440 related articles for article (PubMed ID: 31158339)
1. Apigenin and its methylglyoxal-adduct inhibit advanced glycation end products-induced oxidative stress and inflammation in endothelial cells.
Zhou Q; Cheng KW; Gong J; Li ETS; Wang M
Biochem Pharmacol; 2019 Aug; 166():231-241. PubMed ID: 31158339
[TBL] [Abstract][Full Text] [Related]
2. Phloretin and its methylglyoxal adduct: Implications against advanced glycation end products-induced inflammation in endothelial cells.
Zhou Q; Gong J; Wang M
Food Chem Toxicol; 2019 Jul; 129():291-300. PubMed ID: 31059746
[TBL] [Abstract][Full Text] [Related]
3. Curcumin inhibits advanced glycation end product-induced oxidative stress and inflammatory responses in endothelial cell damage via trapping methylglyoxal.
Sun YP; Gu JF; Tan XB; Wang CF; Jia XB; Feng L; Liu JP
Mol Med Rep; 2016 Feb; 13(2):1475-86. PubMed ID: 26718010
[TBL] [Abstract][Full Text] [Related]
4. Salidroside ameliorates endothelial inflammation and oxidative stress by regulating the AMPK/NF-κB/NLRP3 signaling pathway in AGEs-induced HUVECs.
Hu R; Wang MQ; Ni SH; Wang M; Liu LY; You HY; Wu XH; Wang YJ; Lu L; Wei LB
Eur J Pharmacol; 2020 Jan; 867():172797. PubMed ID: 31747547
[TBL] [Abstract][Full Text] [Related]
5. Salvia miltiorrhiza Prevents Methylglyoxal-Induced Glucotoxicity via the Regulation of Apoptosis-Related Pathways and the Glyoxalase System in Human Umbilical Vein Endothelial Cells.
Kim JS; Lee JH; Hong SM; Cho KH; Kim SY
Biol Pharm Bull; 2022 Jan; 45(1):51-62. PubMed ID: 34732594
[TBL] [Abstract][Full Text] [Related]
6. Caffeic Acid Inhibits the Formation of Advanced Glycation End Products (AGEs) and Mitigates the AGEs-Induced Oxidative Stress and Inflammation Reaction in Human Umbilical Vein Endothelial Cells (HUVECs).
Cao X; Xia Y; Zeng M; Wang W; He Y; Liu J
Chem Biodivers; 2019 Oct; 16(10):e1900174. PubMed ID: 31419039
[TBL] [Abstract][Full Text] [Related]
7. Methylglyoxal-derived hydroimidazolone-1 evokes inflammatory reactions in endothelial cells via an interaction with receptor for advanced glycation end products.
Ishibashi Y; Matsui T; Nakamura N; Sotokawauchi A; Higashimoto Y; Yamagishi SI
Diab Vasc Dis Res; 2017 Sep; 14(5):450-453. PubMed ID: 28631505
[TBL] [Abstract][Full Text] [Related]
8. Methylglyoxal-induced apoptosis is dependent on the suppression of c-FLIP
Jang JH; Kim EA; Park HJ; Sung EG; Song IH; Kim JY; Woo CH; Doh KO; Kim KH; Lee TJ
J Cell Mol Med; 2017 Nov; 21(11):2720-2731. PubMed ID: 28444875
[TBL] [Abstract][Full Text] [Related]
9. Methylglyoxal-derived hemoglobin advanced glycation end products induce apoptosis and oxidative stress in human umbilical vein endothelial cells.
Lee JH; Samsuzzaman M; Park MG; Park SJ; Kim SY
Int J Biol Macromol; 2021 Sep; 187():409-421. PubMed ID: 34271050
[TBL] [Abstract][Full Text] [Related]
10. The protective effect of Smilax glabra extract on advanced glycation end products-induced endothelial dysfunction in HUVECs via RAGE-ERK1/2-NF-κB pathway.
