340 related articles for article (PubMed ID: 29286079)
1. Exogenous hydrogen sulfide protects human umbilical vein endothelial cells against high glucose‑induced injury by inhibiting the necroptosis pathway.
Lin J; Chen M; Liu D; Guo R; Lin K; Deng H; Zhi X; Zhang W; Feng J; Wu W
Int J Mol Med; 2018 Mar; 41(3):1477-1486. PubMed ID: 29286079
[TBL] [Abstract][Full Text] [Related]
2. Exogenous sodium hydrosulfide protects against high glucose‑induced injury and inflammation in human umbilical vein endothelial cells by inhibiting necroptosis via the p38 MAPK signaling pathway.
Lin J; Li X; Lin Y; Huang Z; Wu W
Mol Med Rep; 2021 Jan; 23(1):. PubMed ID: 33215220
[TBL] [Abstract][Full Text] [Related]
3. Hydrogen Sulfide Protects Against High Glucose-Induced Human Umbilical Vein Endothelial Cell Injury Through Activating PI3K/Akt/eNOS Pathway.
Lin F; Yang Y; Wei S; Huang X; Peng Z; Ke X; Zeng Z; Song Y
Drug Des Devel Ther; 2020; 14():621-633. PubMed ID: 32103904
[TBL] [Abstract][Full Text] [Related]
4. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
Xu W; Chen J; Lin J; Liu D; Mo L; Pan W; Feng J; Wu W; Zheng D
Int J Mol Med; 2015 Jan; 35(1):177-86. PubMed ID: 25412187
[TBL] [Abstract][Full Text] [Related]
5. The Opening of ATP-Sensitive K+ Channels Protects H9c2 Cardiac Cells Against the High Glucose-Induced Injury and Inflammation by Inhibiting the ROS-TLR4-Necroptosis Pathway.
Liang W; Chen M; Zheng D; Li J; Song M; Zhang W; Feng J; Lan J
Cell Physiol Biochem; 2017; 41(3):1020-1034. PubMed ID: 28291959
[TBL] [Abstract][Full Text] [Related]
6. Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.
Xu W; Wu W; Chen J; Guo R; Lin J; Liao X; Feng J
Int J Mol Med; 2013 Oct; 32(4):917-25. PubMed ID: 23912965
[TBL] [Abstract][Full Text] [Related]
7. Alterations in necroptosis during ALDH2‑mediated protection against high glucose‑induced H9c2 cardiac cell injury.
Fang T; Cao R; Wang W; Ye H; Shen L; Li Z; Hu J; Gao Q
Mol Med Rep; 2018 Sep; 18(3):2807-2815. PubMed ID: 30015964
[TBL] [Abstract][Full Text] [Related]
8. ATP-sensitive K⁺ channels contribute to the protective effects of exogenous hydrogen sulfide against high glucose-induced injury in H9c2 cardiac cells.
Liang W; Chen J; Mo L; Ke X; Zhang W; Zheng D; Pan W; Wu S; Feng J; Song M; Liao X
Int J Mol Med; 2016 Mar; 37(3):763-72. PubMed ID: 26820501
[TBL] [Abstract][Full Text] [Related]
9. [Exogenous hydrogen sulfide inhibits high-glucose-induced injuries via regulating leptin/leptin receptor signaling pathway in human umbilical vein endothelial cells].
Wu DB; Chen JF; Xu Q; Lin JQ; Liao JQ; Wu W
Nan Fang Yi Ke Da Xue Xue Bao; 2016 Aug; 36(8):1055-61. PubMed ID: 27578572
[TBL] [Abstract][Full Text] [Related]
10. Exogenous hydrogen sulfide protects against high glucose‑induced inflammation and cytotoxicity in H9c2 cardiac cells.
Huang Z; Dong X; Zhuang X; Hu X; Wang L; Liao X
Mol Med Rep; 2016 Nov; 14(5):4911-4917. PubMed ID: 27748941
[TBL] [Abstract][Full Text] [Related]
11. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibition of the p38 MAPK pathway in H9c2 cells.
