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5. Glutamate receptor antibodies in neurological diseases: anti-AMPA-GluR3 antibodies, anti-NMDA-NR1 antibodies, anti-NMDA-NR2A/B antibodies, anti-mGluR1 antibodies or anti-mGluR5 antibodies are present in subpopulations of patients with either: epilepsy, encephalitis, cerebellar ataxia, systemic lupus erythematosus (SLE) and neuropsychiatric SLE, Sjogren's syndrome, schizophrenia, mania or stroke. These autoimmune anti-glutamate receptor antibodies can bind neurons in few brain regions, activate glutamate receptors, decrease glutamate receptor's expression, impair glutamate-induced signaling and function, activate blood brain barrier endothelial cells, kill neurons, damage the brain, induce behavioral/psychiatric/cognitive abnormalities and ataxia in animal models, and can be removed or silenced in some patients by immunotherapy. Levite M J Neural Transm (Vienna); 2014 Aug; 121(8):1029-75. PubMed ID: 25081016 [TBL] [Abstract][Full Text] [Related]
6. Glutamate Receptor Antibodies in Autoimmune Central Nervous System Disease: Basic Mechanisms, Clinical Features, and Antibody Detection. Scotton WJ; Karim A; Jacob S Methods Mol Biol; 2019; 1941():225-255. PubMed ID: 30707437 [TBL] [Abstract][Full Text] [Related]
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17. Screening for MOG-IgG and 27 other anti-glial and anti-neuronal autoantibodies in 'pattern II multiple sclerosis' and brain biopsy findings in a MOG-IgG-positive case. Jarius S; Metz I; König FB; Ruprecht K; Reindl M; Paul F; Brück W; Wildemann B Mult Scler; 2016 Oct; 22(12):1541-1549. PubMed ID: 26869529 [TBL] [Abstract][Full Text] [Related]