BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

184 related articles for article (PubMed ID: 30484113)

  • 1. In Schizophrenia, Deficits in Natural IgM Isotype Antibodies Including those Directed to Malondialdehyde and Azelaic Acid Strongly Predict Negative Symptoms, Neurocognitive Impairments, and the Deficit Syndrome.
    Maes M; Kanchanatawan B; Sirivichayakul S; Carvalho AF
    Mol Neurobiol; 2019 Jul; 56(7):5122-5135. PubMed ID: 30484113
    [TBL] [Abstract][Full Text] [Related]  

  • 2. In Schizophrenia, Increased Plasma IgM/IgA Responses to Gut Commensal Bacteria Are Associated with Negative Symptoms, Neurocognitive Impairments, and the Deficit Phenotype.
    Maes M; Kanchanatawan B; Sirivichayakul S; Carvalho AF
    Neurotox Res; 2019 Apr; 35(3):684-698. PubMed ID: 30552634
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Increased Levels of Plasma Tumor Necrosis Factor-α Mediate Schizophrenia Symptom Dimensions and Neurocognitive Impairments and Are Inversely Associated with Natural IgM Directed to Malondialdehyde and Paraoxonase 1 Activity.
    Maes M; Sirivichayakul S; Matsumoto AK; Maes A; Michelin AP; de Oliveira Semeão L; de Lima Pedrão JV; Moreira EG; Barbosa DS; Geffard M; Carvalho AF; Kanchanatawan B
    Mol Neurobiol; 2020 May; 57(5):2333-2345. PubMed ID: 32040834
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Deficit Schizophrenia Is Characterized by Defects in IgM-Mediated Responses to Tryptophan Catabolites (TRYCATs): a Paradigm Shift Towards Defects in Natural Self-Regulatory Immune Responses Coupled with Mucosa-Derived TRYCAT Pathway Activation.
    Kanchanatawan B; Sirivichayakul S; Ruxrungtham K; Carvalho AF; Geffard M; Anderson G; Maes M
    Mol Neurobiol; 2018 Mar; 55(3):2214-2226. PubMed ID: 28290151
    [TBL] [Abstract][Full Text] [Related]  

  • 5. IgM-mediated autoimmune responses to oxidative specific epitopes, but not nitrosylated adducts, are significantly decreased in pregnancy: association with bacterial translocation, perinatal and lifetime major depression and the tryptophan catabolite (TRYCAT) pathway.
    Roomruangwong C; Kanchanatawan B; Sirivichayakul S; Anderson G; Carvalho AF; Duleu S; Geffard M; Maes M
    Metab Brain Dis; 2017 Oct; 32(5):1571-1583. PubMed ID: 28600633
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Natural regulatory IgM-mediated autoimmune responses directed against malondialdehyde regulate oxidative and nitrosative pathways and coupled with IgM responses to nitroso adducts attenuate depressive and physiosomatic symptoms at the end of term pregnancy.
    Roomruangwong C; Barbosa DS; de Farias CC; Matsumoto AK; Baltus THL; Morelli NR; Kanchanatawan B; Duleu S; Geffard M; Maes M
    Psychiatry Clin Neurosci; 2018 Feb; 72(2):116-130. PubMed ID: 29194848
    [TBL] [Abstract][Full Text] [Related]  

  • 7. The Effects of Tryptophan Catabolites on Negative Symptoms and Deficit Schizophrenia are Partly Mediated by Executive Impairments: Results of Partial Least Squares Path Modeling.
    Kanchanatawan B; Maes M
    CNS Neurol Disord Drug Targets; 2018; 17(6):473-486. PubMed ID: 29968545
    [TBL] [Abstract][Full Text] [Related]  

  • 8. In schizophrenia, psychomotor retardation is associated with executive and memory impairments, negative and psychotic symptoms, neurotoxic immune products and lower natural IgM to malondialdehyde.
    Maes M; Sirivichayakul S; Kanchanatawan B; Carvalho AF
    World J Biol Psychiatry; 2020 Jun; 21(5):383-401. PubMed ID: 32031479
    [No Abstract]   [Full Text] [Related]  

