These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

167 related articles for article (PubMed ID: 32716967)

  • 1. The CaMKII K42M and K42R mutations are equivalent in suppressing kinase activity and targeting.
    Tullis JE; Rumian NL; Brown CN; Bayer KU
    PLoS One; 2020; 15(7):e0236478. PubMed ID: 32716967
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B.
    Barcomb K; Coultrap SJ; Bayer KU
    Mol Pharmacol; 2013 Dec; 84(6):834-43. PubMed ID: 24056996
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Glu
    Madhavan M; Mohanan AG; Jacob RS; Gunasekaran S; Nair RR; Omkumar RV
    Arch Biochem Biophys; 2020 May; 685():108348. PubMed ID: 32198047
    [TBL] [Abstract][Full Text] [Related]  

  • 4. CaMKII T286 phosphorylation has distinct essential functions in three forms of long-term plasticity.
    Cook SG; Rumian NL; Bayer KU
    J Biol Chem; 2022 Sep; 298(9):102299. PubMed ID: 35872016
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Nucleotides and phosphorylation bi-directionally modulate Ca2+/calmodulin-dependent protein kinase II (CaMKII) binding to the N-methyl-D-aspartate (NMDA) receptor subunit GluN2B.
    O'Leary H; Liu WH; Rorabaugh JM; Coultrap SJ; Bayer KU
    J Biol Chem; 2011 Sep; 286(36):31272-81. PubMed ID: 21768120
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strength.
    Barcomb K; Buard I; Coultrap SJ; Kulbe JR; O'Leary H; Benke TA; Bayer KU
    FASEB J; 2014 Aug; 28(8):3810-9. PubMed ID: 24843070
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Substrate-selective and calcium-independent activation of CaMKII by α-actinin.
    Jalan-Sakrikar N; Bartlett RK; Baucum AJ; Colbran RJ
    J Biol Chem; 2012 May; 287(19):15275-83. PubMed ID: 22427672
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Phosphorylation status of the NR2B subunit of NMDA receptor regulates its interaction with calcium/calmodulin-dependent protein kinase II.
    Raveendran R; Devi Suma Priya S; Mayadevi M; Steephan M; Santhoshkumar TR; Cheriyan J; Sanalkumar R; Pradeep KK; James J; Omkumar RV
    J Neurochem; 2009 Jul; 110(1):92-105. PubMed ID: 19453375
    [TBL] [Abstract][Full Text] [Related]  

  • 9. DAPK1 Mediates LTD by Making CaMKII/GluN2B Binding LTP Specific.
    Goodell DJ; Zaegel V; Coultrap SJ; Hell JW; Bayer KU
    Cell Rep; 2017 Jun; 19(11):2231-2243. PubMed ID: 28614711
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Transition from reversible to persistent binding of CaMKII to postsynaptic sites and NR2B.
    Bayer KU; LeBel E; McDonald GL; O'Leary H; Schulman H; De Koninck P
    J Neurosci; 2006 Jan; 26(4):1164-74. PubMed ID: 16436603
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Stimulating β-adrenergic receptors promotes synaptic potentiation by switching CaMKII movement from LTD to LTP mode.
    Larsen ME; Buonarati OR; Qian H; Hell JW; Bayer KU
    J Biol Chem; 2023 Jun; 299(6):104706. PubMed ID: 37061000
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Ethanol inhibition of recombinant NMDA receptors is not altered by coexpression of CaMKII-alpha or CaMKII-beta.
    Xu M; Chandler LJ; Woodward JJ
    Alcohol; 2008 Aug; 42(5):425-32. PubMed ID: 18562151
    [TBL] [Abstract][Full Text] [Related]  

  • 13. The CaMKII/GluN2B Protein Interaction Maintains Synaptic Strength.
    Barcomb K; Hell JW; Benke TA; Bayer KU
    J Biol Chem; 2016 Jul; 291(31):16082-9. PubMed ID: 27246855
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Dynamic control of CaMKII translocation and localization in hippocampal neurons by NMDA receptor stimulation.
    Shen K; Meyer T
    Science; 1999 Apr; 284(5411):162-6. PubMed ID: 10102820
    [TBL] [Abstract][Full Text] [Related]  

  • 15. CaMKII binding to GluN2B is differentially affected by macromolecular crowding reagents.
    Goodell DJ; Eliseeva TA; Coultrap SJ; Bayer KU
    PLoS One; 2014; 9(5):e96522. PubMed ID: 24796865
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Protection of α-CaMKII from Dephosphorylation by GluN2B Subunit of NMDA Receptor Is Abolished by Mutation of Glu96 or His282 of α-CaMKII.
    Mayadevi M; Lakshmi K; Suma Priya SD; John S; Omkumar RV
    PLoS One; 2016; 11(9):e0162011. PubMed ID: 27610621
    [TBL] [Abstract][Full Text] [Related]  

  • 17. CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac Arrest.
    Buonarati OR; Cook SG; Goodell DJ; Chalmers NE; Rumian NL; Tullis JE; Restrepo S; Coultrap SJ; Quillinan N; Herson PS; Bayer KU
    Cell Rep; 2020 Jan; 30(1):1-8.e4. PubMed ID: 31914378
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Differential stimulus-dependent synaptic recruitment of CaMKIIα by intracellular determinants of GluN2B.
    She K; Rose JK; Craig AM
    Mol Cell Neurosci; 2012 Nov; 51(3-4):68-78. PubMed ID: 22902837
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Multivalent interactions of calcium/calmodulin-dependent protein kinase II with the postsynaptic density proteins NR2B, densin-180, and alpha-actinin-2.
    Robison AJ; Bass MA; Jiao Y; MacMillan LB; Carmody LC; Bartlett RK; Colbran RJ
    J Biol Chem; 2005 Oct; 280(42):35329-36. PubMed ID: 16120608
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Simultaneous Live Imaging of Multiple Endogenous Proteins Reveals a Mechanism for Alzheimer's-Related Plasticity Impairment.
    Cook SG; Goodell DJ; Restrepo S; Arnold DB; Bayer KU
    Cell Rep; 2019 Apr; 27(3):658-665.e4. PubMed ID: 30995464
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.