239 related articles for article (PubMed ID: 30552634)
1. In Schizophrenia, Increased Plasma IgM/IgA Responses to Gut Commensal Bacteria Are Associated with Negative Symptoms, Neurocognitive Impairments, and the Deficit Phenotype.
Maes M; Kanchanatawan B; Sirivichayakul S; Carvalho AF
Neurotox Res; 2019 Apr; 35(3):684-698. PubMed ID: 30552634
[TBL] [Abstract][Full Text] [Related]
2. In Schizophrenia, Deficits in Natural IgM Isotype Antibodies Including those Directed to Malondialdehyde and Azelaic Acid Strongly Predict Negative Symptoms, Neurocognitive Impairments, and the Deficit Syndrome.
Maes M; Kanchanatawan B; Sirivichayakul S; Carvalho AF
Mol Neurobiol; 2019 Jul; 56(7):5122-5135. PubMed ID: 30484113
[TBL] [Abstract][Full Text] [Related]
3. Increased IgA and IgM responses against gut commensals in chronic depression: further evidence for increased bacterial translocation or leaky gut.
Maes M; Kubera M; Leunis JC; Berk M
J Affect Disord; 2012 Dec; 141(1):55-62. PubMed ID: 22410503
[TBL] [Abstract][Full Text] [Related]
4. IgM-mediated autoimmune responses to oxidative specific epitopes, but not nitrosylated adducts, are significantly decreased in pregnancy: association with bacterial translocation, perinatal and lifetime major depression and the tryptophan catabolite (TRYCAT) pathway.
Roomruangwong C; Kanchanatawan B; Sirivichayakul S; Anderson G; Carvalho AF; Duleu S; Geffard M; Maes M
Metab Brain Dis; 2017 Oct; 32(5):1571-1583. PubMed ID: 28600633
[TBL] [Abstract][Full Text] [Related]
5. Deficit Schizophrenia Is Characterized by Defects in IgM-Mediated Responses to Tryptophan Catabolites (TRYCATs): a Paradigm Shift Towards Defects in Natural Self-Regulatory Immune Responses Coupled with Mucosa-Derived TRYCAT Pathway Activation.
Kanchanatawan B; Sirivichayakul S; Ruxrungtham K; Carvalho AF; Geffard M; Anderson G; Maes M
Mol Neurobiol; 2018 Mar; 55(3):2214-2226. PubMed ID: 28290151
[TBL] [Abstract][Full Text] [Related]
6. Breakdown of the Paracellular Tight and Adherens Junctions in the Gut and Blood Brain Barrier and Damage to the Vascular Barrier in Patients with Deficit Schizophrenia.
Maes M; Sirivichayakul S; Kanchanatawan B; Vodjani A
Neurotox Res; 2019 Aug; 36(2):306-322. PubMed ID: 31077000
[TBL] [Abstract][Full Text] [Related]
7. Upregulation of the Intestinal Paracellular Pathway with Breakdown of Tight and Adherens Junctions in Deficit Schizophrenia.
Maes M; Sirivichayakul S; Kanchanatawan B; Vodjani A
Mol Neurobiol; 2019 Oct; 56(10):7056-7073. PubMed ID: 30972627
[TBL] [Abstract][Full Text] [Related]
8. Normalization of leaky gut in chronic fatigue syndrome (CFS) is accompanied by a clinical improvement: effects of age, duration of illness and the translocation of LPS from gram-negative bacteria.
Maes M; Leunis JC
Neuro Endocrinol Lett; 2008 Dec; 29(6):902-10. PubMed ID: 19112401
[TBL] [Abstract][Full Text] [Related]
9. Inflammatory and Oxidative Pathways Are New Drug Targets in Multiple Episode Schizophrenia and Leaky Gut, Klebsiella pneumoniae, and C1q Immune Complexes Are Additional Drug Targets in First Episode Schizophrenia.
Maes M; Vojdani A; Sirivichayakul S; Barbosa DS; Kanchanatawan B
Mol Neurobiol; 2021 Jul; 58(7):3319-3334. PubMed ID: 33675500
[TBL] [Abstract][Full Text] [Related]
10. Increased Levels of Plasma Tumor Necrosis Factor-α Mediate Schizophrenia Symptom Dimensions and Neurocognitive Impairments and Are Inversely Associated with Natural IgM Directed to Malondialdehyde and Paraoxonase 1 Activity.
Maes M; Sirivichayakul S; Matsumoto AK; Maes A; Michelin AP; de Oliveira Semeão L; de Lima Pedrão JV; Moreira EG; Barbosa DS; Geffard M; Carvalho AF; Kanchanatawan B
Mol Neurobiol; 2020 May; 57(5):2333-2345. PubMed ID: 32040834
[TBL] [Abstract][Full Text] [Related]
11. Schizophrenia phenomenology comprises a bifactorial general severity and a single-group factor, which are differently associated with neurotoxic immune and immune-regulatory pathways.
