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6. Loss of tafazzin results in decreased myoblast differentiation in C2C12 cells: A myoblast model of Barth syndrome and cardiolipin deficiency. Lou W; Reynolds CA; Li Y; Liu J; Hüttemann M; Schlame M; Stevenson D; Strathdee D; Greenberg ML Biochim Biophys Acta Mol Cell Biol Lipids; 2018 Aug; 1863(8):857-865. PubMed ID: 29694924 [TBL] [Abstract][Full Text] [Related]
8. Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy. Hsu P; Liu X; Zhang J; Wang HG; Ye JM; Shi Y Autophagy; 2015 Apr; 11(4):643-52. PubMed ID: 25919711 [TBL] [Abstract][Full Text] [Related]
9. Substantial Decrease in Plasmalogen in the Heart Associated with Tafazzin Deficiency. Kimura T; Kimura AK; Ren M; Berno B; Xu Y; Schlame M; Epand RM Biochemistry; 2018 Apr; 57(14):2162-2175. PubMed ID: 29557170 [TBL] [Abstract][Full Text] [Related]
10. Cardiac metabolic pathways affected in the mouse model of barth syndrome. Huang Y; Powers C; Madala SK; Greis KD; Haffey WD; Towbin JA; Purevjav E; Javadov S; Strauss AW; Khuchua Z PLoS One; 2015; 10(6):e0128561. PubMed ID: 26030409 [TBL] [Abstract][Full Text] [Related]
11. Tafazzin Deficiency Reduces Basal Insulin Secretion and Mitochondrial Function in Pancreatic Islets From Male Mice. Cole LK; Agarwal P; Doucette CA; Fonseca M; Xiang B; Sparagna GC; Seshadri N; Vandel M; Dolinsky VW; Hatch GM Endocrinology; 2021 Jul; 162(7):. PubMed ID: 34019639 [TBL] [Abstract][Full Text] [Related]
12. Cardiolipin remodeling: a regulatory hub for modulating cardiolipin metabolism and function. Ye C; Shen Z; Greenberg ML J Bioenerg Biomembr; 2016 Apr; 48(2):113-23. PubMed ID: 25432572 [TBL] [Abstract][Full Text] [Related]
14. Tafazzin deficiency causes substantial remodeling in the lipidome of a mouse model of Barth Syndrome cardiomyopathy. Hachmann M; Gülcan G; Rajendran R; Höring M; Liebisch G; Bachhuka A; Kohlhaas M; Maack C; Ergün S; Dudek J; Karnati S Front Mol Med; 2024; 4():1389456. PubMed ID: 39086433 [TBL] [Abstract][Full Text] [Related]
15. Cardiolipin Remodeling Defects Impair Mitochondrial Architecture and Function in a Murine Model of Barth Syndrome Cardiomyopathy. Zhu S; Chen Z; Zhu M; Shen Y; Leon LJ; Chi L; Spinozzi S; Tan C; Gu Y; Nguyen A; Zhou Y; Feng W; Vaz FM; Wang X; Gustafsson AB; Evans SM; Kunfu O; Fang X Circ Heart Fail; 2021 Jun; 14(6):e008289. PubMed ID: 34129362 [TBL] [Abstract][Full Text] [Related]
16. Restoration of mitophagy ameliorates cardiomyopathy in Barth syndrome. Zhang J; Liu X; Nie J; Shi Y Autophagy; 2022 Sep; 18(9):2134-2149. PubMed ID: 34985382 [TBL] [Abstract][Full Text] [Related]
17. Cardiolipin-deficient cells have decreased levels of the iron-sulfur biogenesis protein frataxin. Li Y; Lou W; Grevel A; Böttinger L; Liang Z; Ji J; Patil VA; Liu J; Ye C; Hüttemann M; Becker T; Greenberg ML J Biol Chem; 2020 Aug; 295(33):11928-11937. PubMed ID: 32636300 [TBL] [Abstract][Full Text] [Related]
18. Deletion of the cardiolipin-specific phospholipase Cld1 rescues growth and life span defects in the tafazzin mutant: implications for Barth syndrome. Ye C; Lou W; Li Y; Chatzispyrou IA; Hüttemann M; Lee I; Houtkooper RH; Vaz FM; Chen S; Greenberg ML J Biol Chem; 2014 Feb; 289(6):3114-25. PubMed ID: 24318983 [TBL] [Abstract][Full Text] [Related]
19. Decreased pyruvate dehydrogenase activity in Tafazzin-deficient cells is caused by dysregulation of pyruvate dehydrogenase phosphatase 1 (PDP1). Liang Z; Ralph-Epps T; Schmidtke MW; Kumar V; Greenberg ML J Biol Chem; 2024 Mar; 300(3):105697. PubMed ID: 38301889 [TBL] [Abstract][Full Text] [Related]
20. Barth syndrome: cellular compensation of mitochondrial dysfunction and apoptosis inhibition due to changes in cardiolipin remodeling linked to tafazzin (TAZ) gene mutation. Gonzalvez F; D'Aurelio M; Boutant M; Moustapha A; Puech JP; Landes T; Arnauné-Pelloquin L; Vial G; Taleux N; Slomianny C; Wanders RJ; Houtkooper RH; Bellenguer P; Møller IM; Gottlieb E; Vaz FM; Manfredi G; Petit PX Biochim Biophys Acta; 2013 Aug; 1832(8):1194-206. PubMed ID: 23523468 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]