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2. Neuroleptic-like disruption of the conditioned avoidance response requires destruction of both the mesolimbic and nigrostriatal dopamine systems. Koob GF; Simon H; Herman JP; Le Moal M Brain Res; 1984 Jun; 303(2):319-29. PubMed ID: 6430466 [TBL] [Abstract][Full Text] [Related]
3. Mesolimbic dopamine and early post-6-OHDA lesion enhanced responses to d-amphetamine. Lynch MR; Carey RJ Pharmacol Biochem Behav; 1989 Feb; 32(2):577-80. PubMed ID: 2498910 [TBL] [Abstract][Full Text] [Related]
4. The role of mesolimbic dopamine in conditioned locomotion produced by amphetamine. Gold LH; Swerdlow NR; Koob GF Behav Neurosci; 1988 Aug; 102(4):544-52. PubMed ID: 3139012 [TBL] [Abstract][Full Text] [Related]
5. Dopaminergic substrates of amphetamine-induced place preference conditioning. Spyraki C; Fibiger HC; Phillips AG Brain Res; 1982 Dec; 253(1-2):185-93. PubMed ID: 6817850 [TBL] [Abstract][Full Text] [Related]
6. Relationship of limbic dopamine levels to amphetamine- and anticholinergic-induced hyperactivity in the rat. Carey RJ Biol Psychiatry; 1986 Mar; 21(3):317-21. PubMed ID: 3081055 [No Abstract] [Full Text] [Related]
7. Multiple receptors for brain dopamine in behavior regulation: concept of dopamine-E and dopamine-I receptors. Cools AR; van Rossum JM Life Sci; 1980 Oct; 27(14):1237-53. PubMed ID: 6255271 [No Abstract] [Full Text] [Related]
8. Animal models related to developmental disorders: theoretical and pharmacological analyses. Mailman RB; Lewis MH; Kilts CD Appl Res Ment Retard; 1981; 2(1):1-12. PubMed ID: 6171193 [No Abstract] [Full Text] [Related]