These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
506 related articles for article (PubMed ID: 33256980)
1. Klotho ameliorates diabetic nephropathy by activating Nrf2 signaling pathway in podocytes. Xing L; Guo H; Meng S; Zhu B; Fang J; Huang J; Chen J; Wang Y; Wang L; Yao X; Wang H Biochem Biophys Res Commun; 2021 Jan; 534():450-456. PubMed ID: 33256980 [TBL] [Abstract][Full Text] [Related]
2. Astragaloside IV protects against podocyte apoptosis by inhibiting oxidative stress via activating PPARγ-Klotho-FoxO1 axis in diabetic nephropathy. Xing L; Fang J; Zhu B; Wang L; Chen J; Wang Y; Huang J; Wang H; Yao X Life Sci; 2021 Mar; 269():119068. PubMed ID: 33476631 [TBL] [Abstract][Full Text] [Related]
3. Perilipin 5 ameliorates high-glucose-induced podocyte injury via Akt/GSK-3β/Nrf2-mediated suppression of apoptosis, oxidative stress, and inflammation. Feng J; Xie L; Yu X; Liu C; Dong H; Lu W; Kong R Biochem Biophys Res Commun; 2021 Mar; 544():22-30. PubMed ID: 33516878 [TBL] [Abstract][Full Text] [Related]
4. Isorhapontigenin ameliorates high glucose-induced podocyte and vascular endothelial cell injuries via mitigating oxidative stress and autophagy through the AMPK/Nrf2 pathway. Tian H; Zheng X; Wang H Int Urol Nephrol; 2023 Feb; 55(2):423-436. PubMed ID: 35960477 [TBL] [Abstract][Full Text] [Related]
5. Protective effects of klotho on palmitate-induced podocyte injury in diabetic nephropathy. Suk Kang J; Son SS; Lee JH; Lee SW; Jeong AR; Lee ES; Cha SK; Chung CH; Lee EY PLoS One; 2021; 16(4):e0250666. PubMed ID: 33891667 [TBL] [Abstract][Full Text] [Related]
6. Klotho inhibits PKCα/p66SHC-mediated podocyte injury in diabetic nephropathy. Jiang W; Xiao T; Han W; Xiong J; He T; Liu Y; Huang Y; Yang K; Bi X; Xu X; Yu Y; Li Y; Gu J; Zhang J; Huang Y; Zhang B; Zhao J Mol Cell Endocrinol; 2019 Aug; 494():110490. PubMed ID: 31207271 [TBL] [Abstract][Full Text] [Related]
7. Klotho plays a protective role against glomerular hypertrophy in a cell cycle-dependent manner in diabetic nephropathy. Oh HJ; Nam BY; Wu M; Kim S; Park J; Kang S; Park JT; Yoo TH; Kang SW; Han SH Am J Physiol Renal Physiol; 2018 Oct; 315(4):F791-F805. PubMed ID: 29638159 [TBL] [Abstract][Full Text] [Related]
8. Sulfiredoxin-1 alleviates high glucose-induced podocyte injury though promoting Nrf2/ARE signaling via inactivation of GSK-3β. Shen Y; Chen S; Zhao Y Biochem Biophys Res Commun; 2019 Sep; 516(4):1137-1144. PubMed ID: 31284950 [TBL] [Abstract][Full Text] [Related]
9. TRIM32 Inhibition Attenuates Apoptosis, Oxidative Stress, and Inflammatory Injury in Podocytes Induced by High Glucose by Modulating the Akt/GSK-3β/Nrf2 Pathway. Chen Z; Tian L; Wang L; Ma X; Lei F; Chen X; Fu R Inflammation; 2022 Jun; 45(3):992-1006. PubMed ID: 34783942 [TBL] [Abstract][Full Text] [Related]
10. RhoA protects the podocytes against high glucose-induced apoptosis through YAP and plays critical role in diabetic nephropathy. Huang Z; Peng Y; Yu H; Yu X; Zhou J; Xiao J Biochem Biophys Res Commun; 2018 Oct; 504(4):949-956. PubMed ID: 30220545 [TBL] [Abstract][Full Text] [Related]
11. Klotho attenuates diabetic nephropathy in db/db mice and ameliorates high glucose-induced injury of human renal glomerular endothelial cells. Wang Q; Ren D; Li Y; Xu G Cell Cycle; 2019; 18(6-7):696-707. PubMed ID: 30784349 [TBL] [Abstract][Full Text] [Related]
12. BRD4 contributes to high-glucose-induced podocyte injury by modulating Keap1/Nrf2/ARE signaling. Zuo H; Wang S; Feng J; Liu X Biochimie; 2019 Oct; 165():100-107. PubMed ID: 31325480 [TBL] [Abstract][Full Text] [Related]
13. Astragaloside IV Attenuates High-Glucose-Induced Impairment in Diabetic Nephropathy by Increasing Klotho Expression via the NF- He J; Cui J; Shi Y; Wang T; Xin J; Li Y; Shan X; Zhu Z; Gao Y J Diabetes Res; 2023; 2023():7423661. PubMed ID: 37261217 [TBL] [Abstract][Full Text] [Related]
14. Down-regulation of SETD6 protects podocyte against high glucose and palmitic acid-induced apoptosis, and mitochondrial dysfunction via activating Nrf2-Keap1 signaling pathway in diabetic nephropathy. Wang X; Liu Q; Kong D; Long Z; Guo Y; Wang S; Liu R; Hai C J Mol Histol; 2020 Oct; 51(5):549-558. PubMed ID: 32803470 [TBL] [Abstract][Full Text] [Related]
15. A novel compound AB38b attenuates oxidative stress and ECM protein accumulation in kidneys of diabetic mice through modulation of Keap1/Nrf2 signaling. Du L; Wang L; Wang B; Wang J; Hao M; Chen YB; Li XZ; Li Y; Jiang YF; Li CC; Yang H; Gu XK; Yin XX; Lu Q Acta Pharmacol Sin; 2020 Mar; 41(3):358-372. PubMed ID: 31645661 [TBL] [Abstract][Full Text] [Related]
16. NFAT2 inhibitor ameliorates diabetic nephropathy and podocyte injury in db/db mice. Zhang L; Li R; Shi W; Liang X; Liu S; Ye Z; Yu C; Chen Y; Zhang B; Wang W; Lai Y; Ma J; Li Z; Tan X Br J Pharmacol; 2013 Sep; 170(2):426-39. PubMed ID: 23826864 [TBL] [Abstract][Full Text] [Related]
18. Solasonine alleviates high glucose-induced podocyte injury through increasing Nrf2-medicated inhibition of NLRP3 activation. Zhang Q; Hu Y; Hu JE; Zhang M Drug Dev Res; 2022 Nov; 83(7):1697-1706. PubMed ID: 36048966 [TBL] [Abstract][Full Text] [Related]
19. CKIP-1 affects the polyubiquitination of Nrf2 and Keap1 via mediating Smurf1 to resist HG-induced renal fibrosis in GMCs and diabetic mice kidneys. Gong W; Chen Z; Zou Y; Zhang L; Huang J; Liu P; Huang H Free Radic Biol Med; 2018 Feb; 115():338-350. PubMed ID: 29248720 [TBL] [Abstract][Full Text] [Related]
20. Activation of Akt-dependent Nrf2/ARE pathway by restoration of Brg-1 remits high glucose-induced oxidative stress and ECM accumulation in podocytes. Yan H; Xu F; Xu J; Song MA; Wang K; Wang L J Biochem Mol Toxicol; 2021 Mar; 35(3):e22672. PubMed ID: 33270355 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]