Maes M; Vojdani A; Geffard M; Moreira EG; Barbosa DS; Michelin AP; Semeão LO; Sirivichayakul S; Kanchanatawan B
Biomol Concepts; 2019 Nov; 10(1):209-225. PubMed ID: 31734647
[TBL] [Abstract][Full Text] [Related]
12. The gut-brain barrier in major depression: intestinal mucosal dysfunction with an increased translocation of LPS from gram negative enterobacteria (leaky gut) plays a role in the inflammatory pathophysiology of depression.
Maes M; Kubera M; Leunis JC
Neuro Endocrinol Lett; 2008 Feb; 29(1):117-24. PubMed ID: 18283240
[TBL] [Abstract][Full Text] [Related]
13. Increased Serum Immunoglobulin Responses to Gut Commensal Gram-Negative Bacteria in Unipolar Major Depression and Bipolar Disorder Type 1, Especially When Melancholia Is Present.
Simeonova D; Stoyanov D; Leunis JC; Carvalho AF; Kubera M; Murdjeva M; Maes M
Neurotox Res; 2020 Feb; 37(2):338-348. PubMed ID: 31802379
[TBL] [Abstract][Full Text] [Related]
14. Changes in Tryptophan Catabolite (TRYCAT) Pathway Patterning Are Associated with Mild Impairments in Declarative Memory in Schizophrenia and Deficits in Semantic and Episodic Memory Coupled with Increased False-Memory Creation in Deficit Schizophrenia.
Kanchanatawan B; Hemrungrojn S; Thika S; Sirivichayakul S; Ruxrungtham K; Carvalho AF; Geffard M; Anderson G; Maes M
Mol Neurobiol; 2018 Jun; 55(6):5184-5201. PubMed ID: 28875464
[TBL] [Abstract][Full Text] [Related]
15. IgA/IgM Responses to Gram-Negative Bacteria are not Associated with Perinatal Depression, but with Physio-somatic Symptoms and Activation of the Tryptophan Catabolite Pathway at the End of Term and Postnatal Anxiety.
Roomruangwong C; Kanchanatawan B; Sirivichayakul S; Anderson G; Carvalho AF; Duleu S; Geffard M; Maes M
CNS Neurol Disord Drug Targets; 2017; 16(4):472-483. PubMed ID: 28403801
[TBL] [Abstract][Full Text] [Related]
16. Deficit schizophrenia is a discrete diagnostic category defined by neuro-immune and neurocognitive features: results of supervised machine learning.
Kanchanatawan B; Sriswasdi S; Thika S; Sirivichayakul S; Carvalho AF; Geffard M; Kubera M; Maes M
Metab Brain Dis; 2018 Aug; 33(4):1053-1067. PubMed ID: 29527624
[TBL] [Abstract][Full Text] [Related]
17. Increased serum IgA and IgM against LPS of enterobacteria in chronic fatigue syndrome (CFS): indication for the involvement of gram-negative enterobacteria in the etiology of CFS and for the presence of an increased gut-intestinal permeability.
Maes M; Mihaylova I; Leunis JC
J Affect Disord; 2007 Apr; 99(1-3):237-40. PubMed ID: 17007934
[TBL] [Abstract][Full Text] [Related]
18. In depression, bacterial translocation may drive inflammatory responses, oxidative and nitrosative stress (O&NS), and autoimmune responses directed against O&NS-damaged neoepitopes.
Maes M; Kubera M; Leunis JC; Berk M; Geffard M; Bosmans E
Acta Psychiatr Scand; 2013 May; 127(5):344-54. PubMed ID: 22900942
[TBL] [Abstract][Full Text] [Related]
19. The Effects of Tryptophan Catabolites on Negative Symptoms and Deficit Schizophrenia are Partly Mediated by Executive Impairments: Results of Partial Least Squares Path Modeling.
Kanchanatawan B; Maes M
CNS Neurol Disord Drug Targets; 2018; 17(6):473-486. PubMed ID: 29968545
[TBL] [Abstract][Full Text] [Related]
20. Depressive, anxiety and hypomanic symptoms in schizophrenia may be driven by tryptophan catabolite (TRYCAT) patterning of IgA and IgM responses directed to TRYCATs.
Kanchanatawan B; Sirivichayakul S; Carvalho AF; Anderson G; Galecki P; Maes M
Prog Neuropsychopharmacol Biol Psychiatry; 2018 Jan; 80(Pt C):205-216. PubMed ID: 28690204
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
