295 related articles for article (PubMed ID: 30420907)
1. Possible Clues for Brain Energy Translation via Endolysosomal Trafficking of APP-CTFs in Alzheimer's Disease.
Sivanesan S; Mundugaru R; Rajadas J
Oxid Med Cell Longev; 2018; 2018():2764831. PubMed ID: 30420907
[TBL] [Abstract][Full Text] [Related]
2. APP overexpression in the absence of NPC1 exacerbates metabolism of amyloidogenic proteins of Alzheimer's disease.
Maulik M; Peake K; Chung J; Wang Y; Vance JE; Kar S
Hum Mol Genet; 2015 Dec; 24(24):7132-50. PubMed ID: 26433932
[TBL] [Abstract][Full Text] [Related]
3. Phosphorylation of amyloid precursor carboxy-terminal fragments enhances their processing by a gamma-secretase-dependent mechanism.
Vingtdeux V; Hamdane M; Gompel M; Bégard S; Drobecq H; Ghestem A; Grosjean ME; Kostanjevecki V; Grognet P; Vanmechelen E; Buée L; Delacourte A; Sergeant N
Neurobiol Dis; 2005 Nov; 20(2):625-37. PubMed ID: 15936948
[TBL] [Abstract][Full Text] [Related]
4. Accumulation of APP C-terminal fragments causes endolysosomal dysfunction through the dysregulation of late endosome to lysosome-ER contact sites.
Bretou M; Sannerud R; Escamilla-Ayala A; Leroy T; Vrancx C; Van Acker ZP; Perdok A; Vermeire W; Vorsters I; Van Keymolen S; Maxson M; Pavie B; Wierda K; Eskelinen EL; Annaert W
Dev Cell; 2024 Jun; 59(12):1571-1592.e9. PubMed ID: 38626765
[TBL] [Abstract][Full Text] [Related]
5. Amino-terminal modification and tyrosine phosphorylation of [corrected] carboxy-terminal fragments of the amyloid precursor protein in Alzheimer's disease and Down's syndrome brain.
Russo C; Salis S; Dolcini V; Venezia V; Song XH; Teller JK; Schettini G
Neurobiol Dis; 2001 Feb; 8(1):173-80. PubMed ID: 11162251
[TBL] [Abstract][Full Text] [Related]
6. Amyloid precursor protein and endosomal-lysosomal dysfunction in Alzheimer's disease: inseparable partners in a multifactorial disease.
Nixon RA
FASEB J; 2017 Jul; 31(7):2729-2743. PubMed ID: 28663518
[TBL] [Abstract][Full Text] [Related]
7. Does Intraneuronal Accumulation of Carboxyl-terminal Fragments of the Amyloid Precursor Protein Trigger Early Neurotoxicity in Alzheimer's Disease?
Lauritzen I; Pardossi-Piquard R; Bourgeois A; Bécot A; Checler F
Curr Alzheimer Res; 2019; 16(5):453-457. PubMed ID: 30907322
[TBL] [Abstract][Full Text] [Related]
8. Macroautophagy is not directly involved in the metabolism of amyloid precursor protein.
Boland B; Smith DA; Mooney D; Jung SS; Walsh DM; Platt FM
J Biol Chem; 2010 Nov; 285(48):37415-26. PubMed ID: 20864542
[TBL] [Abstract][Full Text] [Related]
9. Upregulation of RIN3 induces endosomal dysfunction in Alzheimer's disease.
Shen R; Zhao X; He L; Ding Y; Xu W; Lin S; Fang S; Yang W; Sung K; Spencer B; Rissman RA; Lei M; Ding J; Wu C
Transl Neurodegener; 2020 Jun; 9(1):26. PubMed ID: 32552912
[TBL] [Abstract][Full Text] [Related]
10. Reduction of brain beta-amyloid (Abeta) by fluvastatin, a hydroxymethylglutaryl-CoA reductase inhibitor, through increase in degradation of amyloid precursor protein C-terminal fragments (APP-CTFs) and Abeta clearance.