  • 9. Changes in Tryptophan Catabolite (TRYCAT) Pathway Patterning Are Associated with Mild Impairments in Declarative Memory in Schizophrenia and Deficits in Semantic and Episodic Memory Coupled with Increased False-Memory Creation in Deficit Schizophrenia.
    Kanchanatawan B; Hemrungrojn S; Thika S; Sirivichayakul S; Ruxrungtham K; Carvalho AF; Geffard M; Anderson G; Maes M
    Mol Neurobiol; 2018 Jun; 55(6):5184-5201. PubMed ID: 28875464
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Supervised machine learning to decipher the complex associations between neuro-immune biomarkers and quality of life in schizophrenia.
    Kanchanatawan B; Sriswasdi S; Maes M
    Metab Brain Dis; 2019 Feb; 34(1):267-282. PubMed ID: 30467771
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Deficit schizophrenia is a discrete diagnostic category defined by neuro-immune and neurocognitive features: results of supervised machine learning.
    Kanchanatawan B; Sriswasdi S; Thika S; Sirivichayakul S; Carvalho AF; Geffard M; Kubera M; Maes M
    Metab Brain Dis; 2018 Aug; 33(4):1053-1067. PubMed ID: 29527624
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Lowered Antioxidant Defenses and Increased Oxidative Toxicity Are Hallmarks of Deficit Schizophrenia: a Nomothetic Network Psychiatry Approach.
    Maes M; Sirivichayakul S; Matsumoto AK; Michelin AP; de Oliveira Semeão L; de Lima Pedrão JV; Moreira EG; Barbosa DS; Carvalho AF; Solmi M; Kanchanatawan B
    Mol Neurobiol; 2020 Nov; 57(11):4578-4597. PubMed ID: 32754898
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Upregulation of the Intestinal Paracellular Pathway with Breakdown of Tight and Adherens Junctions in Deficit Schizophrenia.
    Maes M; Sirivichayakul S; Kanchanatawan B; Vodjani A
    Mol Neurobiol; 2019 Oct; 56(10):7056-7073. PubMed ID: 30972627
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Towards a new classification of stable phase schizophrenia into major and simple neuro-cognitive psychosis: Results of unsupervised machine learning analysis.
    Kanchanatawan B; Sriswasdi S; Thika S; Stoyanov D; Sirivichayakul S; Carvalho AF; Geffard M; Maes M
    J Eval Clin Pract; 2018 Aug; 24(4):879-891. PubMed ID: 29790237
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Attenuation of autoimmune responses to oxidative specific epitopes, but not nitroso-adducts, is associated with a better clinical outcome in Myalgic Encephalomyelitis/chronic fatigue syndrome.
    Maes M; Leunis JC
    Neuro Endocrinol Lett; 2014; 35(7):577-85. PubMed ID: 25617880
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Schizophrenia phenomenology comprises a bifactorial general severity and a single-group factor, which are differently associated with neurotoxic immune and immune-regulatory pathways.
    Maes M; Vojdani A; Geffard M; Moreira EG; Barbosa DS; Michelin AP; Semeão LO; Sirivichayakul S; Kanchanatawan B
    Biomol Concepts; 2019 Nov; 10(1):209-225. PubMed ID: 31734647
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Chronic fatigue syndrome is accompanied by an IgM-related immune response directed against neopitopes formed by oxidative or nitrosative damage to lipids and proteins.
    Maes M; Mihaylova I; Leunis JC
    Neuro Endocrinol Lett; 2006 Oct; 27(5):615-21. PubMed ID: 17159817
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Physio-somatic symptoms in schizophrenia: association with depression, anxiety, neurocognitive deficits and the tryptophan catabolite pathway.
    Kanchanatawan B; Sirivichayakul S; Thika S; Ruxrungtham K; Carvalho AF; Geffard M; Anderson G; Noto C; Ivanova R; Maes M
    Metab Brain Dis; 2017 Aug; 32(4):1003-1016. PubMed ID: 28258445
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Construction of a nitro-oxidative stress-driven, mechanistic model of mood disorders: A nomothetic network approach.
    Simeonova D; Stoyanov D; Leunis JC; Murdjeva M; Maes M
    Nitric Oxide; 2021 Jan; 106():45-54. PubMed ID: 33186727
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Breakdown of the Paracellular Tight and Adherens Junctions in the Gut and Blood Brain Barrier and Damage to the Vascular Barrier in Patients with Deficit Schizophrenia.
    Maes M; Sirivichayakul S; Kanchanatawan B; Vodjani A
    Neurotox Res; 2019 Aug; 36(2):306-322. PubMed ID: 31077000
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 10.