348 related articles for article (PubMed ID: 31006168)
1. Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.
Bohnert BN; Daiminger S; Wörn M; Sure F; Staudner T; Ilyaskin AV; Batbouta F; Janessa A; Schneider JC; Essigke D; Kanse S; Haerteis S; Korbmacher C; Artunc F
Acta Physiol (Oxf); 2019 Dec; 227(4):e13286. PubMed ID: 31006168
[TBL] [Abstract][Full Text] [Related]
2. Aberrant glomerular filtration of urokinase-type plasminogen activator in nephrotic syndrome leads to amiloride-sensitive plasminogen activation in urine.
Stæhr M; Buhl KB; Andersen RF; Svenningsen P; Nielsen F; Hinrichs GR; Bistrup C; Jensen BL
Am J Physiol Renal Physiol; 2015 Aug; 309(3):F235-41. PubMed ID: 25972510
[TBL] [Abstract][Full Text] [Related]
3. Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice.
Hinrichs GR; Weyer K; Friis UG; Svenningsen P; Lund IK; Nielsen R; Mollet G; Antignac C; Bistrup C; Jensen BL; Birn H
Acta Physiol (Oxf); 2019 Dec; 227(4):e13362. PubMed ID: 31423748
[TBL] [Abstract][Full Text] [Related]
4. Plasmin in nephrotic urine activates the epithelial sodium channel.
Svenningsen P; Bistrup C; Friis UG; Bertog M; Haerteis S; Krueger B; Stubbe J; Jensen ON; Thiesson HC; Uhrenholt TR; Jespersen B; Jensen BL; Korbmacher C; Skøtt O
J Am Soc Nephrol; 2009 Feb; 20(2):299-310. PubMed ID: 19073825
[TBL] [Abstract][Full Text] [Related]
5. Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.
Bohnert BN; Menacher M; Janessa A; Wörn M; Schork A; Daiminger S; Kalbacher H; Häring HU; Daniel C; Amann K; Sure F; Bertog M; Haerteis S; Korbmacher C; Artunc F
Kidney Int; 2018 Jan; 93(1):159-172. PubMed ID: 29042083
[TBL] [Abstract][Full Text] [Related]
6. Proteolytic regulation of epithelial sodium channels by urokinase plasminogen activator: cutting edge and cleavage sites.
Ji HL; Zhao R; Komissarov AA; Chang Y; Liu Y; Matthay MA
J Biol Chem; 2015 Feb; 290(9):5241-55. PubMed ID: 25555911
[TBL] [Abstract][Full Text] [Related]
7. Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome.
Xiao M; Bohnert BN; Aypek H; Kretz O; Grahammer F; Aukschun U; Wörn M; Janessa A; Essigke D; Daniel C; Amann K; Huber TB; Plow EF; Birkenfeld AL; Artunc F
Acta Physiol (Oxf); 2021 Jan; 231(1):e13512. PubMed ID: 32455507
[TBL] [Abstract][Full Text] [Related]
8. Regulation of epithelial sodium channels in urokinase plasminogen activator deficiency.
Chen Z; Zhao R; Zhao M; Liang X; Bhattarai D; Dhiman R; Shetty S; Idell S; Ji HL
Am J Physiol Lung Cell Mol Physiol; 2014 Oct; 307(8):L609-17. PubMed ID: 25172911
[TBL] [Abstract][Full Text] [Related]
9. Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice.
Haerteis S; Schork A; Dörffel T; Bohnert BN; Nacken R; Wörn M; Xiao M; Essigke D; Janessa A; Schmaier AH; Feener EP; Häring HU; Bertog M; Korbmacher C; Artunc F
Acta Physiol (Oxf); 2018 Sep; 224(1):e13060. PubMed ID: 29489063
[TBL] [Abstract][Full Text] [Related]
10. Proteolytic activation of the epithelial sodium channel (ENaC) by factor VII activating protease (FSAP) and its relevance for sodium retention in nephrotic mice.
