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9. Partial podocyte replenishment in experimental FSGS derives from nonpodocyte sources. Kaverina NV; Eng DG; Schneider RR; Pippin JW; Shankland SJ Am J Physiol Renal Physiol; 2016 Jun; 310(11):F1397-413. PubMed ID: 27076646 [TBL] [Abstract][Full Text] [Related]
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13. The direction and role of phenotypic transition between podocytes and parietal epithelial cells in focal segmental glomerulosclerosis. Sakamoto K; Ueno T; Kobayashi N; Hara S; Takashima Y; Pastan I; Matsusaka T; Nagata M Am J Physiol Renal Physiol; 2014 Jan; 306(1):F98-F104. PubMed ID: 24154691 [TBL] [Abstract][Full Text] [Related]
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16. Phenotypic change of glomerular podocytes in primary focal segmental glomerulosclerosis: developmental paradigm? Ohtaka A; Ootaka T; Sato H; Ito S Nephrol Dial Transplant; 2002; 17 Suppl 9():11-5. PubMed ID: 12386275 [TBL] [Abstract][Full Text] [Related]
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19. Charting the transcriptional landscape of cells of renin lineage following podocyte depletion. McClelland AD; Lichtnekert J; Eng DG; Pippin JW; Gross KW; Gharib SA; Shankland SJ PLoS One; 2017; 12(12):e0189084. PubMed ID: 29232382 [TBL] [Abstract][Full Text] [Related]
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