133 related articles for article (PubMed ID: 2918368)
1. The two major phosphoproteins in growth cones are probably identical to two protein kinase C substrates correlated with persistence of long-term potentiation.
Nelson RB; Linden DJ; Hyman C; Pfenninger KH; Routtenberg A
J Neurosci; 1989 Feb; 9(2):381-9. PubMed ID: 2918368
[TBL] [Abstract][Full Text] [Related]
2. Phosphoprotein F1: purification and characterization of a brain kinase C substrate related to plasticity.
Chan SY; Murakami K; Routtenberg A
J Neurosci; 1986 Dec; 6(12):3618-27. PubMed ID: 3794793
[TBL] [Abstract][Full Text] [Related]
3. Phosphoproteins localized to presynaptic terminal linked to persistence of long-term potentiation (LTP): quantitative analysis of two-dimensional gels.
Nelson RB; Linden DJ; Routtenberg A
Brain Res; 1989 Sep; 497(1):30-42. PubMed ID: 2790456
[TBL] [Abstract][Full Text] [Related]
4. Direct relation of long-term synaptic potentiation to phosphorylation of membrane protein F1, a substrate for membrane protein kinase C.
Lovinger DM; Colley PA; Akers RF; Nelson RB; Routtenberg A
Brain Res; 1986 Dec; 399(2):205-11. PubMed ID: 3828760
[TBL] [Abstract][Full Text] [Related]
5. Characterization of protein F1 (47 kDa, 4.5 pI): a kinase C substrate directly related to neural plasticity.
Nelson RB; Routtenberg A
Exp Neurol; 1985 Jul; 89(1):213-24. PubMed ID: 3159591
[TBL] [Abstract][Full Text] [Related]
6. Calcium-promoted translocation of protein kinase C to synaptic membranes: relation to the phosphorylation of an endogenous substrate (protein F1) involved in synaptic plasticity.
Akers RF; Routtenberg A
J Neurosci; 1987 Dec; 7(12):3976-83. PubMed ID: 3121805
[TBL] [Abstract][Full Text] [Related]
7. Membrane proteins of the nerve growth cone and their developmental regulation.
Simkowitz P; Ellis L; Pfenninger KH
J Neurosci; 1989 Mar; 9(3):1004-17. PubMed ID: 2926476
[TBL] [Abstract][Full Text] [Related]
8. Time course and involvement of protein kinase C-mediated phosphorylation of F1/GAP-43 in area CA3 after mossy fiber stimulation.
Son H; Davis PJ; Carpenter DO
Cell Mol Neurobiol; 1997 Apr; 17(2):171-94. PubMed ID: 9140696
[TBL] [Abstract][Full Text] [Related]
9. Protein kinase C activation leading to protein F1 phosphorylation may regulate synaptic plasticity by presynaptic terminal growth.
Routtenberg A
Behav Neural Biol; 1985 Sep; 44(2):186-200. PubMed ID: 3904711
[TBL] [Abstract][Full Text] [Related]
10. Protein kinase C phosphorylates a 47 Mr protein (F1) directly related to synaptic plasticity.
Akers RF; Routtenberg A
Brain Res; 1985 May; 334(1):147-51. PubMed ID: 3158377
[TBL] [Abstract][Full Text] [Related]
11. Increased phosphorylation of a 17-kDa protein kinase C substrate (P17) in long-term potentiation.
Klann E; Chen SJ; Sweatt JD
J Neurochem; 1992 Apr; 58(4):1576-9. PubMed ID: 1548487
[TBL] [Abstract][Full Text] [Related]
12. Arachidonic acid, but not sodium nitroprusside, stimulates presynaptic protein kinase C and phosphorylation of GAP-43 in rat hippocampal slices and synaptosomes.
Luo Y; Vallano ML
J Neurochem; 1995 Apr; 64(4):1808-18. PubMed ID: 7891109
[TBL] [Abstract][Full Text] [Related]
13. NMDA receptor blockade prevents the increase in protein kinase C substrate (protein F1) phosphorylation produced by long-term potentiation.
Linden DJ; Wong KL; Sheu FS; Routtenberg A
Brain Res; 1988 Aug; 458(1):142-6. PubMed ID: 2905192
[TBL] [Abstract][Full Text] [Related]
14. Neuron-specific protein F1/GAP-43 shows substrate specificity for the beta subtype of protein kinase C.
Sheu FS; Marais RM; Parker PJ; Bazan NG; Routtenberg A
Biochem Biophys Res Commun; 1990 Sep; 171(3):1236-43. PubMed ID: 2145833
[TBL] [Abstract][Full Text] [Related]
15. Inhibition of protein kinase C blocks two components of LTP persistence, leaving initial potentiation intact.
Colley PA; Sheu FS; Routtenberg A
J Neurosci; 1990 Oct; 10(10):3353-60. PubMed ID: 2213144
[TBL] [Abstract][Full Text] [Related]
16. Contrasting patterns of protein phosphorylation in human normal and Alzheimer brain: focus on protein kinase C and protein F1/GAP-43.
Florez JC; Nelson RB; Routtenberg A
Exp Neurol; 1991 Jun; 112(3):264-72. PubMed ID: 1827625
[TBL] [Abstract][Full Text] [Related]
17. Long-term potentiation and synaptic protein phosphorylation.
Pasinelli P; Ramakers GM; Urban IJ; Hens JJ; Oestreicher AB; de Graan PN; Gispen WH
Behav Brain Res; 1995 Jan; 66(1-2):53-9. PubMed ID: 7755899
[TBL] [Abstract][Full Text] [Related]
18. Selective decline in protein F1 phosphorylation in hippocampus of senescent rats.
Barnes CA; Mizumori SJ; Lovinger DM; Sheu FS; Murakami K; Chan SY; Linden DJ; Nelson RB; Routtenberg A
Neurobiol Aging; 1988; 9(4):393-8. PubMed ID: 3185858
[TBL] [Abstract][Full Text] [Related]
19. Dephosphin, a 96,000 Da substrate of protein kinase C in synaptosomal cytosol, is phosphorylated in intact synaptosomes.
Robinson PJ
FEBS Lett; 1991 May; 282(2):388-92. PubMed ID: 2037055
[TBL] [Abstract][Full Text] [Related]
20. Activation of protein kinase C by arachidonic acid selectively enhances the phosphorylation of GAP-43 in nerve terminal membranes.
Schaechter JD; Benowitz LI
J Neurosci; 1993 Oct; 13(10):4361-71. PubMed ID: 8410192
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
