285 related articles for article (PubMed ID: 30348658)
1. Pillars Article: Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme.
Muramatsu M; Kinoshita K; Fagarasan S; Yamada S; Shinkai Y; Honjo T
J Immunol; 2018 Nov; 201(9):2530-2540. PubMed ID: 30348658
[No Abstract] [Full Text] [Related]
2. The Common Key to Class-Switch Recombination and Somatic Hypermutation: Discovery of AID and Its Role in Antibody Gene Diversification.
Leeman-Neill RJ; Lim J; Basu U
J Immunol; 2018 Nov; 201(9):2527-2529. PubMed ID: 30348657
[No Abstract] [Full Text] [Related]
3. DNA targets of AID evolutionary link between antibody somatic hypermutation and class switch recombination.
Hackney JA; Misaghi S; Senger K; Garris C; Sun Y; Lorenzo MN; Zarrin AA
Adv Immunol; 2009; 101():163-89. PubMed ID: 19231595
[TBL] [Abstract][Full Text] [Related]
4. Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme.
Muramatsu M; Kinoshita K; Fagarasan S; Yamada S; Shinkai Y; Honjo T
Cell; 2000 Sep; 102(5):553-63. PubMed ID: 11007474
[TBL] [Abstract][Full Text] [Related]
5. Activation-induced deaminase (AID)-directed hypermutation in the immunoglobulin Smu region: implication of AID involvement in a common step of class switch recombination and somatic hypermutation.
Nagaoka H; Muramatsu M; Yamamura N; Kinoshita K; Honjo T
J Exp Med; 2002 Feb; 195(4):529-34. PubMed ID: 11854365
[TBL] [Abstract][Full Text] [Related]
6. [AID controls immunoglobulin class switch recombination].
Muramatsu M
Seikagaku; 2006 Aug; 78(8):719-24. PubMed ID: 16986721
[No Abstract] [Full Text] [Related]
7. RNA-editing cytidine deaminase Apobec-1 is unable to induce somatic hypermutation in mammalian cells.
Eto T; Kinoshita K; Yoshikawa K; Muramatsu M; Honjo T
Proc Natl Acad Sci U S A; 2003 Oct; 100(22):12895-8. PubMed ID: 14559972
[TBL] [Abstract][Full Text] [Related]
8. The role of Apex2 in class-switch recombination of immunoglobulin genes.
Guikema JE; Stavnezer J; Schrader CE
Int Immunol; 2010 Mar; 22(3):213; author reply 213-4. PubMed ID: 20185435
[No Abstract] [Full Text] [Related]
9. AID expression during B-cell development: searching for answers.
Kuraoka M; McWilliams L; Kelsoe G
Immunol Res; 2011 Apr; 49(1-3):3-13. PubMed ID: 21136202
[TBL] [Abstract][Full Text] [Related]
10. Activation induced cytidine deaminase mutant (AID-His130Pro) from Hyper IgM 2 patient retained mutagenic activity on SHM artificial substrate.
Ouadani H; Ben-Mustapha I; Ben-Ali M; Larguèche B; Jovanic T; Garcia S; Arcangioli B; Elloumi-Zghal H; Fathallah D; Hachicha M; Masmoudi H; Rougeon F; Barbouche MR
Mol Immunol; 2016 Nov; 79():77-82. PubMed ID: 27716525
[TBL] [Abstract][Full Text] [Related]
11. Secret sharers in the immune system: a novel RNA editing activity links switch recombination and somatic hypermutation.
Maizels N
Genome Biol; 2000; 1(4):REVIEWS1025. PubMed ID: 11178252
[TBL] [Abstract][Full Text] [Related]
12. Independent Roles of Switching and Hypermutation in the Development and Persistence of B Lymphocyte Memory.
Gitlin AD; von Boehmer L; Gazumyan A; Shulman Z; Oliveira TY; Nussenzweig MC
Immunity; 2016 Apr; 44(4):769-81. PubMed ID: 26944202
[TBL] [Abstract][Full Text] [Related]
13. Human uracil-DNA glycosylase deficiency associated with profoundly impaired immunoglobulin class-switch recombination.
Imai K; Slupphaug G; Lee WI; Revy P; Nonoyama S; Catalan N; Yel L; Forveille M; Kavli B; Krokan HE; Ochs HD; Fischer A; Durandy A
Nat Immunol; 2003 Oct; 4(10):1023-8. PubMed ID: 12958596
[TBL] [Abstract][Full Text] [Related]
14. Somatic hypermutation of immunoglobulin genes: merging mechanisms for genetic diversity.
Papavasiliou FN; Schatz DG
Cell; 2002 Apr; 109 Suppl():S35-44. PubMed ID: 11983151
[TBL] [Abstract][Full Text] [Related]
15. Negative regulation of activation-induced cytidine deaminase in B cells.
Muto T; Okazaki IM; Yamada S; Tanaka Y; Kinoshita K; Muramatsu M; Nagaoka H; Honjo T
Proc Natl Acad Sci U S A; 2006 Feb; 103(8):2752-7. PubMed ID: 16477013
[TBL] [Abstract][Full Text] [Related]
16. A novel mouse model for the hyper-IgM syndrome: a spontaneous activation-induced cytidine deaminase mutation leading to complete loss of Ig class switching and reduced somatic hypermutation.
Dahlberg CI; He M; Visnes T; Torres ML; Cortizas EM; Verdun RE; Westerberg LS; Severinson E; Ström L
J Immunol; 2014 Nov; 193(9):4732-8. PubMed ID: 25252954
[TBL] [Abstract][Full Text] [Related]
17. A Hyper-IgM Syndrome Mutation in Activation-Induced Cytidine Deaminase Disrupts G-Quadruplex Binding and Genome-wide Chromatin Localization.
Yewdell WT; Kim Y; Chowdhury P; Lau CM; Smolkin RM; Belcheva KT; Fernandez KC; Cols M; Yen WF; Vaidyanathan B; Angeletti D; McDermott AB; Yewdell JW; Sun JC; Chaudhuri J
Immunity; 2020 Nov; 53(5):952-970.e11. PubMed ID: 33098766
[TBL] [Abstract][Full Text] [Related]
18. Separate domains of AID are required for somatic hypermutation and class-switch recombination.
Shinkura R; Ito S; Begum NA; Nagaoka H; Muramatsu M; Kinoshita K; Sakakibara Y; Hijikata H; Honjo T
Nat Immunol; 2004 Jul; 5(7):707-12. PubMed ID: 15195091
[TBL] [Abstract][Full Text] [Related]
19. AID Phosphorylation Regulates Mismatch Repair-Dependent Class Switch Recombination and Affinity Maturation.
Choi JE; Matthews AJ; Michel G; Vuong BQ
J Immunol; 2020 Jan; 204(1):13-22. PubMed ID: 31757865
[TBL] [Abstract][Full Text] [Related]
20. B cell development leads off with a base hit: dU:dG mismatches in class switching and hypermutation.
Lee GS; Brandt VL; Roth DB
Mol Cell; 2004 Nov; 16(4):505-8. PubMed ID: 15546611
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]