173 related articles for article (PubMed ID: 31690769)
1. Method and its Composition for encapsulation, stabilization, and delivery of siRNA in Anionic polymeric nanoplex: An In vitro- In vivo Assessment.
Raval N; Jogi H; Gondaliya P; Kalia K; Tekade RK
Sci Rep; 2019 Nov; 9(1):16047. PubMed ID: 31690769
[TBL] [Abstract][Full Text] [Related]
2. Cyclo-RGD Truncated Polymeric Nanoconstruct with Dendrimeric Templates for Targeted HDAC4 Gene Silencing in a Diabetic Nephropathy Mouse Model.
Raval N; Jogi H; Gondaliya P; Kalia K; Tekade RK
Mol Pharm; 2021 Feb; 18(2):641-666. PubMed ID: 32453574
[TBL] [Abstract][Full Text] [Related]
3. Histone deacetylase 4 mediates high glucose-induced podocyte apoptosis via upregulation of calcineurin.
Shi W; Huang Y; Zhao X; Xie Z; Dong W; Li R; Chen Y; Li Z; Wang W; Ye Z; Liu S; Zhang L; Liang X
Biochem Biophys Res Commun; 2020 Dec; 533(4):1061-1068. PubMed ID: 33019979
[TBL] [Abstract][Full Text] [Related]
4. HDAC4 blocks autophagy to trigger podocyte injury: non-epigenetic action in diabetic nephropathy.
Wei Q; Dong Z
Kidney Int; 2014 Oct; 86(4):666-8. PubMed ID: 25265947
[TBL] [Abstract][Full Text] [Related]
5. Histone deacetylase 4 selectively contributes to podocyte injury in diabetic nephropathy.
Wang X; Liu J; Zhen J; Zhang C; Wan Q; Liu G; Wei X; Zhang Y; Wang Z; Han H; Xu H; Bao C; Song Z; Zhang X; Li N; Yi F
Kidney Int; 2014 Oct; 86(4):712-25. PubMed ID: 24717296
[TBL] [Abstract][Full Text] [Related]
6. Dysregulation of low-density lipoprotein receptor contributes to podocyte injuries in diabetic nephropathy.
Zhang Y; Ma KL; Liu J; Wu Y; Hu ZB; Liu L; Liu BC
Am J Physiol Endocrinol Metab; 2015 Jun; 308(12):E1140-8. PubMed ID: 25921580
[TBL] [Abstract][Full Text] [Related]
7. RhoA protects the podocytes against high glucose-induced apoptosis through YAP and plays critical role in diabetic nephropathy.
Huang Z; Peng Y; Yu H; Yu X; Zhou J; Xiao J
Biochem Biophys Res Commun; 2018 Oct; 504(4):949-956. PubMed ID: 30220545
[TBL] [Abstract][Full Text] [Related]
8. Inhibition of high mobility group box 1 (HMGB1) attenuates podocyte apoptosis and epithelial-mesenchymal transition by regulating autophagy flux.
Jin J; Gong J; Zhao L; Zhang H; He Q; Jiang X
J Diabetes; 2019 Oct; 11(10):826-836. PubMed ID: 30864227
[TBL] [Abstract][Full Text] [Related]
9. LncRNA MALAT1 is dysregulated in diabetic nephropathy and involved in high glucose-induced podocyte injury via its interplay with β-catenin.
Hu M; Wang R; Li X; Fan M; Lin J; Zhen J; Chen L; Lv Z
J Cell Mol Med; 2017 Nov; 21(11):2732-2747. PubMed ID: 28444861
[TBL] [Abstract][Full Text] [Related]
10. Glycogen synthase kinase‑3β is required for epithelial‑mesenchymal transition and barrier dysfunction in mouse podocytes under high glucose conditions.
Guo J; Yang L; Qiao Y; Liu Z
Mol Med Rep; 2016 Nov; 14(5):4091-4098. PubMed ID: 27748847
[TBL] [Abstract][Full Text] [Related]
11. Sam68 mediates high glucose‑induced podocyte apoptosis through modulation of Bax/Bcl‑2.
Chen Y; Zhang L; Liu S; Yao B; Zhang H; Liang S; Ma J; Liang X; Shi W
Mol Med Rep; 2019 Oct; 20(4):3728-3734. PubMed ID: 31485651
[TBL] [Abstract][Full Text] [Related]
12. PRMT1 mediates podocyte injury and glomerular fibrosis through phosphorylation of ERK pathway.
Zhu Y
Biochem Biophys Res Commun; 2018 Jan; 495(1):828-838. PubMed ID: 29129692
[TBL] [Abstract][Full Text] [Related]
13. In vivo application of RNA interference: from functional genomics to therapeutics.
Lu PY; Xie F; Woodle MC
Adv Genet; 2005; 54():117-42. PubMed ID: 16096010
[TBL] [Abstract][Full Text] [Related]
14. MDM2 is implicated in high-glucose-induced podocyte mitotic catastrophe via Notch1 signalling.
Tang H; Lei CT; Ye C; Gao P; Wan C; Chen S; He FF; Wang YM; Su H; Zhang C
J Cell Mol Med; 2017 Dec; 21(12):3435-3444. PubMed ID: 28643424
[TBL] [Abstract][Full Text] [Related]
15. Septin 7 mediates high glucose-induced podocyte apoptosis.
Li R; Dong W; Zhang S; Yang H; Zhang L; Ye Z; Zhao X; Zhang H; Li Z; Xu L; Liu S; Shi W; Liang X
Biochem Biophys Res Commun; 2018 Nov; 506(3):522-528. PubMed ID: 30361092
[TBL] [Abstract][Full Text] [Related]
16. Functionalized bioengineered spider silk spheres improve nuclease resistance and activity of oligonucleotide therapeutics providing a strategy for cancer treatment.
Kozlowska AK; Florczak A; Smialek M; Dondajewska E; Mackiewicz A; Kortylewski M; Dams-Kozlowska H
Acta Biomater; 2017 Sep; 59():221-233. PubMed ID: 28694238
[TBL] [Abstract][Full Text] [Related]
17. DUSP26 regulates podocyte oxidative stress and fibrosis in a mouse model with diabetic nephropathy through the mediation of ROS.
Huang F; Sheng XX; Zhang HJ
Biochem Biophys Res Commun; 2019 Jul; 515(3):410-416. PubMed ID: 31155289
[TBL] [Abstract][Full Text] [Related]
18. TMEM16A exacerbates renal injury by activating P38/JNK signaling pathway to promote podocyte apoptosis in diabetic nephropathy mice.
Lian H; Cheng Y; Wu X
Biochem Biophys Res Commun; 2017 May; 487(2):201-208. PubMed ID: 28392397
[TBL] [Abstract][Full Text] [Related]
19. Parathyroid hormone-related protein induces hypertrophy in podocytes via TGF-beta(1) and p27(Kip1): implications for diabetic nephropathy.
Romero M; Ortega A; Izquierdo A; López-Luna P; Bosch RJ
Nephrol Dial Transplant; 2010 Aug; 25(8):2447-57. PubMed ID: 20200004
[TBL] [Abstract][Full Text] [Related]
20. Dopamine 1 receptor activation protects mouse diabetic podocytes injury via regulating the PKA/NOX-5/p38 MAPK axis.
Shao X; Zhang X; Hu J; Gao T; Chen J; Xu C; Wei C
Exp Cell Res; 2020 Mar; 388(2):111849. PubMed ID: 31954110
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
