141 related articles for article (PubMed ID: 28990565)
1. [Calpain as a new therapeutic target for treating spasticity after a spinal cord injury].
Plantier V; Brocard F
Med Sci (Paris); 2017; 33(6-7):629-636. PubMed ID: 28990565
[TBL] [Abstract][Full Text] [Related]
2. Calpain fosters the hyperexcitability of motoneurons after spinal cord injury and leads to spasticity.
Plantier V; Sanchez-Brualla I; Dingu N; Brocard C; Liabeuf S; Gackière F; Brocard F
Elife; 2019 Dec; 8():. PubMed ID: 31815668
[TBL] [Abstract][Full Text] [Related]
3. Cleavage of Na(+) channels by calpain increases persistent Na(+) current and promotes spasticity after spinal cord injury.
Brocard C; Plantier V; Boulenguez P; Liabeuf S; Bouhadfane M; Viallat-Lieutaud A; Vinay L; Brocard F
Nat Med; 2016 Apr; 22(4):404-11. PubMed ID: 26974309
[TBL] [Abstract][Full Text] [Related]
4. Knockdown of calpain1 in lumbar motoneurons reduces spasticity after spinal cord injury in adult rats.
Kerzonkuf M; Verneuil J; Brocard C; Dingu N; Trouplin V; Ramirez Franco JJ; Bartoli M; Brocard F; Bras H
Mol Ther; 2024 Apr; 32(4):1096-1109. PubMed ID: 38291756
[TBL] [Abstract][Full Text] [Related]
5. Prochlorperazine Increases KCC2 Function and Reduces Spasticity after Spinal Cord Injury.
Liabeuf S; Stuhl-Gourmand L; Gackière F; Mancuso R; Sanchez Brualla I; Marino P; Brocard F; Vinay L
J Neurotrauma; 2017 Dec; 34(24):3397-3406. PubMed ID: 28747093
[TBL] [Abstract][Full Text] [Related]
6. Multi-session transcutaneous spinal cord stimulation prevents chloride homeostasis imbalance and the development of hyperreflexia after spinal cord injury in rat.
Malloy DC; Côté MP
Exp Neurol; 2024 Jun; 376():114754. PubMed ID: 38493983
[TBL] [Abstract][Full Text] [Related]
7. Furosemide Unmasks Inhibitory Dysfunction after Spinal Cord Injury in Humans: Implications for Spasticity.
Klomjai W; Roche N; Lamy JC; Bede P; Giron A; Bussel B; Bensmail D; Katz R; Lackmy-Vallée A
J Neurotrauma; 2019 May; 36(9):1469-1477. PubMed ID: 30417726
[TBL] [Abstract][Full Text] [Related]
8. Management of spasticity after spinal cord injury: current techniques and future directions.
Elbasiouny SM; Moroz D; Bakr MM; Mushahwar VK
Neurorehabil Neural Repair; 2010 Jan; 24(1):23-33. PubMed ID: 19723923
[TBL] [Abstract][Full Text] [Related]
9. Rehabilitation Decreases Spasticity by Restoring Chloride Homeostasis through the Brain-Derived Neurotrophic Factor-KCC2 Pathway after Spinal Cord Injury.
Beverungen H; Klaszky SC; Klaszky M; Côté MP
J Neurotrauma; 2020 Mar; 37(6):846-859. PubMed ID: 31578924
[TBL] [Abstract][Full Text] [Related]
10. BDNF Induced by Treadmill Training Contributes to the Suppression of Spasticity and Allodynia After Spinal Cord Injury via Upregulation of KCC2.
Tashiro S; Shinozaki M; Mukaino M; Renault-Mihara F; Toyama Y; Liu M; Nakamura M; Okano H
Neurorehabil Neural Repair; 2015 Aug; 29(7):677-89. PubMed ID: 25527489
[TBL] [Abstract][Full Text] [Related]
11. Alteration of glycinergic receptor expression in lumbar spinal motoneurons is involved in the mechanisms underlying spasticity after spinal cord injury.
Sadlaoud K; Khalki L; Brocard F; Vinay L; Boulenguez P; Bras H
J Chem Neuroanat; 2020 Jul; 106():101787. PubMed ID: 32339654
[TBL] [Abstract][Full Text] [Related]
12. The time course of serotonin 2A receptor expression after spinal transection of rats: an immunohistochemical study.
Kong XY; Wienecke J; Chen M; Hultborn H; Zhang M
Neuroscience; 2011 Mar; 177():114-26. PubMed ID: 21211552
[TBL] [Abstract][Full Text] [Related]
13. Enhancing KCC2 activity decreases hyperreflexia and spasticity after chronic spinal cord injury.
Bilchak JN; Yeakle K; Caron G; Malloy D; Côté MP
Exp Neurol; 2021 Apr; 338():113605. PubMed ID: 33453210
[TBL] [Abstract][Full Text] [Related]
14. Spinal cord stimulation for the control of spasticity in patients with chronic spinal cord injury: I. Clinical observations.
Dimitrijevic MM; Dimitrijevic MR; Illis LS; Nakajima K; Sharkey PC; Sherwood AM
Cent Nerv Syst Trauma; 1986; 3(2):129-44. PubMed ID: 3490312
[TBL] [Abstract][Full Text] [Related]
15. Suppressing the excitability of spinal motoneurons by extracellularly applied electrical fields: insights from computer simulations.
Elbasiouny SM; Mushahwar VK
J Appl Physiol (1985); 2007 Nov; 103(5):1824-36. PubMed ID: 17702836
[TBL] [Abstract][Full Text] [Related]
16. Global gene expression analysis of rodent motor neurons following spinal cord injury associates molecular mechanisms with development of postinjury spasticity.
Wienecke J; Westerdahl AC; Hultborn H; Kiehn O; Ryge J
J Neurophysiol; 2010 Feb; 103(2):761-78. PubMed ID: 19939961
[TBL] [Abstract][Full Text] [Related]
17. Down-regulation of the potassium-chloride cotransporter KCC2 contributes to spasticity after spinal cord injury.
Boulenguez P; Liabeuf S; Bos R; Bras H; Jean-Xavier C; Brocard C; Stil A; Darbon P; Cattaert D; Delpire E; Marsala M; Vinay L
Nat Med; 2010 Mar; 16(3):302-7. PubMed ID: 20190766
[TBL] [Abstract][Full Text] [Related]
18. Combination of repetitive transcranial magnetic stimulation and treadmill training reduces hyperreflexia by rebalancing motoneuron excitability in rats after spinal cord contusion.
Wang S; Wang P; Yin R; Xiao M; Zhang Y; Reinhardt JD; Wang H; Xu G
Neurosci Lett; 2022 Apr; 775():136536. PubMed ID: 35183693
[TBL] [Abstract][Full Text] [Related]
19. Modulation of motoneuronal firing behavior after spinal cord injury using intraspinal microstimulation current pulses: a modeling study.
Elbasiouny SM; Mushahwar VK
J Appl Physiol (1985); 2007 Jul; 103(1):276-86. PubMed ID: 17234800
[TBL] [Abstract][Full Text] [Related]
20. Repeated anodal trans-spinal direct current stimulation results in long-term reduction of spasticity in mice with spinal cord injury.
Mekhael W; Begum S; Samaddar S; Hassan M; Toruno P; Ahmed M; Gorin A; Maisano M; Ayad M; Ahmed Z
J Physiol; 2019 Apr; 597(8):2201-2223. PubMed ID: 30689208
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
