115 related articles for article (PubMed ID: 3158299)
1. Selective increase in phosphorylation of a 47-kDa protein (F1) directly related to long-term potentiation.
Routtenberg A; Lovinger DM
Behav Neural Biol; 1985 Jan; 43(1):3-11. PubMed ID: 3158299
[TBL] [Abstract][Full Text] [Related]
2. Direct relation of long-term synaptic potentiation to phosphorylation of membrane protein F1, a substrate for membrane protein kinase C.
Lovinger DM; Colley PA; Akers RF; Nelson RB; Routtenberg A
Brain Res; 1986 Dec; 399(2):205-11. PubMed ID: 3828760
[TBL] [Abstract][Full Text] [Related]
3. Protein kinase C activation leading to protein F1 phosphorylation may regulate synaptic plasticity by presynaptic terminal growth.
Routtenberg A
Behav Neural Biol; 1985 Sep; 44(2):186-200. PubMed ID: 3904711
[TBL] [Abstract][Full Text] [Related]
4. Demonstration of presynaptic protein kinase C activation following long-term potentiation in rat hippocampal slices.
Leahy JC; Luo Y; Kent CS; Meiri KF; Vallano ML
Neuroscience; 1993 Feb; 52(3):563-74. PubMed ID: 8095708
[TBL] [Abstract][Full Text] [Related]
5. Phosphorylation of the presynaptic protein B-50 (GAP-43) is increased during electrically induced long-term potentiation.
Gianotti C; Nunzi MG; Gispen WH; Corradetti R
Neuron; 1992 May; 8(5):843-8. PubMed ID: 1534012
[TBL] [Abstract][Full Text] [Related]
6. A selective increase in phosporylation of protein F1, a protein kinase C substrate, directly related to three day growth of long term synaptic enhancement.
Lovinger DM; Akers RF; Nelson RB; Barnes CA; McNaughton BL; Routtenberg A
Brain Res; 1985 Sep; 343(1):137-43. PubMed ID: 2994827
[TBL] [Abstract][Full Text] [Related]
7. Surface protein phosphorylation by ecto-protein kinase is required for the maintenance of hippocampal long-term potentiation.
Chen W; Wieraszko A; Hogan MV; Yang HA; Kornecki E; Ehrlich YH
Proc Natl Acad Sci U S A; 1996 Aug; 93(16):8688-93. PubMed ID: 8710932
[TBL] [Abstract][Full Text] [Related]
8. Phosphoprotein regulation of memory formation: enhancement and control of synaptic plasticity by protein kinase C and protein F1.
Routtenberg A
Ann N Y Acad Sci; 1985; 444():203-11. PubMed ID: 2990291
[No Abstract] [Full Text] [Related]
9. Protein F1 and protein kinase C may regulate the persistence, not the initiation, of synaptic potentiation in the hippocampus.
Lovinger DM; Routtenberg A
Adv Exp Med Biol; 1987; 221():313-30. PubMed ID: 3324691
[No Abstract] [Full Text] [Related]
10. Characterization of protein F1 (47 kDa, 4.5 pI): a kinase C substrate directly related to neural plasticity.
Nelson RB; Routtenberg A
Exp Neurol; 1985 Jul; 89(1):213-24. PubMed ID: 3159591
[TBL] [Abstract][Full Text] [Related]
11. Phosphoproteins localized to presynaptic terminal linked to persistence of long-term potentiation (LTP): quantitative analysis of two-dimensional gels.
Nelson RB; Linden DJ; Routtenberg A
Brain Res; 1989 Sep; 497(1):30-42. PubMed ID: 2790456
[TBL] [Abstract][Full Text] [Related]
12. Phosphoprotein F1: purification and characterization of a brain kinase C substrate related to plasticity.
Chan SY; Murakami K; Routtenberg A
J Neurosci; 1986 Dec; 6(12):3618-27. PubMed ID: 3794793
[TBL] [Abstract][Full Text] [Related]
13. NMDA receptor blockade prevents the increase in protein kinase C substrate (protein F1) phosphorylation produced by long-term potentiation.
Linden DJ; Wong KL; Sheu FS; Routtenberg A
Brain Res; 1988 Aug; 458(1):142-6. PubMed ID: 2905192
[TBL] [Abstract][Full Text] [Related]
14. Long-term potentiation and synaptic protein phosphorylation.
Pasinelli P; Ramakers GM; Urban IJ; Hens JJ; Oestreicher AB; de Graan PN; Gispen WH
Behav Brain Res; 1995 Jan; 66(1-2):53-9. PubMed ID: 7755899
[TBL] [Abstract][Full Text] [Related]
15. Protein kinase C and F1/GAP-43 gene expression in hippocampus inversely related to synaptic enhancement lasting 3 days.
Meberg PJ; Barnes CA; McNaughton BL; Routtenberg A
Proc Natl Acad Sci U S A; 1993 Dec; 90(24):12050-4. PubMed ID: 8265669
[TBL] [Abstract][Full Text] [Related]
16. Characterization of the anoxia-induced long-term synaptic potentiation in area CA1 of the rat hippocampus.
Hsu KS; Huang CC
Br J Pharmacol; 1997 Oct; 122(4):671-81. PubMed ID: 9375963
[TBL] [Abstract][Full Text] [Related]
17. Biochemical and physiological studies of long-term synaptic plasticity.
Lynch G; Browning M; Bennett WF
Fed Proc; 1979 Jun; 38(7):2117-22. PubMed ID: 36301
[TBL] [Abstract][Full Text] [Related]
18. The two major phosphoproteins in growth cones are probably identical to two protein kinase C substrates correlated with persistence of long-term potentiation.
Nelson RB; Linden DJ; Hyman C; Pfenninger KH; Routtenberg A
J Neurosci; 1989 Feb; 9(2):381-9. PubMed ID: 2918368
[TBL] [Abstract][Full Text] [Related]
19. Mechanism of protein kinase C activation during the induction and maintenance of long-term potentiation probed using a selective peptide substrate.
Klann E; Chen SJ; Sweatt JD
Proc Natl Acad Sci U S A; 1993 Sep; 90(18):8337-41. PubMed ID: 8378303
[TBL] [Abstract][Full Text] [Related]
20. Protein kinase C phosphorylates a 47 Mr protein (F1) directly related to synaptic plasticity.
Akers RF; Routtenberg A
Brain Res; 1985 May; 334(1):147-51. PubMed ID: 3158377
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]