Guo R; Lin J; Xu W; Shen N; Mo L; Zhang C; Feng J
Int J Mol Med; 2013 Mar; 31(3):644-50. PubMed ID: 23338126
[TBL] [Abstract][Full Text] [Related]
12. Ang-(1-7) protects HUVECs from high glucose-induced injury and inflammation via inhibition of the JAK2/STAT3 pathway.
Chen J; Zhang W; Xu Q; Zhang J; Chen W; Xu Z; Li C; Wang Z; Zhang Y; Zhen Y; Feng J; Chen J; Chen J
Int J Mol Med; 2018 May; 41(5):2865-2878. PubMed ID: 29484371
[TBL] [Abstract][Full Text] [Related]
13. Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.
Zhuang XD; Hu X; Long M; Dong XB; Liu DH; Liao XX
Mol Cell Biochem; 2014 Jun; 391(1-2):147-55. PubMed ID: 24687304
[TBL] [Abstract][Full Text] [Related]
14. Exogenous hydrogen sulfide ameliorates high glucose-induced myocardial injury & inflammation via the CIRP-MAPK signaling pathway in H9c2 cardiac cells.
Zhao HL; Wu BQ; Luo Y; Zhang WY; Hao YL; Liang JJ; Fang F; Liu W; Chen XH
Life Sci; 2018 Sep; 208():315-324. PubMed ID: 29857073
[TBL] [Abstract][Full Text] [Related]
15. Necrostatin‑1 protects mice from acute lung injury by suppressing necroptosis and reactive oxygen species.
Lin B; Jin Z; Chen X; Zhao L; Weng C; Chen B; Tang Y; Lin L
Mol Med Rep; 2020 May; 21(5):2171-2181. PubMed ID: 32323764
[TBL] [Abstract][Full Text] [Related]
16. Ezetimibe Protects Endothelial Cells against Oxidative Stress through Akt/GSK-3β Pathway.
Qin J; Wang LL; Liu ZY; Zou YL; Fei YJ; Liu ZX
Curr Med Sci; 2018 Jun; 38(3):398-404. PubMed ID: 30074204
[TBL] [Abstract][Full Text] [Related]
17. Klotho attenuates angiotensin II‑induced cardiotoxicity through suppression of necroptosis and oxidative stress.
Yu S; Yang H; Guo X; Sun Y
Mol Med Rep; 2021 Jan; 23(1):. PubMed ID: 33215215
[TBL] [Abstract][Full Text] [Related]
18. Exogenous Hydrogen Sulfide Attenuates High Glucose-Induced Cardiotoxicity by Inhibiting NLRP3 Inflammasome Activation by Suppressing TLR4/NF-κB Pathway in H9c2 Cells.
Huang Z; Zhuang X; Xie C; Hu X; Dong X; Guo Y; Li S; Liao X
Cell Physiol Biochem; 2016; 40(6):1578-1590. PubMed ID: 27997926
[TBL] [Abstract][Full Text] [Related]
19. Epigallocatechin-3-Gallate Ameliorates Angiotensin II-Induced Oxidative Stress and Apoptosis in Human Umbilical Vein Endothelial Cells through the Activation of Nrf2/Caspase-3 Signaling.
Zhou X; Liang L; Zhao Y; Zhang H
J Vasc Res; 2017; 54(5):299-308. PubMed ID: 28942440
[TBL] [Abstract][Full Text] [Related]
20. 2,3,5,4'-tetrahydroxystilbene-2-O-β-D-glucoside protects human umbilical vein endothelial cells against lysophosphatidylcholine-induced apoptosis by upregulating superoxide dismutase and glutathione peroxidase.
Zhao J; Xu S; Song F; Nian L; Zhou X; Wang S
IUBMB Life; 2014 Oct; 66(10):711-22. PubMed ID: 25382724
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
