BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

134 related articles for article (PubMed ID: 32122205)

  • 41. Tyrosine kinase inhibitors induce alternative spliced BCR-ABL
    Yuda J; Odawara J; Minami M; Muta T; Kohno K; Tanimoto K; Eto T; Shima T; Kikushige Y; Kato K; Takenaka K; Iwasaki H; Minami Y; Ohkawa Y; Akashi K; Miyamoto T
    Cancer Sci; 2020 Jul; 111(7):2361-2373. PubMed ID: 32314454
    [TBL] [Abstract][Full Text] [Related]  

  • 42. BCR-ABL1 mutation screening in chronic myeloid leukaemia: is next now?
    Soverini S; Martelli M; Bavaro L
    Lancet Haematol; 2019 May; 6(5):e236-e237. PubMed ID: 31036315
    [No Abstract]   [Full Text] [Related]  

  • 43. Molecular monitoring of chronic myeloid leukemia: present and future.
    Yeung CC; Egan D; Radich JP
    Expert Rev Mol Diagn; 2016 Oct; 16(10):1083-1091. PubMed ID: 27552202
    [TBL] [Abstract][Full Text] [Related]  

  • 44. [Chronic myeloid leukemia with variant e13a3 (b2a3) BCR-ABL1 as an ABL1 tyrosine kinase inhibitor-sensitive subtype].
    Togitani K; Asagiri T; Kamioka M; Kojima K
    Rinsho Ketsueki; 2021; 62(3):180-185. PubMed ID: 33828011
    [TBL] [Abstract][Full Text] [Related]  

  • 45. Minimal Residual Disease Eradication in CML: Does It Really Matter?
    Tantravahi SK; Guthula RS; O'Hare T; Deininger MW
    Curr Hematol Malig Rep; 2017 Oct; 12(5):495-505. PubMed ID: 28852963
    [TBL] [Abstract][Full Text] [Related]  

  • 46. The impact of multiple low-level BCR-ABL1 mutations on response to ponatinib.
    Parker WT; Yeung DT; Yeoman AL; Altamura HK; Jamison BA; Field CR; Hodgson JG; Lustgarten S; Rivera VM; Hughes TP; Branford S
    Blood; 2016 Apr; 127(15):1870-80. PubMed ID: 26773037
    [TBL] [Abstract][Full Text] [Related]  

  • 47. BCR-ABL1 e6a2 transcript in chronic myeloid leukemia: biological features and molecular monitoring by droplet digital PCR.
    Zagaria A; Anelli L; Coccaro N; Tota G; Casieri P; Cellamare A; Impera L; Brunetti C; Minervini A; Minervini CF; Delia M; Cumbo C; Orsini P; Specchia G; Albano F
    Virchows Arch; 2015 Sep; 467(3):357-63. PubMed ID: 26149409
    [TBL] [Abstract][Full Text] [Related]  

  • 48. Reconsideration of BCR-ABL protein flow cytometric immunobead assay: how potent to diagnose and monitor chronic myeloid leukemia?
    Kelani R; Monem F
    Int J Lab Hematol; 2015 Oct; 37(5):723-8. PubMed ID: 26059167
    [TBL] [Abstract][Full Text] [Related]  

  • 49. Clinical Implications of Discordant Early Molecular Responses in CML Patients Treated with Imatinib.
    Stella S; Zammit V; Vitale SR; Pennisi MS; Massimino M; Tirrò E; Forte S; Spitaleri A; Antolino A; Siracusa S; Accurso V; Mannina D; Impera S; Musolino C; Russo S; Malato A; Mineo G; Musso M; Porretto F; Martino B; Di Raimondo F; Manzella L; Vigneri P; Stagno F
    Int J Mol Sci; 2019 May; 20(9):. PubMed ID: 31064152
    [TBL] [Abstract][Full Text] [Related]  

  • 50. F317L BCR-ABL1 kinase domain mutation associated with a sustained major molecular response in a CML patient on dasatinib.
    Oyekunle AA; Castagnetti F; Gugliotta G; Soverini S; Baccarani M; Rosti G
    Leuk Res; 2011 Jul; 35(7):e118-20. PubMed ID: 21489624
    [No Abstract]   [Full Text] [Related]  