Shinohara M; Sato N; Kurinami H; Takeuchi D; Takeda S; Shimamura M; Yamashita T; Uchiyama Y; Rakugi H; Morishita R
J Biol Chem; 2010 Jul; 285(29):22091-102. PubMed ID: 20472556
[TBL] [Abstract][Full Text] [Related]
11. NRBF2 is involved in the autophagic degradation process of APP-CTFs in Alzheimer disease models.
Yang C; Cai CZ; Song JX; Tan JQ; Durairajan SSK; Iyaswamy A; Wu MY; Chen LL; Yue Z; Li M; Lu JH
Autophagy; 2017; 13(12):2028-2040. PubMed ID: 28980867
[TBL] [Abstract][Full Text] [Related]
12. Progressive decrease of amyloid precursor protein carboxy terminal fragments (APP-CTFs), associated with tau pathology stages, in Alzheimer's disease.
Sergeant N; David JP; Champain D; Ghestem A; Wattez A; Delacourte A
J Neurochem; 2002 May; 81(4):663-72. PubMed ID: 12065626
[TBL] [Abstract][Full Text] [Related]
13. Decoding Alzheimer's disease from perturbed cerebral glucose metabolism: implications for diagnostic and therapeutic strategies.
Chen Z; Zhong C
Prog Neurobiol; 2013 Sep; 108():21-43. PubMed ID: 23850509
[TBL] [Abstract][Full Text] [Related]
14. A novel fluorescent probe reveals starvation controls the commitment of amyloid precursor protein to the lysosome.
Hein LK; Apaja PM; Hattersley K; Grose RH; Xie J; Proud CG; Sargeant TJ
Biochim Biophys Acta Mol Cell Res; 2017 Oct; 1864(10):1554-1565. PubMed ID: 28641977
[TBL] [Abstract][Full Text] [Related]
15. Alzheimer's disease.
De-Paula VJ; Radanovic M; Diniz BS; Forlenza OV
Subcell Biochem; 2012; 65():329-52. PubMed ID: 23225010
[TBL] [Abstract][Full Text] [Related]
16. Retromers in Alzheimer's disease.
Siegenthaler BM; Rajendran L
Neurodegener Dis; 2012; 10(1-4):116-21. PubMed ID: 22398391
[TBL] [Abstract][Full Text] [Related]
17. Identification of truncated C-terminal fragments of the Alzheimer's disease amyloid protein precursor derived from sequential proteolytic pathways.
Mosser S; Gerber H; Fraering PC
J Neurochem; 2021 Mar; 156(6):943-956. PubMed ID: 32757390
[TBL] [Abstract][Full Text] [Related]
18. Proteolytic processing of the Alzheimer's disease amyloid precursor protein in brain and platelets.
Evin G; Zhu A; Holsinger RM; Masters CL; Li QX
J Neurosci Res; 2003 Nov; 74(3):386-92. PubMed ID: 14598315
[TBL] [Abstract][Full Text] [Related]
19. α-secretase cleaved amyloid precursor protein (APP) accumulates in cholinergic dystrophic neurites in normal, aged hippocampus.
Yoon SY; Choi JU; Cho MH; Yang KM; Ha H; Chung IJ; Cho GS; Kim DH
Neuropathol Appl Neurobiol; 2013 Dec; 39(7):800-16. PubMed ID: 23414335
[TBL] [Abstract][Full Text] [Related]
20. ErbB2 regulates autophagic flux to modulate the proteostasis of APP-CTFs in Alzheimer's disease.
Wang BJ; Her GM; Hu MK; Chen YW; Tung YT; Wu PY; Hsu WM; Lee H; Jin LW; Hwang SL; Chen RP; Huang CJ; Liao YF
Proc Natl Acad Sci U S A; 2017 Apr; 114(15):E3129-E3138. PubMed ID: 28351972
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]