These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
116 related articles for article (PubMed ID: 32598986)
21. The Cysteine (Cys) Residues Cys-6 and Cys-111 in Mutant Superoxide Dismutase 1 (SOD1) A4V Are Required for Induction of Endoplasmic Reticulum Stress in Amyotrophic Lateral Sclerosis. Perri ER; Parakh S; Vidal M; Mehta P; Ma Y; Walker AK; Atkin JD J Mol Neurosci; 2020 Sep; 70(9):1357-1368. PubMed ID: 32445072 [TBL] [Abstract][Full Text] [Related]
22. Effect of overexpression of wild-type and mutant Cu/Zn-superoxide dismutases on oxidative stress and cell death induced by hydrogen peroxide, 4-hydroxynonenal or serum deprivation: potentiation of injury by ALS-related mutant superoxide dismutases and protection by Bcl-2. Lee M; Hyun DH; Halliwell B; Jenner P J Neurochem; 2001 Jul; 78(2):209-20. PubMed ID: 11461956 [TBL] [Abstract][Full Text] [Related]
23. Disulfide bond mediates aggregation, toxicity, and ubiquitylation of familial amyotrophic lateral sclerosis-linked mutant SOD1. Niwa J; Yamada S; Ishigaki S; Sone J; Takahashi M; Katsuno M; Tanaka F; Doyu M; Sobue G J Biol Chem; 2007 Sep; 282(38):28087-95. PubMed ID: 17666395 [TBL] [Abstract][Full Text] [Related]
24. The rate and equilibrium constants for a multistep reaction sequence for the aggregation of superoxide dismutase in amyotrophic lateral sclerosis. Khare SD; Caplow M; Dokholyan NV Proc Natl Acad Sci U S A; 2004 Oct; 101(42):15094-9. PubMed ID: 15475574 [TBL] [Abstract][Full Text] [Related]
25. Advanced glycation endproduct-modified superoxide dismutase-1 (SOD1)-positive inclusions are common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutations and transgenic mice expressing human SOD1 with a G85R mutation. Kato S; Horiuchi S; Liu J; Cleveland DW; Shibata N; Nakashima K; Nagai R; Hirano A; Takikawa M; Kato M; Nakano I; Ohama E Acta Neuropathol; 2000 Nov; 100(5):490-505. PubMed ID: 11045671 [TBL] [Abstract][Full Text] [Related]
26. Wild-type Cu/Zn superoxide dismutase (SOD1) does not facilitate, but impedes the formation of protein aggregates of amyotrophic lateral sclerosis causing mutant SOD1. Witan H; Gorlovoy P; Kaya AM; Koziollek-Drechsler I; Neumann H; Behl C; Clement AM Neurobiol Dis; 2009 Nov; 36(2):331-42. PubMed ID: 19660548 [TBL] [Abstract][Full Text] [Related]
27. A copper-deficient form of mutant Cu/Zn-superoxide dismutase as an early pathological species in amyotrophic lateral sclerosis. Tokuda E; Nomura T; Ohara S; Watanabe S; Yamanaka K; Morisaki Y; Misawa H; Furukawa Y Biochim Biophys Acta Mol Basis Dis; 2018 Jun; 1864(6 Pt A):2119-2130. PubMed ID: 29551730 [TBL] [Abstract][Full Text] [Related]
29. Heterodimer formation of wild-type and amyotrophic lateral sclerosis-causing mutant Cu/Zn-superoxide dismutase induces toxicity independent of protein aggregation. Witan H; Kern A; Koziollek-Drechsler I; Wade R; Behl C; Clement AM Hum Mol Genet; 2008 May; 17(10):1373-85. PubMed ID: 18211954 [TBL] [Abstract][Full Text] [Related]
30. SOD1 in neurotoxicity and its controversial roles in SOD1 mutation-negative ALS. Hayashi Y; Homma K; Ichijo H Adv Biol Regul; 2016 Jan; 60():95-104. PubMed ID: 26563614 [TBL] [Abstract][Full Text] [Related]
31. Oxidation-induced misfolding and aggregation of superoxide dismutase and its implications for amyotrophic lateral sclerosis. Rakhit R; Cunningham P; Furtos-Matei A; Dahan S; Qi XF; Crow JP; Cashman NR; Kondejewski LH; Chakrabartty A J Biol Chem; 2002 Dec; 277(49):47551-6. PubMed ID: 12356748 [TBL] [Abstract][Full Text] [Related]
32. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues. Watanabe M; Dykes-Hoberg M; Culotta VC; Price DL; Wong PC; Rothstein JD Neurobiol Dis; 2001 Dec; 8(6):933-41. PubMed ID: 11741389 [TBL] [Abstract][Full Text] [Related]
33. Small-molecule-mediated stabilization of familial amyotrophic lateral sclerosis-linked superoxide dismutase mutants against unfolding and aggregation. Ray SS; Nowak RJ; Brown RH; Lansbury PT Proc Natl Acad Sci U S A; 2005 Mar; 102(10):3639-44. PubMed ID: 15738401 [TBL] [Abstract][Full Text] [Related]
34. Computational insight into in silico analysis and molecular dynamics simulation of the dimer interface residues of ALS-linked hSOD1 forms in apo/holo states: a combined experimental and bioinformatic perspective. Zaji HD; Seyedalipour B; Hanun HM; Baziyar P; Hosseinkhani S; Akhlaghi M 3 Biotech; 2023 Mar; 13(3):92. PubMed ID: 36845075 [TBL] [Abstract][Full Text] [Related]
35. Nonoxidative protein glycation is implicated in familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation. Shibata N; Nagai R; Miyata S; Jono T; Horiuchi S; Hirano A; Kato S; Sasaki S; Asayama K; Kobayashi M Acta Neuropathol; 2000 Sep; 100(3):275-84. PubMed ID: 10965797 [TBL] [Abstract][Full Text] [Related]
36. [Prion-like Properties of Misfolded Cu/Zn-superoxide Dismutase in Amyotrophic Lateral Sclerosis: Update and Perspectives]. Tokuda E; Marklund SL; Furukawa Y Yakugaku Zasshi; 2019; 139(7):1015-1019. PubMed ID: 31257248 [TBL] [Abstract][Full Text] [Related]
37. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations. Jeon GS; Shim YM; Lee DY; Kim JS; Kang M; Ahn SH; Shin JY; Geum D; Hong YH; Sung JJ Mol Neurobiol; 2019 Mar; 56(3):2007-2021. PubMed ID: 29982983 [TBL] [Abstract][Full Text] [Related]
38. Loss of charge mutations in solvent exposed Lys residues of superoxide dismutase 1 do not induce inclusion formation in cultured cell models. Crosby K; Crown AM; Roberts BL; Brown H; Ayers JI; Borchelt DR PLoS One; 2018; 13(11):e0206751. PubMed ID: 30399166 [TBL] [Abstract][Full Text] [Related]
39. Alterations in local stability and dynamics of A4V SOD1 in the presence of trifluoroethanol. Kumar V; Prakash A; Lynn AM Biopolymers; 2018 Mar; 109(3):e23102. PubMed ID: 29369331 [TBL] [Abstract][Full Text] [Related]