  • 51. Prevalence and outcomes of uncommon BCR-ABL1 fusion transcripts in patients with chronic myeloid leukaemia: data from a single centre.
    Qin YZ; Jiang Q; Jiang H; Lai YY; Shi HX; Chen WM; Yu L; Huang XJ
    Br J Haematol; 2018 Sep; 182(5):693-700. PubMed ID: 29974949
    [TBL] [Abstract][Full Text] [Related]  

  • 52. Clinical Efficacy and Safety of First-Line Dasatinib Therapy and the Relevance of Velocity of BCR-ABL1 Transcript Decline for Achievement of Molecular Responses in Newly Diagnosed Chronic-Phase Chronic Myeloid Leukemia: Report from the Juntendo Yamanashi Cooperative Study Group.
    Takaku T; Iriyama N; Mitsumori T; Sato E; Gotoh A; Kirito K; Noguchi M; Koike M; Sakamoto J; Oba K; Komatsu N
    Oncology; 2018; 94(2):85-91. PubMed ID: 29151104
    [TBL] [Abstract][Full Text] [Related]  

  • 53. Population-Based Testing and Treatment Characteristics for Chronic Myelogenous Leukemia.
    Styles T; Wu M; Wilson R; Babcock F; Butterworth D; West DW; Richardson LC
    J Registry Manag; 2016; 41(3):134-142. PubMed ID: 28121314
    [TBL] [Abstract][Full Text] [Related]  

  • 54. [Chronic myelogenous leukemia: diagnosis and treatment].
    Demeter J; Poros A; Bödör C; Horváth L; Masszi T
    Orv Hetil; 2016 Sep; 157(37):1459-68. PubMed ID: 27615196
    [TBL] [Abstract][Full Text] [Related]  

  • 55. Ponatinib in the treatment of chronic myeloid leukemia and philadelphia chromosome positive acute lymphoblastic leukemia.
    Pavlovsky C; Chan O; Talati C; Pinilla-Ibarz J
    Future Oncol; 2019 Jan; 15(3):257-269. PubMed ID: 30251548
    [TBL] [Abstract][Full Text] [Related]  

  • 56. Molecular monitoring and mutations in chronic myeloid leukemia: how to get the most out of your tyrosine kinase inhibitor.
    Baccarani M; Soverini S; De Benedittis C
    Am Soc Clin Oncol Educ Book; 2014; ():167-75. PubMed ID: 24857074
    [TBL] [Abstract][Full Text] [Related]  

  • 57. Monitoring disease burden in chronic myeloid leukemia: Past, present, and future.
    Egan D; Radich J
    Am J Hematol; 2016 Jul; 91(7):742-6. PubMed ID: 27059166
    [TBL] [Abstract][Full Text] [Related]  

  • 58. Contemporary treatment methods of adult patients with BCR/ABL1 positive chronic myeloid leukemia.
    Slezáková K; Mistrík M; Bátorová A
    Vnitr Lek; 2020; 66(4):214-224. PubMed ID: 32972178
    [TBL] [Abstract][Full Text] [Related]  

  • 59. A BCR-ABL1 cutoff of 1.5% at 3 months, determined by the GeneXpert system, predicts an optimal response in patients with chronic myeloid leukemia.
    García-Gutiérrez V; Gómez-Casares MT; Puerta JM; Alonso-Domínguez JM; Osorio S; Hernández-Boluda JC; Collado R; Ramírez MJ; Ibáñez F; Martín ML; Rodríguez-Gambarte JD; Martínez-Laperche C; Gómez M; Fiallo DV; Redondo S; Rodríguez A; Ruiz-Nuño C; Steegmann JL; Jiménez-Velasco A;
    PLoS One; 2017; 12(3):e0173532. PubMed ID: 28278193
    [TBL] [Abstract][Full Text] [Related]  

  • 60. Extreme mutational selectivity of axitinib limits its potential use as a targeted therapeutic for BCR-ABL1-positive leukemia.
    Zabriskie MS; Eide CA; Yan D; Vellore NA; Pomicter AD; Savage SL; Druker BJ; Deininger MW; O'Hare T
    Leukemia; 2016 Jun; 30(6):1418-21. PubMed ID: 26582647
    [No Abstract]   [Full Text] [Related]  

    [Previous]   [Next]    [New Search]
    of 7.