815 related articles for article (PubMed ID: 29524628)
1. Assessment of metal concentrations in the SOD1
Enge TG; Ecroyd H; Jolley DF; Yerbury JJ; Kalmar B; Dosseto A
Mol Cell Neurosci; 2018 Apr; 88():319-329. PubMed ID: 29524628
[TBL] [Abstract][Full Text] [Related]
2. Longitudinal assessment of metal concentrations and copper isotope ratios in the G93A SOD1 mouse model of amyotrophic lateral sclerosis.
Enge TG; Ecroyd H; Jolley DF; Yerbury JJ; Dosseto A
Metallomics; 2017 Feb; 9(2):161-174. PubMed ID: 28067393
[TBL] [Abstract][Full Text] [Related]
3. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
Beqollari D; Romberg CF; Dobrowolny G; Martini M; Voss AA; Musarò A; Bannister RA
Skelet Muscle; 2016; 6():24. PubMed ID: 27340545
[TBL] [Abstract][Full Text] [Related]
4. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
[TBL] [Abstract][Full Text] [Related]
5. Differential effects of mutant SOD1 on protein structure of skeletal muscle and spinal cord of familial amyotrophic lateral sclerosis: role of chaperone network.
Wei R; Bhattacharya A; Hamilton RT; Jernigan AL; Chaudhuri AR
Biochem Biophys Res Commun; 2013 Aug; 438(1):218-23. PubMed ID: 23886956
[TBL] [Abstract][Full Text] [Related]
6. Galectin-1 deficiency improves axonal swelling of motor neurones in SOD1(G93A) transgenic mice.
Kobayakawa Y; Sakumi K; Kajitani K; Kadoya T; Horie H; Kira J; Nakabeppu Y
Neuropathol Appl Neurobiol; 2015 Feb; 41(2):227-44. PubMed ID: 24707896
[TBL] [Abstract][Full Text] [Related]
7. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
Milanese M; Giribaldi F; Melone M; Bonifacino T; Musante I; Carminati E; Rossi PI; Vergani L; Voci A; Conti F; Puliti A; Bonanno G
Neurobiol Dis; 2014 Apr; 64():48-59. PubMed ID: 24361555
[TBL] [Abstract][Full Text] [Related]
8. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
Li QX; Mok SS; Laughton KM; McLean CA; Volitakis I; Cherny RA; Cheung NS; White AR; Masters CL
Aging Cell; 2006 Apr; 5(2):153-65. PubMed ID: 16626394
[TBL] [Abstract][Full Text] [Related]
9. Overexpression of metallothionein-I, a copper-regulating protein, attenuates intracellular copper dyshomeostasis and extends lifespan in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase-1.
Tokuda E; Okawa E; Watanabe S; Ono S
Hum Mol Genet; 2014 Mar; 23(5):1271-85. PubMed ID: 24163136
[TBL] [Abstract][Full Text] [Related]
10. Transcriptomic indices of fast and slow disease progression in two mouse models of amyotrophic lateral sclerosis.
Nardo G; Iennaco R; Fusi N; Heath PR; Marino M; Trolese MC; Ferraiuolo L; Lawrence N; Shaw PJ; Bendotti C
Brain; 2013 Nov; 136(Pt 11):3305-32. PubMed ID: 24065725
[TBL] [Abstract][Full Text] [Related]
11. MTOR-independent, autophagic enhancer trehalose prolongs motor neuron survival and ameliorates the autophagic flux defect in a mouse model of amyotrophic lateral sclerosis.
Zhang X; Chen S; Song L; Tang Y; Shen Y; Jia L; Le W
Autophagy; 2014 Apr; 10(4):588-602. PubMed ID: 24441414
[TBL] [Abstract][Full Text] [Related]
12. Redox proteomics analysis of oxidatively modified proteins in G93A-SOD1 transgenic mice--a model of familial amyotrophic lateral sclerosis.
Poon HF; Hensley K; Thongboonkerd V; Merchant ML; Lynn BC; Pierce WM; Klein JB; Calabrese V; Butterfield DA
Free Radic Biol Med; 2005 Aug; 39(4):453-62. PubMed ID: 16043017
[TBL] [Abstract][Full Text] [Related]
13. Zinc pre-treatment enhances NMDAR-mediated excitotoxicity in cultured cortical neurons from SOD1(G93A) mouse, a model of amyotrophic lateral sclerosis.
Nutini M; Frazzini V; Marini C; Spalloni A; Sensi SL; Longone P
Neuropharmacology; 2011 Jun; 60(7-8):1200-8. PubMed ID: 21056589
[TBL] [Abstract][Full Text] [Related]
14. The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1
Hilton JB; Kysenius K; White AR; Crouch PJ
Exp Neurol; 2018 Sep; 307():118-128. PubMed ID: 29906423
[TBL] [Abstract][Full Text] [Related]
15. Endogenous Cu in the central nervous system fails to satiate the elevated requirement for Cu in a mutant SOD1 mouse model of ALS.
Hilton JB; White AR; Crouch PJ
Metallomics; 2016 Sep; 8(9):1002-11. PubMed ID: 27357743
[TBL] [Abstract][Full Text] [Related]
16. Microglia and motor neurons during disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis: changes in arginase1 and inducible nitric oxide synthase.
Lewis KE; Rasmussen AL; Bennett W; King A; West AK; Chung RS; Chuah MI
J Neuroinflammation; 2014 Mar; 11():55. PubMed ID: 24655927
[TBL] [Abstract][Full Text] [Related]
17. Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
Clark JA; Southam KA; Blizzard CA; King AE; Dickson TC
J Chem Neuroanat; 2016 Oct; 76(Pt A):35-47. PubMed ID: 27038603
[TBL] [Abstract][Full Text] [Related]
18. Trehalose decreases mutant SOD1 expression and alleviates motor deficiency in early but not end-stage amyotrophic lateral sclerosis in a SOD1-G93A mouse model.
Li Y; Guo Y; Wang X; Yu X; Duan W; Hong K; Wang J; Han H; Li C
Neuroscience; 2015 Jul; 298():12-25. PubMed ID: 25841320
[TBL] [Abstract][Full Text] [Related]
19. Muscle-derived but not centrally derived transgene GDNF is neuroprotective in G93A-SOD1 mouse model of ALS.
Li W; Brakefield D; Pan Y; Hunter D; Myckatyn TM; Parsadanian A
Exp Neurol; 2007 Feb; 203(2):457-71. PubMed ID: 17034790
[TBL] [Abstract][Full Text] [Related]
20. Treatment with an antibody directed against Nogo-A delays disease progression in the SOD1G93A mouse model of Amyotrophic lateral sclerosis.
Bros-Facer V; Krull D; Taylor A; Dick JR; Bates SA; Cleveland MS; Prinjha RK; Greensmith L
Hum Mol Genet; 2014 Aug; 23(16):4187-200. PubMed ID: 24667415
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
