These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
140 related articles for article (PubMed ID: 30535995)
1. Overview of BCL-2 Family Proteins and Therapeutic Potentials. Huska JD; Lamb HM; Hardwick JM Methods Mol Biol; 2019; 1877():1-21. PubMed ID: 30535995 [TBL] [Abstract][Full Text] [Related]
2. Functional identification of the apoptosis effector BH3 domain in cellular protein BNIP1. Yasuda M; Chinnadurai G Oncogene; 2000 May; 19(19):2363-7. PubMed ID: 10822388 [TBL] [Abstract][Full Text] [Related]
3. A comparison of two strategies for affinity maturation of a BH3 peptide toward pro-survival Bcl-2 proteins. Zhang S; Long A; Link AJ ACS Synth Biol; 2012 Mar; 1(3):89-98. PubMed ID: 23651073 [TBL] [Abstract][Full Text] [Related]
4. Molecular dynamics study of peptide segments of the BH3 domain of the proapoptotic proteins Bak, Bax, Bid and Hrk bound to the Bcl-xL and Bcl-2 proteins. Pinto M; Perez JJ; Rubio-Martinez J J Comput Aided Mol Des; 2004 Jan; 18(1):13-22. PubMed ID: 15143800 [TBL] [Abstract][Full Text] [Related]
5. Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins. Willis SN; Chen L; Dewson G; Wei A; Naik E; Fletcher JI; Adams JM; Huang DC Genes Dev; 2005 Jun; 19(11):1294-305. PubMed ID: 15901672 [TBL] [Abstract][Full Text] [Related]
6. BOD (Bcl-2-related ovarian death gene) is an ovarian BH3 domain-containing proapoptotic Bcl-2 protein capable of dimerization with diverse antiapoptotic Bcl-2 members. Hsu SY; Lin P; Hsueh AJ Mol Endocrinol; 1998 Sep; 12(9):1432-40. PubMed ID: 9731710 [TBL] [Abstract][Full Text] [Related]
7. BH3-only proteins Noxa, Bik, Bmf, and Bid activate Bax and Bak indirectly when studied in yeast model. Gérecová G; Kopanicová J; Jaká P; Běhalová L; Juhásová B; Bhatia-Kiššová I; Forte M; Polčic P; Mentel M FEMS Yeast Res; 2013 Dec; 13(8):747-54. PubMed ID: 23991648 [TBL] [Abstract][Full Text] [Related]
8. In non-transformed cells Bak activates upon loss of anti-apoptotic Bcl-XL and Mcl-1 but in the absence of active BH3-only proteins. Senft D; Weber A; Saathoff F; Berking C; Heppt MV; Kammerbauer C; Rothenfusser S; Kellner S; Kurgyis Z; Besch R; Häcker G Cell Death Dis; 2015 Nov; 6(11):e1996. PubMed ID: 26610208 [TBL] [Abstract][Full Text] [Related]
9. Characterization of the anti-apoptotic mechanism of Bcl-B. Zhai D; Ke N; Zhang H; Ladror U; Joseph M; Eichinger A; Godzik A; Ng SC; Reed JC Biochem J; 2003 Nov; 376(Pt 1):229-36. PubMed ID: 12921534 [TBL] [Abstract][Full Text] [Related]
10. Nuclear magnetic resonance study of protein-protein interactions involving apoptosis regulator Diva (Boo) and the BH3 domain of proapoptotic Bcl-2 members. Santiveri CM; Sborgi L; de Alba E J Mol Recognit; 2012 Dec; 25(12):665-73. PubMed ID: 23192964 [TBL] [Abstract][Full Text] [Related]
11. The structural biology of BH3-only proteins. Kvansakul M; Hinds MG Methods Enzymol; 2014; 544():49-74. PubMed ID: 24974286 [TBL] [Abstract][Full Text] [Related]
12. Structural and biochemical analyses of Bcl-xL in complex with the BH3 domain of peroxisomal testis-specific 1. Lim D; Jin S; Shin HC; Kim W; Choi JS; Oh DB; Kim SJ; Seo J; Ku B Biochem Biophys Res Commun; 2022 Oct; 625():174-180. PubMed ID: 35964379 [TBL] [Abstract][Full Text] [Related]
13. Autophagy contributes to modulating the cytotoxicities of Bcl-2 homology domain-3 mimetics. Yu L; Liu S Semin Cancer Biol; 2013 Dec; 23(6 Pt B):553-60. PubMed ID: 24012660 [TBL] [Abstract][Full Text] [Related]
14. Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics. Letai A; Bassik MC; Walensky LD; Sorcinelli MD; Weiler S; Korsmeyer SJ Cancer Cell; 2002 Sep; 2(3):183-92. PubMed ID: 12242151 [TBL] [Abstract][Full Text] [Related]
15. How the Bcl-2 family of proteins interact to regulate apoptosis. van Delft MF; Huang DC Cell Res; 2006 Feb; 16(2):203-13. PubMed ID: 16474435 [TBL] [Abstract][Full Text] [Related]
16. BCL-xL Targeting to Induce Apoptosis and to Eliminate Chemotherapy-Induced Senescent Tumor Cells: From Navitoclax to Platelet-Sparing BCL-xL PROTACs. Skwarska A; Konopleva M Cancer Res; 2023 Nov; 83(21):3501-3503. PubMed ID: 37824434 [TBL] [Abstract][Full Text] [Related]
17. Telomerase Reverse Transcriptase Contains a BH3-Like Motif and Interacts with BCL-2 Family Members. Jin Y; You L; Kim HJ; Lee HW Mol Cells; 2018 Jul; 41(7):684-694. PubMed ID: 29937479 [TBL] [Abstract][Full Text] [Related]