These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
185 related articles for article (PubMed ID: 32056106)
1. Nucleation and kinetics of SOD1 aggregation in human cells for ALS1. Workman A Mol Cell Biochem; 2020 Mar; 466(1-2):117-128. PubMed ID: 32056106 [TBL] [Abstract][Full Text] [Related]
2. Tryptophan residue 32 in human Cu-Zn superoxide dismutase modulates prion-like propagation and strain selection. Crown A; McAlary L; Fagerli E; Brown H; Yerbury JJ; Galaleldeen A; Cashman NR; Borchelt DR; Ayers JI PLoS One; 2020; 15(1):e0227655. PubMed ID: 31999698 [TBL] [Abstract][Full Text] [Related]
3. Glycerolipid Headgroups Control Rate and Mechanism of Superoxide Dismutase-1 Aggregation and Accelerate Fibrillization of Slowly Aggregating Amyotrophic Lateral Sclerosis Mutants. Rasouli S; Abdolvahabi A; Croom CM; Plewman DL; Shi Y; Shaw BF ACS Chem Neurosci; 2018 Jul; 9(7):1743-1756. PubMed ID: 29649360 [TBL] [Abstract][Full Text] [Related]
4. Amyloidogenic regions in beta-strands II and III modulate the aggregation and toxicity of SOD1 in living cells. McAlary L; Nan JR; Shyu C; Sher M; Plotkin SS; Cashman NR Open Biol; 2024 Jun; 14(6):230418. PubMed ID: 38835240 [TBL] [Abstract][Full Text] [Related]
5. Cholesterol secosterol aldehyde adduction and aggregation of Cu,Zn-superoxide dismutase: Potential implications in ALS. Dantas LS; Chaves-Filho AB; Coelho FR; Genaro-Mattos TC; Tallman KA; Porter NA; Augusto O; Miyamoto S Redox Biol; 2018 Oct; 19():105-115. PubMed ID: 30142602 [TBL] [Abstract][Full Text] [Related]
7. β-Methylamino-L-alanine substitution of serine in SOD1 suggests a direct role in ALS etiology. Proctor EA; Mowrey DD; Dokholyan NV PLoS Comput Biol; 2019 Jul; 15(7):e1007225. PubMed ID: 31323035 [TBL] [Abstract][Full Text] [Related]
8. ALS-Related Mutant SOD1 Aggregates Interfere with Mitophagy by Sequestering the Autophagy Receptor Optineurin. Tak YJ; Park JH; Rhim H; Kang S Int J Mol Sci; 2020 Oct; 21(20):. PubMed ID: 33065963 [TBL] [Abstract][Full Text] [Related]
9. Large SOD1 aggregates, unlike trimeric SOD1, do not impact cell viability in a model of amyotrophic lateral sclerosis. Zhu C; Beck MV; Griffith JD; Deshmukh M; Dokholyan NV Proc Natl Acad Sci U S A; 2018 May; 115(18):4661-4665. PubMed ID: 29666246 [TBL] [Abstract][Full Text] [Related]
10. Spinal cord homogenates from SOD1 familial amyotrophic lateral sclerosis induce SOD1 aggregation in living cells. Pokrishevsky E; Hong RH; Mackenzie IR; Cashman NR PLoS One; 2017; 12(9):e0184384. PubMed ID: 28877271 [TBL] [Abstract][Full Text] [Related]
11. Experimental Mutations in Superoxide Dismutase 1 Provide Insight into Potential Mechanisms Involved in Aberrant Aggregation in Familial Amyotrophic Lateral Sclerosis. Crown AM; Roberts BL; Crosby K; Brown H; Ayers JI; Hart PJ; Borchelt DR G3 (Bethesda); 2019 Mar; 9(3):719-728. PubMed ID: 30622123 [TBL] [Abstract][Full Text] [Related]
12. Induction of Cu/Zn Superoxide Dismutase (SOD1) Aggregation in Living Cells. Pokrishevsky E; Nan J; Cashman NR Methods Mol Biol; 2019; 1873():213-224. PubMed ID: 30341612 [TBL] [Abstract][Full Text] [Related]
13. Cellular Redox Systems Impact the Aggregation of Cu,Zn Superoxide Dismutase Linked to Familial Amyotrophic Lateral Sclerosis. Álvarez-Zaldiernas C; Lu J; Zheng Y; Yang H; Blasi J; Solsona C; Holmgren A J Biol Chem; 2016 Aug; 291(33):17197-208. PubMed ID: 27261461 [TBL] [Abstract][Full Text] [Related]
14. Cu/Zn Superoxide Dismutase Forms Amyloid Fibrils under Near-Physiological Quiescent Conditions: The Roles of Disulfide Bonds and Effects of Denaturant. Khan MAI; Respondek M; Kjellström S; Deep S; Linse S; Akke M ACS Chem Neurosci; 2017 Sep; 8(9):2019-2026. PubMed ID: 28585802 [TBL] [Abstract][Full Text] [Related]
15. A copper-deficient form of mutant Cu/Zn-superoxide dismutase as an early pathological species in amyotrophic lateral sclerosis. Tokuda E; Nomura T; Ohara S; Watanabe S; Yamanaka K; Morisaki Y; Misawa H; Furukawa Y Biochim Biophys Acta Mol Basis Dis; 2018 Jun; 1864(6 Pt A):2119-2130. PubMed ID: 29551730 [TBL] [Abstract][Full Text] [Related]
16. Loss of charge mutations in solvent exposed Lys residues of superoxide dismutase 1 do not induce inclusion formation in cultured cell models. Crosby K; Crown AM; Roberts BL; Brown H; Ayers JI; Borchelt DR PLoS One; 2018; 13(11):e0206751. PubMed ID: 30399166 [TBL] [Abstract][Full Text] [Related]
17. Changes in hydrophobicity mainly promotes the aggregation tendency of ALS associated SOD1 mutants. Tompa DR; Kadhirvel S Int J Biol Macromol; 2020 Feb; 145():904-913. PubMed ID: 31669277 [TBL] [Abstract][Full Text] [Related]
18. The molecular tweezer CLR01 inhibits aberrant superoxide dismutase 1 (SOD1) self-assembly Malik R; Meng H; Wongkongkathep P; Corrales CI; Sepanj N; Atlasi RS; Klärner FG; Schrader T; Spencer MJ; Loo JA; Wiedau M; Bitan G J Biol Chem; 2019 Mar; 294(10):3501-3513. PubMed ID: 30602569 [TBL] [Abstract][Full Text] [Related]
19. Heterodimer formation of wild-type and amyotrophic lateral sclerosis-causing mutant Cu/Zn-superoxide dismutase induces toxicity independent of protein aggregation. Witan H; Kern A; Koziollek-Drechsler I; Wade R; Behl C; Clement AM Hum Mol Genet; 2008 May; 17(10):1373-85. PubMed ID: 18211954 [TBL] [Abstract][Full Text] [Related]
20. Beclin1 decreases the RIPA-insoluble fraction of amyotrophic lateral sclerosis-linked SOD1 mutant via autophagy. Wei YM; Han B Neurosci Lett; 2019 Jan; 690():106-111. PubMed ID: 30321575 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]