BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

176 related articles for article (PubMed ID: 29243900)

  • 1. The new line of genetically modified mice with constitutive knockout of the gene alpha synuclein to study pathogenetic aspects of differential loss of dopaminergic neurons .
    Tarasova TV; Ustyugov AA; Ninkina NN; Skvortsova VI
    Patol Fiziol Eksp Ter; 2016; 60(3):4-9. PubMed ID: 29243900
    [TBL] [Abstract][Full Text] [Related]  

  • 2. The role of alpha-synuclein in the development of the dopaminergic neurons in the substantia nigra and ventral tegmental area.
    Tarasova TV; Lytkina OA; Roman AY; Bachurin SO; Ustyugov AA
    Dokl Biol Sci; 2016; 466():5-7. PubMed ID: 27021360
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Aldehyde dehydrogenase 1 defines and protects a nigrostriatal dopaminergic neuron subpopulation.
    Liu G; Yu J; Ding J; Xie C; Sun L; Rudenko I; Zheng W; Sastry N; Luo J; Rudow G; Troncoso JC; Cai H
    J Clin Invest; 2014 Jul; 124(7):3032-46. PubMed ID: 24865427
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Ventral tegmental area dopamine neurons are resistant to human mutant alpha-synuclein overexpression.
    Maingay M; Romero-Ramos M; Carta M; Kirik D
    Neurobiol Dis; 2006 Sep; 23(3):522-32. PubMed ID: 16806952
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Reduced complement of dopaminergic neurons in the substantia nigra pars compacta of mice with a constitutive "low footprint" genetic knockout of alpha-synuclein.
    Goloborshcheva VV; Chaprov KD; Teterina EV; Ovchinnikov R; Buchman VL
    Mol Brain; 2020 May; 13(1):75. PubMed ID: 32393371
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Rab1A over-expression prevents Golgi apparatus fragmentation and partially corrects motor deficits in an alpha-synuclein based rat model of Parkinson's disease.
    Coune PG; Bensadoun JC; Aebischer P; Schneider BL
    J Parkinsons Dis; 2011; 1(4):373-87. PubMed ID: 23939344
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Aquaporin-4 deficiency diminishes the differential degeneration of midbrain dopaminergic neurons in experimental Parkinson's disease.
    Zhang J; Yang B; Sun H; Zhou Y; Liu M; Ding J; Fang F; Fan Y; Hu G
    Neurosci Lett; 2016 Feb; 614():7-15. PubMed ID: 26748031
    [TBL] [Abstract][Full Text] [Related]  

  • 8. α-Synuclein-Dependent Calcium Entry Underlies Differential Sensitivity of Cultured SN and VTA Dopaminergic Neurons to a Parkinsonian Neurotoxin.
    Lieberman OJ; Choi SJ; Kanter E; Saverchenko A; Frier MD; Fiore GM; Wu M; Kondapalli J; Zampese E; Surmeier DJ; Sulzer D; Mosharov EV
    eNeuro; 2017; 4(6):. PubMed ID: 29177188
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Glucocerebrosidase deficiency in dopaminergic neurons induces microglial activation without neurodegeneration.
    Soria FN; Engeln M; Martinez-Vicente M; Glangetas C; López-González MJ; Dovero S; Dehay B; Normand E; Vila M; Favereaux A; Georges F; Lo Bianco C; Bezard E; Fernagut PO
    Hum Mol Genet; 2017 Jul; 26(14):2603-2615. PubMed ID: 28520872
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Lewy body extracts from Parkinson disease brains trigger α-synuclein pathology and neurodegeneration in mice and monkeys.
    Recasens A; Dehay B; Bové J; Carballo-Carbajal I; Dovero S; Pérez-Villalba A; Fernagut PO; Blesa J; Parent A; Perier C; Fariñas I; Obeso JA; Bezard E; Vila M
    Ann Neurol; 2014 Mar; 75(3):351-62. PubMed ID: 24243558
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Selective α-Synuclein Knockdown in Monoamine Neurons by Intranasal Oligonucleotide Delivery: Potential Therapy for Parkinson's Disease.
    Alarcón-Arís D; Recasens A; Galofré M; Carballo-Carbajal I; Zacchi N; Ruiz-Bronchal E; Pavia-Collado R; Chica R; Ferrés-Coy A; Santos M; Revilla R; Montefeltro A; Fariñas I; Artigas F; Vila M; Bortolozzi A
    Mol Ther; 2018 Feb; 26(2):550-567. PubMed ID: 29273501
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Inclusion of a portion of the native SNCA 3'UTR reduces toxicity of human S129A SNCA on striatal-projecting dopamine neurons in rat substantia nigra.
    Khodr CE; Pedapati J; Han Y; Bohn MC
    Dev Neurobiol; 2012 Jun; 72(6):906-17. PubMed ID: 22021082
    [TBL] [Abstract][Full Text] [Related]  

  • 13. K-ATP channels promote the differential degeneration of dopaminergic midbrain neurons.
    Liss B; Haeckel O; Wildmann J; Miki T; Seino S; Roeper J
    Nat Neurosci; 2005 Dec; 8(12):1742-51. PubMed ID: 16299504
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Dopamine and α-synuclein dysfunction in Smad3 null mice.
    Tapia-González S; Giráldez-Pérez RM; Cuartero MI; Casarejos MJ; Mena MÁ; Wang XF; Sánchez-Capelo A
    Mol Neurodegener; 2011 Oct; 6():72. PubMed ID: 21995845
    [TBL] [Abstract][Full Text] [Related]  

  • 15. CNTNAP4 deficiency in dopaminergic neurons initiates parkinsonian phenotypes.
    Zhang W; Zhou M; Lu W; Gong J; Gao F; Li Y; Xu X; Lin Y; Zhang X; Ding L; Zhang Z; Li G; Chen X; Sun X; Zhu X; Xu P; Zhang Y
    Theranostics; 2020; 10(7):3000-3021. PubMed ID: 32194851
    [No Abstract]   [Full Text] [Related]  

  • 16. Mutant α-synuclein enhances firing frequencies in dopamine substantia nigra neurons by oxidative impairment of A-type potassium channels.
    Subramaniam M; Althof D; Gispert S; Schwenk J; Auburger G; Kulik A; Fakler B; Roeper J
    J Neurosci; 2014 Oct; 34(41):13586-99. PubMed ID: 25297088
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Genetic inactivation of alpha-synuclein affects embryonic development of dopaminergic neurons of the substantia nigra, but not the ventral tegmental area, in mouse brain.
    Tarasova TV; Lytkina OA; Goloborshcheva VV; Skuratovskaya LN; Antohin AI; Ovchinnikov RK; Kukharsky MS
    PeerJ; 2018; 6():e4779. PubMed ID: 29785351
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Sigma-1 receptor knockout increases α-synuclein aggregation and phosphorylation with loss of dopaminergic neurons in substantia nigra.
    Hong J; Wang L; Zhang T; Zhang B; Chen L
    Neurobiol Aging; 2017 Nov; 59():171-183. PubMed ID: 28870519
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Motor deficits and beta oscillations are dissociable in an alpha-synuclein model of Parkinson's disease.
    Brys I; Nunes J; Fuentes R
    Eur J Neurosci; 2017 Aug; 46(3):1906-1917. PubMed ID: 28370471
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Thiol Oxidation by Diamide Leads to Dopaminergic Degeneration and Parkinsonism Phenotype in Mice: A Model for Parkinson's Disease.
    Ray A; Kambali M; Ravindranath V
    Antioxid Redox Signal; 2016 Aug; 25(5):252-67. PubMed ID: 27121974
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.