331 related articles for article (PubMed ID: 32201277)
1. Extracellular matrix components isolated from diabetic mice alter cardiac fibroblast function through the AGE/RAGE signaling cascade.
Burr SD; Stewart JA
Life Sci; 2020 Jun; 250():117569. PubMed ID: 32201277
[TBL] [Abstract][Full Text] [Related]
2. Rap1a Regulates Cardiac Fibroblast Contraction of 3D Diabetic Collagen Matrices by Increased Activation of the AGE/RAGE Cascade.
Burr SD; Stewart JA
Cells; 2021 May; 10(6):. PubMed ID: 34067282
[TBL] [Abstract][Full Text] [Related]
3. Rap1a Overlaps the AGE/RAGE Signaling Cascade to Alter Expression of α-SMA, p-NF-κB, and p-PKC-ζ in Cardiac Fibroblasts Isolated from Type 2 Diabetic Mice.
Burr SD; Stewart JA
Cells; 2021 Mar; 10(3):. PubMed ID: 33806572
[TBL] [Abstract][Full Text] [Related]
4. Rap1a Activity Elevated the Impact of Endogenous AGEs in Diabetic Collagen to Stimulate Increased Myofibroblast Transition and Oxidative Stress.
Burr SD; Dorroh CC; Stewart JA
Int J Mol Sci; 2022 Apr; 23(9):. PubMed ID: 35562872
[TBL] [Abstract][Full Text] [Related]
5. RAGE and TGF-β1 Cross-Talk Regulate Extracellular Matrix Turnover and Cytokine Synthesis in AGEs Exposed Fibroblast Cells.
Serban AI; Stanca L; Geicu OI; Munteanu MC; Dinischiotu A
PLoS One; 2016; 11(3):e0152376. PubMed ID: 27015414
[TBL] [Abstract][Full Text] [Related]
6. Targeting extracellular matrix glycation to attenuate fibroblast activation.
Jang M; Oh SW; Lee Y; Kim JY; Ji ES; Kim P
Acta Biomater; 2022 Mar; 141():255-263. PubMed ID: 35081431
[TBL] [Abstract][Full Text] [Related]
7. The Impact of Diabetic Conditions and AGE/RAGE Signaling on Cardiac Fibroblast Migration.
Burr SD; Harmon MB; Jr JAS
Front Cell Dev Biol; 2020; 8():112. PubMed ID: 32158758
[TBL] [Abstract][Full Text] [Related]
8. Stimulatory effects of advanced glycation endproducts (AGEs) on fibronectin matrix assembly.
Pastino AK; Greco TM; Mathias RA; Cristea IM; Schwarzbauer JE
Matrix Biol; 2017 May; 59():39-53. PubMed ID: 27425255
[TBL] [Abstract][Full Text] [Related]
9. Advanced glycation end-products regulate extracellular matrix-adipocyte metabolic crosstalk in diabetes.
Strieder-Barboza C; Baker NA; Flesher CG; Karmakar M; Neeley CK; Polsinelli D; Dimick JB; Finks JF; Ghaferi AA; Varban OA; Lumeng CN; O'Rourke RW
Sci Rep; 2019 Dec; 9(1):19748. PubMed ID: 31875018
[TBL] [Abstract][Full Text] [Related]
10. Glycated collagen - a 3D matrix system to study pathological cell behavior.
Dandia H; Makkad K; Tayalia P
Biomater Sci; 2019 Aug; 7(8):3480-3488. PubMed ID: 31282511
[TBL] [Abstract][Full Text] [Related]
11. Salvianolic Acid A Protects Against Diabetic Nephropathy through Ameliorating Glomerular Endothelial Dysfunction via Inhibiting AGE-RAGE Signaling.
Hou B; Qiang G; Zhao Y; Yang X; Chen X; Yan Y; Wang X; Liu C; Zhang L; Du G
Cell Physiol Biochem; 2017; 44(6):2378-2394. PubMed ID: 29262395
[TBL] [Abstract][Full Text] [Related]
12. AGEs-RAGE axis mediates myocardial fibrosis via activation of cardiac fibroblasts induced by autophagy in heart failure.
Liang B; Zhou Z; Yang Z; Liu J; Zhang L; He J; Li H; Huang Y; Yang Q; Xian S; Wang L
Exp Physiol; 2022 Aug; 107(8):879-891. PubMed ID: 35598104
[TBL] [Abstract][Full Text] [Related]
13. Crosstalk between the renin-angiotensin system and the advance glycation end product axis in the heart: role of the cardiac fibroblast.
Yamazaki KG; Gonzalez E; Zambon AC
J Cardiovasc Transl Res; 2012 Dec; 5(6):805-13. PubMed ID: 23054657
[TBL] [Abstract][Full Text] [Related]
14. Cardiac fibroblast-dependent extracellular matrix accumulation is associated with diastolic stiffness in type 2 diabetes.
Hutchinson KR; Lord CK; West TA; Stewart JA
PLoS One; 2013; 8(8):e72080. PubMed ID: 23991045
[TBL] [Abstract][Full Text] [Related]
15. Persistent organic pollutants (POPs) increase rage signaling to promote downstream cardiovascular remodeling.
Coole JB; Burr SS; Kay AM; Singh JA; Kondakala S; Yang EJ; Kaplan BLF; Howell GE; Stewart JA
Environ Toxicol; 2019 Oct; 34(10):1149-1159. PubMed ID: 31313498
[TBL] [Abstract][Full Text] [Related]
16. Blocking AGE-RAGE Signaling Improved Functional Disorders of Macrophages in Diabetic Wound.
Wang Q; Zhu G; Cao X; Dong J; Song F; Niu Y
J Diabetes Res; 2017; 2017():1428537. PubMed ID: 29119117
[TBL] [Abstract][Full Text] [Related]
17. Increased AGE-RAGE ratio in idiopathic pulmonary fibrosis.
Machahua C; Montes-Worboys A; Llatjos R; Escobar I; Dorca J; Molina-Molina M; Vicens-Zygmunt V
Respir Res; 2016 Nov; 17(1):144. PubMed ID: 27816054
[TBL] [Abstract][Full Text] [Related]
18. Implication of advanced glycation end products (Ages) and their receptor (Rage) on myocardial contractile and mitochondrial functions.
Neviere R; Yu Y; Wang L; Tessier F; Boulanger E
Glycoconj J; 2016 Aug; 33(4):607-17. PubMed ID: 27277623
[TBL] [Abstract][Full Text] [Related]
19. Advanced glycation endproducts produced by in vitro glycation of type I collagen modulate the functional and secretory behavior of dorsal root ganglion cells cultivated in two-dimensional system.
Bufalo MC; Almeida ME; Franca IA; Zambelli VO; Martins Sant'anna MB; Kimura LF; Giardini AC; Cury Y; Sampaio SC
Exp Cell Res; 2019 Sep; 382(2):111475. PubMed ID: 31255600
[TBL] [Abstract][Full Text] [Related]
20. AGE-RAGE axis blockade in diabetic nephropathy: Current status and future directions.
Sanajou D; Ghorbani Haghjo A; Argani H; Aslani S
Eur J Pharmacol; 2018 Aug; 833():158-164. PubMed ID: 29883668
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