Sang HQ; Gu JF; Yuan JR; Zhang MH; Jia XB; Feng L
J Ethnopharmacol; 2014 Aug; 155(1):785-95. PubMed ID: 24953033
[TBL] [Abstract][Full Text] [Related]
11. Inhibition of Methylglyoxal-Induced AGEs/RAGE Expression Contributes to Dermal Protection by N-Acetyl-L-Cysteine.
Yang CT; Meng FH; Chen L; Li X; Cen LJ; Wen YH; Li CC; Zhang H
Cell Physiol Biochem; 2017; 41(2):742-754. PubMed ID: 28214842
[TBL] [Abstract][Full Text] [Related]
12. Sulforaphane reduces advanced glycation end products (AGEs)-induced inflammation in endothelial cells and rat aorta.
Matsui T; Nakamura N; Ojima A; Nishino Y; Yamagishi SI
Nutr Metab Cardiovasc Dis; 2016 Sep; 26(9):797-807. PubMed ID: 27212619
[TBL] [Abstract][Full Text] [Related]
13. Edaravone protected human brain microvascular endothelial cells from methylglyoxal-induced injury by inhibiting AGEs/RAGE/oxidative stress.
Li W; Xu H; Hu Y; He P; Ni Z; Xu H; Zhang Z; Dai H
PLoS One; 2013; 8(9):e76025. PubMed ID: 24098758
[TBL] [Abstract][Full Text] [Related]
14. Fructose causes endothelial cell damage via activation of advanced glycation end products-receptor system.
Sotokawauchi A; Matsui T; Higashimoto Y; Yamagishi SI
Diab Vasc Dis Res; 2019 Nov; 16(6):556-561. PubMed ID: 31375034
[TBL] [Abstract][Full Text] [Related]
15. Glyceraldehyde-derived pyridinium (GLAP) evokes oxidative stress and inflammatory and thrombogenic reactions in endothelial cells via the interaction with RAGE.
Matsui T; Oda E; Higashimoto Y; Yamagishi S
Cardiovasc Diabetol; 2015 Jan; 14():1. PubMed ID: 25582325
[TBL] [Abstract][Full Text] [Related]
16. Apigenin and baicalein ameliorate thoracic aortic structural deterioration and cognitive deficit via inhibiting AGEs/RAGE/NF-κB pathway in D-galactose-induced aging rats.
Ma Y; Ma Z; Zhang Y; Luo C; Huang P; Tong J; Ding H; Liu H
Eur J Pharmacol; 2024 Aug; 976():176660. PubMed ID: 38795756
[TBL] [Abstract][Full Text] [Related]
17. Pterostilbene prevents methylglyoxal-induced cytotoxicity in endothelial cells by regulating glyoxalase, oxidative stress and apoptosis.
Tang D; Xiao W; Gu WT; Zhang ZT; Xu SH; Chen ZQ; Xu YH; Zhang LY; Wang SM; Nie H
Food Chem Toxicol; 2021 Jul; 153():112244. PubMed ID: 33930484
[TBL] [Abstract][Full Text] [Related]
18. Phosphocreatine protects endothelial cells from Methylglyoxal induced oxidative stress and apoptosis via the regulation of PI3K/Akt/eNOS and NF-κB pathway.
Chu P; Han G; Ahsan A; Sun Z; Liu S; Zhang Z; Sun B; Song Y; Lin Y; Peng J; Tang Z
Vascul Pharmacol; 2017 Apr; 91():26-35. PubMed ID: 27590258
[TBL] [Abstract][Full Text] [Related]
19. Liquiritin attenuates advanced glycation end products-induced endothelial dysfunction via RAGE/NF-κB pathway in human umbilical vein endothelial cells.
Zhang X; Song Y; Han X; Feng L; Wang R; Zhang M; Zhu M; Jia X; Hu S
Mol Cell Biochem; 2013 Feb; 374(1-2):191-201. PubMed ID: 23229233
[TBL] [Abstract][Full Text] [Related]
20. Lespedeza bicolor ameliorates endothelial dysfunction induced by methylglyoxal glucotoxicity.
Do MH; Lee JH; Wahedi HM; Pak C; Lee CH; Yeo EJ; Lim Y; Ha SK; Choi I; Kim SY
Phytomedicine; 2017 Dec; 36():26-36. PubMed ID: 29157823
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
