216 related articles for article (PubMed ID: 31115163)
1. Feline XLF accumulates at DNA damage sites in a Ku-dependent manner.
Koike M; Yutoku Y; Koike A
FEBS Open Bio; 2019 Jun; 9(6):1052-1062. PubMed ID: 31115163
[TBL] [Abstract][Full Text] [Related]
2. Cloning, localization and focus formation at DNA damage sites of canine XLF.
Koike M; Yutoku Y; Koike A
J Vet Med Sci; 2017 Jan; 79(1):22-28. PubMed ID: 27746407
[TBL] [Abstract][Full Text] [Related]
3. Genetic interaction between DNA repair factors PAXX, XLF, XRCC4 and DNA-PKcs in human cells.
Xing M; Oksenych V
FEBS Open Bio; 2019 Jul; 9(7):1315-1326. PubMed ID: 31141305
[TBL] [Abstract][Full Text] [Related]
4. KARP-1 works as a heterodimer with Ku70, but the function of KARP-1 cannot perfectly replace that of Ku80 in DSB repair.
Koike M; Yutoku Y; Koike A
Exp Cell Res; 2011 Oct; 317(16):2267-75. PubMed ID: 21756904
[TBL] [Abstract][Full Text] [Related]
5. An Intrinsically Disordered APLF Links Ku, DNA-PKcs, and XRCC4-DNA Ligase IV in an Extended Flexible Non-homologous End Joining Complex.
Hammel M; Yu Y; Radhakrishnan SK; Chokshi C; Tsai MS; Matsumoto Y; Kuzdovich M; Remesh SG; Fang S; Tomkinson AE; Lees-Miller SP; Tainer JA
J Biol Chem; 2016 Dec; 291(53):26987-27006. PubMed ID: 27875301
[TBL] [Abstract][Full Text] [Related]
6. Feline XRCC4 undergoes rapid Ku-dependent recruitment to DNA damage sites.
Koike M; Yutoku Y; Koike A
FEBS Open Bio; 2022 Apr; 12(4):798-810. PubMed ID: 35000298
[TBL] [Abstract][Full Text] [Related]
7. Cernunnos/XLF: a new player in DNA double-strand break repair.
Yano K; Morotomi-Yano K; Akiyama H
Int J Biochem Cell Biol; 2009 Jun; 41(6):1237-40. PubMed ID: 18992362
[TBL] [Abstract][Full Text] [Related]
8. X-ray scattering reveals disordered linkers and dynamic interfaces in complexes and mechanisms for DNA double-strand break repair impacting cell and cancer biology.
Hammel M; Tainer JA
Protein Sci; 2021 Sep; 30(9):1735-1756. PubMed ID: 34056803
[TBL] [Abstract][Full Text] [Related]
9. Cloning, localization and focus formation at DNA damage sites of canine XRCC4.
Koike M; Yutoku Y; Koike A
J Vet Med Sci; 2017 Jan; 78(12):1865-1871. PubMed ID: 27644316
[TBL] [Abstract][Full Text] [Related]
10. DNA repair. PAXX, a paralog of XRCC4 and XLF, interacts with Ku to promote DNA double-strand break repair.
Ochi T; Blackford AN; Coates J; Jhujh S; Mehmood S; Tamura N; Travers J; Wu Q; Draviam VM; Robinson CV; Blundell TL; Jackson SP
Science; 2015 Jan; 347(6218):185-188. PubMed ID: 25574025
[TBL] [Abstract][Full Text] [Related]
11. Ku recruits XLF to DNA double-strand breaks.
Yano K; Morotomi-Yano K; Wang SY; Uematsu N; Lee KJ; Asaithamby A; Weterings E; Chen DJ
EMBO Rep; 2008 Jan; 9(1):91-6. PubMed ID: 18064046
[TBL] [Abstract][Full Text] [Related]
12. Deficiency of XLF and PAXX prevents DNA double-strand break repair by non-homologous end joining in lymphocytes.
Hung PJ; Chen BR; George R; Liberman C; Morales AJ; Colon-Ortiz P; Tyler JK; Sleckman BP; Bredemeyer AL
Cell Cycle; 2017 Feb; 16(3):286-295. PubMed ID: 27830975
[TBL] [Abstract][Full Text] [Related]
13. Functional significance of the interaction with Ku in DNA double-strand break recognition of XLF.
Yano K; Morotomi-Yano K; Lee KJ; Chen DJ
FEBS Lett; 2011 Mar; 585(6):841-6. PubMed ID: 21349273
[TBL] [Abstract][Full Text] [Related]
14. PAXX Is an Accessory c-NHEJ Factor that Associates with Ku70 and Has Overlapping Functions with XLF.
Tadi SK; Tellier-Lebègue C; Nemoz C; Drevet P; Audebert S; Roy S; Meek K; Charbonnier JB; Modesti M
Cell Rep; 2016 Oct; 17(2):541-555. PubMed ID: 27705800
[TBL] [Abstract][Full Text] [Related]
15. Cloning, localization and focus formation at DNA damage sites of canine Ku70.
Koike M; Yutoku Y; Koike A
J Vet Med Sci; 2017 Mar; 79(3):554-561. PubMed ID: 28163277
[TBL] [Abstract][Full Text] [Related]
16. Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells.
Fattah F; Lee EH; Weisensel N; Wang Y; Lichter N; Hendrickson EA
PLoS Genet; 2010 Feb; 6(2):e1000855. PubMed ID: 20195511
[TBL] [Abstract][Full Text] [Related]
17. PAXX and XLF DNA repair factors are functionally redundant in joining DNA breaks in a G1-arrested progenitor B-cell line.
Kumar V; Alt FW; Frock RL
Proc Natl Acad Sci U S A; 2016 Sep; 113(38):10619-24. PubMed ID: 27601633
[TBL] [Abstract][Full Text] [Related]
18. Dynamic changes in subcellular localization of cattle XLF during cell cycle, and focus formation of cattle XLF at DNA damage sites immediately after irradiation.
Koike M; Yutoku Y; Koike A
J Vet Med Sci; 2015 Sep; 77(9):1109-14. PubMed ID: 25947322
[TBL] [Abstract][Full Text] [Related]
19. Akt-mediated phosphorylation of XLF impairs non-homologous end-joining DNA repair.
Liu P; Gan W; Guo C; Xie A; Gao D; Guo J; Zhang J; Willis N; Su A; Asara JM; Scully R; Wei W
Mol Cell; 2015 Feb; 57(4):648-661. PubMed ID: 25661488
[TBL] [Abstract][Full Text] [Related]
20. TDP1 promotes assembly of non-homologous end joining protein complexes on DNA.
Heo J; Li J; Summerlin M; Hays A; Katyal S; McKinnon PJ; Nitiss KC; Nitiss JL; Hanakahi LA
DNA Repair (Amst); 2015 Jun; 30():28-37. PubMed ID: 25841101
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
