These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
350 related articles for article (PubMed ID: 32325157)
1. Protective effects of phenelzine administration on synaptic and non-synaptic cortical mitochondrial function and lipid peroxidation-mediated oxidative damage following TBI in young adult male rats. Hill RL; Singh IN; Wang JA; Kulbe JR; Hall ED Exp Neurol; 2020 Aug; 330():113322. PubMed ID: 32325157 [TBL] [Abstract][Full Text] [Related]
2. Effects of Phenelzine Administration on Mitochondrial Function, Calcium Handling, and Cytoskeletal Degradation after Experimental Traumatic Brain Injury. Hill RL; Singh IN; Wang JA; Hall ED J Neurotrauma; 2019 Apr; 36(8):1231-1251. PubMed ID: 30358485 [TBL] [Abstract][Full Text] [Related]
3. Phenelzine Protects Brain Mitochondrial Function In Vitro and In Vivo following Traumatic Brain Injury by Scavenging the Reactive Carbonyls 4-Hydroxynonenal and Acrolein Leading to Cortical Histological Neuroprotection. Cebak JE; Singh IN; Hill RL; Wang JA; Hall ED J Neurotrauma; 2017 Apr; 34(7):1302-1317. PubMed ID: 27750484 [TBL] [Abstract][Full Text] [Related]
4. Continuous Infusion of Phenelzine, Cyclosporine A, or Their Combination: Evaluation of Mitochondrial Bioenergetics, Oxidative Damage, and Cytoskeletal Degradation following Severe Controlled Cortical Impact Traumatic Brain Injury in Rats. Kulbe JR; Singh IN; Wang JA; Cebak JE; Hall ED J Neurotrauma; 2018 Jun; 35(11):1280-1293. PubMed ID: 29336204 [TBL] [Abstract][Full Text] [Related]
5. Phenelzine mitochondrial functional preservation and neuroprotection after traumatic brain injury related to scavenging of the lipid peroxidation-derived aldehyde 4-hydroxy-2-nonenal. Singh IN; Gilmer LK; Miller DM; Cebak JE; Wang JA; Hall ED J Cereb Blood Flow Metab; 2013 Apr; 33(4):593-9. PubMed ID: 23321786 [TBL] [Abstract][Full Text] [Related]
6. Pharmacological inhibition of lipid peroxidative damage by the 21-aminosteroid U-74389G improves cortical mitochondrial function following traumatic brain injury in young adult male rats. Hill RL; Singh IN; Brelsfoard J; Hall ED Neuropharmacology; 2020 Jun; 170():108023. PubMed ID: 32142792 [TBL] [Abstract][Full Text] [Related]
7. Synaptic Mitochondria are More Susceptible to Traumatic Brain Injury-induced Oxidative Damage and Respiratory Dysfunction than Non-synaptic Mitochondria. Hill RL; Kulbe JR; Singh IN; Wang JA; Hall ED Neuroscience; 2018 Aug; 386():265-283. PubMed ID: 29960045 [TBL] [Abstract][Full Text] [Related]
8. Time courses of post-injury mitochondrial oxidative damage and respiratory dysfunction and neuronal cytoskeletal degradation in a rat model of focal traumatic brain injury. Hill RL; Singh IN; Wang JA; Hall ED Neurochem Int; 2017 Dec; 111():45-56. PubMed ID: 28342966 [TBL] [Abstract][Full Text] [Related]
9. Mitochondrial protection after traumatic brain injury by scavenging lipid peroxyl radicals. Mustafa AG; Singh IN; Wang J; Carrico KM; Hall ED J Neurochem; 2010 Jul; 114(1):271-80. PubMed ID: 20403083 [TBL] [Abstract][Full Text] [Related]
10. Synaptic Mitochondria Sustain More Damage than Non-Synaptic Mitochondria after Traumatic Brain Injury and Are Protected by Cyclosporine A. Kulbe JR; Hill RL; Singh IN; Wang JA; Hall ED J Neurotrauma; 2017 Apr; 34(7):1291-1301. PubMed ID: 27596283 [TBL] [Abstract][Full Text] [Related]
11. Nrf2/HO-1 mediates the neuroprotective effects of pramipexole by attenuating oxidative damage and mitochondrial perturbation after traumatic brain injury in rats. Salman M; Tabassum H; Parvez S Dis Model Mech; 2020 Aug; 13(8):. PubMed ID: 32540990 [TBL] [Abstract][Full Text] [Related]
12. Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice. Miller DM; Singh IN; Wang JA; Hall ED Exp Neurol; 2015 Feb; 264():103-10. PubMed ID: 25432068 [TBL] [Abstract][Full Text] [Related]
13. Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapy. Singh IN; Sullivan PG; Deng Y; Mbye LH; Hall ED J Cereb Blood Flow Metab; 2006 Nov; 26(11):1407-18. PubMed ID: 16538231 [TBL] [Abstract][Full Text] [Related]
14. Post-Injury Administration of Mitochondrial Uncouplers Increases Tissue Sparing and Improves Behavioral Outcome following Traumatic Brain Injury in Rodents. Pandya JD; Pauly JR; Nukala VN; Sebastian AH; Day KM; Korde AS; Maragos WF; Hall ED; Sullivan PG J Neurotrauma; 2007 May; 24(5):798-811. PubMed ID: 17518535 [TBL] [Abstract][Full Text] [Related]
15. Guanosine protects against behavioural and mitochondrial bioenergetic alterations after mild traumatic brain injury. Courtes AA; Gonçalves DF; Hartmann DD; da Rosa PC; Cassol G; Royes LFF; de Carvalho NR; Soares FAA Brain Res Bull; 2020 Oct; 163():31-39. PubMed ID: 32681970 [TBL] [Abstract][Full Text] [Related]
16. Sinomenine reduces neuronal cell apoptosis in mice after traumatic brain injury via its effect on mitochondrial pathway. Fu C; Wang Q; Zhai X; Gao J Drug Des Devel Ther; 2018; 12():77-84. PubMed ID: 29379271 [TBL] [Abstract][Full Text] [Related]
17. Low-Molecular-Weight Fucoidan Attenuates Mitochondrial Dysfunction and Improves Neurological Outcome After Traumatic Brain Injury in Aged Mice: Involvement of Sirt3. Wang T; Zhu M; He ZZ Cell Mol Neurobiol; 2016 Nov; 36(8):1257-1268. PubMed ID: 26743530 [TBL] [Abstract][Full Text] [Related]
18. The optimal dosage and window of opportunity to maintain mitochondrial homeostasis following traumatic brain injury using the uncoupler FCCP. Pandya JD; Pauly JR; Sullivan PG Exp Neurol; 2009 Aug; 218(2):381-9. PubMed ID: 19477175 [TBL] [Abstract][Full Text] [Related]