251 related articles for article (PubMed ID: 30807703)
1. Ligase 3-mediated end-joining maintains genome stability of human embryonic stem cells.
Kohutova A; Raška J; Kruta M; Seneklova M; Barta T; Fojtik P; Jurakova T; Walter CA; Hampl A; Dvorak P; Rotrekl V
FASEB J; 2019 Jun; 33(6):6778-6788. PubMed ID: 30807703
[TBL] [Abstract][Full Text] [Related]
2. Telomere-Internal Double-Strand Breaks Are Repaired by Homologous Recombination and PARP1/Lig3-Dependent End-Joining.
Doksani Y; de Lange T
Cell Rep; 2016 Nov; 17(6):1646-1656. PubMed ID: 27806302
[TBL] [Abstract][Full Text] [Related]
3. Regulation of DNA repair in the absence of classical non-homologous end joining.
Kang YJ; Yan CT
DNA Repair (Amst); 2018 Aug; 68():34-40. PubMed ID: 29929045
[TBL] [Abstract][Full Text] [Related]
4. A human iPSC model of Ligase IV deficiency reveals an important role for NHEJ-mediated-DSB repair in the survival and genomic stability of induced pluripotent stem cells and emerging haematopoietic progenitors.
Tilgner K; Neganova I; Moreno-Gimeno I; Al-Aama JY; Burks D; Yung S; Singhapol C; Saretzki G; Evans J; Gorbunova V; Gennery A; Przyborski S; Stojkovic M; Armstrong L; Jeggo P; Lako M
Cell Death Differ; 2013 Aug; 20(8):1089-100. PubMed ID: 23722522
[TBL] [Abstract][Full Text] [Related]
5. Marked contribution of alternative end-joining to chromosome-translocation-formation by stochastically induced DNA double-strand-breaks in G2-phase human cells.
Soni A; Siemann M; Pantelias GE; Iliakis G
Mutat Res Genet Toxicol Environ Mutagen; 2015 Nov; 793():2-8. PubMed ID: 26520366
[TBL] [Abstract][Full Text] [Related]
6. Time-dependent predominance of nonhomologous DNA end-joining pathways during embryonic development in mice.
Chiruvella KK; Sebastian R; Sharma S; Karande AA; Choudhary B; Raghavan SC
J Mol Biol; 2012 Mar; 417(3):197-211. PubMed ID: 22306462
[TBL] [Abstract][Full Text] [Related]
7. Deregulation of DNA double-strand break repair in multiple myeloma: implications for genome stability.
Herrero AB; San Miguel J; Gutierrez NC
PLoS One; 2015; 10(3):e0121581. PubMed ID: 25790254
[TBL] [Abstract][Full Text] [Related]
8. Efficient ligase 3-dependent microhomology-mediated end joining repair of DNA double-strand breaks in zebrafish embryos.
He MD; Zhang FH; Wang HL; Wang HP; Zhu ZY; Sun YH
Mutat Res; 2015 Oct; 780():86-96. PubMed ID: 26318124
[TBL] [Abstract][Full Text] [Related]
9. Microhomology-mediated end joining is the principal mediator of double-strand break repair during mitochondrial DNA lesions.
Tadi SK; Sebastian R; Dahal S; Babu RK; Choudhary B; Raghavan SC
Mol Biol Cell; 2016 Jan; 27(2):223-35. PubMed ID: 26609070
[TBL] [Abstract][Full Text] [Related]
10. Repair of DNA double-strand breaks by mammalian alternative end-joining pathways.
Sallmyr A; Tomkinson AE
J Biol Chem; 2018 Jul; 293(27):10536-10546. PubMed ID: 29530982
[TBL] [Abstract][Full Text] [Related]
11. Ligase I and ligase III mediate the DNA double-strand break ligation in alternative end-joining.
Lu G; Duan J; Shu S; Wang X; Gao L; Guo J; Zhang Y
Proc Natl Acad Sci U S A; 2016 Feb; 113(5):1256-60. PubMed ID: 26787905
[TBL] [Abstract][Full Text] [Related]
12. Human DNA ligases in replication and repair.
Sallmyr A; Rashid I; Bhandari SK; Naila T; Tomkinson AE
DNA Repair (Amst); 2020 Sep; 93():102908. PubMed ID: 33087274
[TBL] [Abstract][Full Text] [Related]
13. Deficiency of ligase IV leads to reduced NHEJ, accumulation of DNA damage, and can sensitize cells to cancer therapeutics.
Kumari N; Antil H; Kumari S; Raghavan SC
Genomics; 2023 Nov; 115(6):110731. PubMed ID: 37871849
[TBL] [Abstract][Full Text] [Related]
14. Decrease in abundance of apurinic/apyrimidinic endonuclease causes failure of base excision repair in culture-adapted human embryonic stem cells.
Krutá M; Bálek L; Hejnová R; Dobšáková Z; Eiselleová L; Matulka K; Bárta T; Fojtík P; Fajkus J; Hampl A; Dvořák P; Rotrekl V
Stem Cells; 2013 Apr; 31(4):693-702. PubMed ID: 23315699
[TBL] [Abstract][Full Text] [Related]
15. The Clustered DNA Lesions - Types, Pathways of Repair and Relevance to Human Health.
Bukowska B; Karwowski BT
Curr Med Chem; 2018; 25(23):2722-2735. PubMed ID: 29484975
[TBL] [Abstract][Full Text] [Related]
16. 53BP1: Keeping It under Control, Even at a Distance from DNA Damage.
Rass E; Willaume S; Bertrand P
Genes (Basel); 2022 Dec; 13(12):. PubMed ID: 36553657
[TBL] [Abstract][Full Text] [Related]
17. Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1.
Bakr A; Köcher S; Volquardsen J; Petersen C; Borgmann K; Dikomey E; Rothkamm K; Mansour WY
Oncotarget; 2016 Sep; 7(36):57679-57693. PubMed ID: 27494840
[TBL] [Abstract][Full Text] [Related]
18. The Determinant of DNA Repair Pathway Choices in Ionising Radiation-Induced DNA Double-Strand Breaks.
Zhao L; Bao C; Shang Y; He X; Ma C; Lei X; Mi D; Sun Y
Biomed Res Int; 2020; 2020():4834965. PubMed ID: 32908893
[TBL] [Abstract][Full Text] [Related]
19. Beta human papillomavirus 8E6 promotes alternative end joining.
Hu C; Bugbee T; Palinski R; Akinyemi IA; McIntosh MT; MacCarthy T; Bhaduri-McIntosh S; Wallace N
Elife; 2023 Jan; 12():. PubMed ID: 36692284
[TBL] [Abstract][Full Text] [Related]
20. Nucleoside Analogs Radiosensitize G0 Cells by Activating DNA End Resection and Alternative End-Joining.
Magin S; Meher PK; Iliakis G
Radiat Res; 2021 May; 195(5):412-426. PubMed ID: 33755161
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
