-mediated Glycosylation, and Mutations That Inhibit This Process Cause Dwarfism.
    Dickey DM; Edmund AB; Otto NM; Chaffee TS; Robinson JW; Potter LR
    J Biol Chem; 2016 May; 291(21):11385-93. PubMed ID: 26980729
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Skeletal overgrowth-causing mutations mimic an allosterically activated conformation of guanylyl cyclase-B that is inhibited by 2,4,6,-trinitrophenyl ATP.
    Dickey DM; Otto NM; Potter LR
    J Biol Chem; 2017 Jun; 292(24):10220-10229. PubMed ID: 28450398
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The indolocarbazole, Gö6976, inhibits guanylyl cyclase-A and -B.
    Robinson JW; Lou X; Potter LR
    Br J Pharmacol; 2011 Sep; 164(2b):499-506. PubMed ID: 21366551
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Sustained phosphorylation of mutated FGFR3 is a crucial feature of genetic dwarfism and induces apoptosis in the ATDC5 chondrogenic cell line via PLCgamma-activated STAT1.
    Harada D; Yamanaka Y; Ueda K; Nishimura R; Morishima T; Seino Y; Tanaka H
    Bone; 2007 Aug; 41(2):273-81. PubMed ID: 17561467
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Reciprocal antagonism coordinates C-type natriuretic peptide and mitogen-signaling pathways in fibroblasts.
    Chrisman TD; Garbers DL
    J Biol Chem; 1999 Feb; 274(7):4293-9. PubMed ID: 9933630
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Complementary antagonistic actions between C-type natriuretic peptide and the MAPK pathway through FGFR-3 in ATDC5 cells.
    Ozasa A; Komatsu Y; Yasoda A; Miura M; Sakuma Y; Nakatsuru Y; Arai H; Itoh N; Nakao K
    Bone; 2005 Jun; 36(6):1056-64. PubMed ID: 15869918
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Activation of protein kinase C stimulates the dephosphorylation of natriuretic peptide receptor-B at a single serine residue: a possible mechanism of heterologous desensitization.
    Potter LR; Hunter T
    J Biol Chem; 2000 Oct; 275(40):31099-106. PubMed ID: 10915802
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Stabilization of Perivascular Mast Cells by Endothelial CNP (C-Type Natriuretic Peptide).
    Chen W; Werner F; Illerhaus A; Knopp T; Völker K; Potapenko T; Hofmann U; Frantz S; Baba HA; Rösch M; Zernecke A; Karbach S; Wenzel P; Kuhn M
    Arterioscler Thromb Vasc Biol; 2020 Mar; 40(3):682-696. PubMed ID: 31893950
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Dephosphorylation and inactivation of NPR2 guanylyl cyclase in granulosa cells contributes to the LH-induced decrease in cGMP that causes resumption of meiosis in rat oocytes.
    Egbert JR; Shuhaibar LC; Edmund AB; Van Helden DA; Robinson JW; Uliasz TF; Baena V; Geerts A; Wunder F; Potter LR; Jaffe LA
    Development; 2014 Sep; 141(18):3594-604. PubMed ID: 25183874
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Dephosphorylation of juxtamembrane serines and threonines of the NPR2 guanylyl cyclase is required for rapid resumption of oocyte meiosis in response to luteinizing hormone.
    Shuhaibar LC; Egbert JR; Edmund AB; Uliasz TF; Dickey DM; Yee SP; Potter LR; Jaffe LA
    Dev Biol; 2016 Jan; 409(1):194-201. PubMed ID: 26522847
    [TBL] [Abstract][Full Text] [Related]  

  • 17. The pseudokinase domains of guanylyl cyclase-A and -B allosterically increase the affinity of their catalytic domains for substrate.
    Edmund AB; Walseth TF; Levinson NM; Potter LR
    Sci Signal; 2019 Jan; 12(566):. PubMed ID: 30696704
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Lysophosphatidic acid inhibits C-type natriuretic peptide activation of guanylyl cyclase-B.
    Abbey SE; Potter LR
    Endocrinology; 2003 Jan; 144(1):240-6. PubMed ID: 12488350
    [TBL] [Abstract][Full Text] [Related]  

  • 19. C-type natriuretic Peptide down-regulates expression of its cognate receptor in rat aortic smooth muscle cells.
    Rahmutula D; Gardner DG
    Endocrinology; 2005 Nov; 146(11):4968-74. PubMed ID: 16109786
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Expression of guanylyl cyclase-B (GC-B/NPR2) receptors in normal human fetal pituitaries and human pituitary adenomas implicates a role for C-type natriuretic peptide.
    Thompson IR; Chand AN; King PJ; Ansorge O; Karavitaki N; Jones CA; Rahmutula D; Gardner DG; Zivkovic V; Wheeler-Jones CP; McGonnell IM; Korbonits M; Anderson RA; Wass JA; McNeilly AS; Fowkes RC
    Endocr Relat Cancer; 2012 Aug; 19(4):497-508. PubMed ID: 22645228
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.