Artunc F; Bohnert BN; Schneider JC; Staudner T; Sure F; Ilyaskin AV; Wörn M; Essigke D; Janessa A; Nielsen NV; Birkenfeld AL; Etscheid M; Haerteis S; Korbmacher C; Kanse SM
Pflugers Arch; 2022 Feb; 474(2):217-229. PubMed ID: 34870751
[TBL] [Abstract][Full Text] [Related]
11. Amiloride Reduces Urokinase/Plasminogen-Driven Intratubular Complement Activation in Glomerular Proteinuria.
Isaksson GL; Hinrichs GR; Andersen H; Bach ML; Weyer K; Zachar R; Henriksen JE; Madsen K; Lund IK; Mollet G; Bistrup C; Birn H; Jensen BL; Palarasah Y
J Am Soc Nephrol; 2024 Apr; 35(4):410-425. PubMed ID: 38254266
[TBL] [Abstract][Full Text] [Related]
12. Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome.
Larionov A; Dahlke E; Kunke M; Zanon Rodriguez L; Schiessl IM; Magnin JL; Kern U; Alli AA; Mollet G; Schilling O; Castrop H; Theilig F
J Cell Mol Med; 2019 Oct; 23(10):6543-6553. PubMed ID: 31368174
[TBL] [Abstract][Full Text] [Related]
13. Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S.
Haerteis S; Krappitz M; Bertog M; Krappitz A; Baraznenok V; Henderson I; Lindström E; Murphy JE; Bunnett NW; Korbmacher C
Pflugers Arch; 2012 Oct; 464(4):353-65. PubMed ID: 22864553
[TBL] [Abstract][Full Text] [Related]
14. Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC.
Andersen RF; Buhl KB; Jensen BL; Svenningsen P; Friis UG; Jespersen B; Rittig S
Pediatr Nephrol; 2013 Aug; 28(8):1227-34. PubMed ID: 23503750
[TBL] [Abstract][Full Text] [Related]
15. Mechanisms of renal NaCl retention in proteinuric disease.
Svenningsen P; Friis UG; Versland JB; Buhl KB; Møller Frederiksen B; Andersen H; Zachar RM; Bistrup C; Skøtt O; Jørgensen JS; Andersen RF; Jensen BL
Acta Physiol (Oxf); 2013 Mar; 207(3):536-45. PubMed ID: 23216619
[TBL] [Abstract][Full Text] [Related]
16. Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na
Bohnert BN; Essigke D; Janessa A; Schneider JC; Wörn M; Kalo MZ; Xiao M; Kong L; Omage K; Hennenlotter J; Amend B; Birkenfeld AL; Artunc F
Am J Physiol Renal Physiol; 2021 Oct; 321(4):F480-F493. PubMed ID: 34423678
[TBL] [Abstract][Full Text] [Related]
17. Plasminogen Deficiency and Amiloride Mitigate Angiotensin II-Induced Hypertension in Type 1 Diabetic Mice Suggesting Effects Through the Epithelial Sodium Channel.
Andersen H; Hansen MH; Buhl KB; Stæhr M; Friis UG; Enggaard C; Supramaniyam S; Lund IK; Svenningsen P; Hansen PBL; Jensen BL
J Am Heart Assoc; 2020 Dec; 9(23):e016387. PubMed ID: 33215566
[TBL] [Abstract][Full Text] [Related]
18. Mechanisms of sodium retention in nephrotic syndrome.
Hinrichs GR; Jensen BL; Svenningsen P
Curr Opin Nephrol Hypertens; 2020 Mar; 29(2):207-212. PubMed ID: 31789848
[TBL] [Abstract][Full Text] [Related]
19. Evans Blue is not a suitable inhibitor of the epithelial sodium channel δ-subunit.
Perniss A; Wolf A; Wichmann L; Schönberger M; Althaus M
Biochem Biophys Res Commun; 2015 Oct; 466(3):468-74. PubMed ID: 26365349
[TBL] [Abstract][Full Text] [Related]
20. Plasmin and chymotrypsin have distinct preferences for channel activating cleavage sites in the γ subunit of the human epithelial sodium channel.
Haerteis S; Krappitz M; Diakov A; Krappitz A; Rauh R; Korbmacher C
J Gen Physiol; 2012 Oct; 140(4):375-89. PubMed ID: 22966015
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
