BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

406 related articles for article (PubMed ID: 33271328)

  • 21. G-CSF attenuates neuroinflammation and neuronal apoptosis via the mTOR/p70SK6 signaling pathway in neonatal Hypoxia-Ischemia rat model.
    Dumbuya JS; Chen L; Shu SY; Ma L; Luo W; Li F; Wu JY; Wang B
    Brain Res; 2020 Jul; 1739():146817. PubMed ID: 32246916
    [TBL] [Abstract][Full Text] [Related]  

  • 22. Alteration in Downstream Hypoxia Gene Signaling in Neonatal Glutathione Peroxidase Overexpressing Mouse Brain after Hypoxia-Ischemia.
    Sheldon RA; Sadjadi R; Lam M; Fitzgerald R; Ferriero DM
    Dev Neurosci; 2015; 37(4-5):398-406. PubMed ID: 25792071
    [TBL] [Abstract][Full Text] [Related]  

  • 23. Dendrimer-mediated delivery of N-acetyl cysteine to microglia in a mouse model of Rett syndrome.
    Nance E; Kambhampati SP; Smith ES; Zhang Z; Zhang F; Singh S; Johnston MV; Kannan RM; Blue ME; Kannan S
    J Neuroinflammation; 2017 Dec; 14(1):252. PubMed ID: 29258545
    [TBL] [Abstract][Full Text] [Related]  

  • 24. Mitochondrial JNK phosphorylation as a novel therapeutic target to inhibit neuroinflammation and apoptosis after neonatal ischemic brain damage.
    Nijboer CH; Bonestroo HJ; Zijlstra J; Kavelaars A; Heijnen CJ
    Neurobiol Dis; 2013 Jun; 54():432-44. PubMed ID: 23376684
    [TBL] [Abstract][Full Text] [Related]  

  • 25. The prolyl hydroxylase inhibitor GSK1120360A reduces early brain injury, but protection is not maintained in a neonatal rat model of hypoxic ischaemic encephalopathy.
    Jones NM; Nathanson AD; Chell S; DeAngelis E; Whelan G; Willé D; Cheng K
    Int J Dev Neurosci; 2022 Aug; 82(5):423-435. PubMed ID: 35662244
    [TBL] [Abstract][Full Text] [Related]  

  • 26. Systemic dendrimer-drug treatment of ischemia-induced neonatal white matter injury.
    Nance E; Porambo M; Zhang F; Mishra MK; Buelow M; Getzenberg R; Johnston M; Kannan RM; Fatemi A; Kannan S
    J Control Release; 2015 Sep; 214():112-20. PubMed ID: 26184052
    [TBL] [Abstract][Full Text] [Related]  

  • 27. Hypothermia modulates myeloid cell polarization in neonatal hypoxic-ischemic brain injury.
    Seitz M; Köster C; Dzietko M; Sabir H; Serdar M; Felderhoff-Müser U; Bendix I; Herz J
    J Neuroinflammation; 2021 Nov; 18(1):266. PubMed ID: 34772426
    [TBL] [Abstract][Full Text] [Related]  

  • 28. Involvement of CXCL1/CXCR2 During Microglia Activation Following Inflammation-Sensitized Hypoxic-Ischemic Brain Injury in Neonatal Rats.
    Serdar M; Kempe K; Herrmann R; Picard D; Remke M; Herz J; Bendix I; Felderhoff-Müser U; Sabir H
    Front Neurol; 2020; 11():540878. PubMed ID: 33123073
    [No Abstract]   [Full Text] [Related]  

  • 29. Sex-related differences in effects of progesterone following neonatal hypoxic brain injury.
    Peterson BL; Won S; Geddes RI; Sayeed I; Stein DG
    Behav Brain Res; 2015 Jun; 286():152-65. PubMed ID: 25746450
    [TBL] [Abstract][Full Text] [Related]  

  • 30. Manipulation of antioxidant pathways in neonatal murine brain.
    Sheldon RA; Jiang X; Francisco C; Christen S; Vexler ZS; Täuber MG; Ferriero DM
    Pediatr Res; 2004 Oct; 56(4):656-62. PubMed ID: 15295091
    [TBL] [Abstract][Full Text] [Related]  

  • 31. Hypoxia-inducible factor: role in cell survival in superoxide dismutase overexpressing mice after neonatal hypoxia-ischemia.
    Jeon GW; Sheldon RA; Ferriero DM
    Korean J Pediatr; 2019 Dec; 62(12):444-449. PubMed ID: 31870086
    [TBL] [Abstract][Full Text] [Related]  

  • 32. Osteopontin Is a Blood Biomarker for Microglial Activation and Brain Injury in Experimental Hypoxic-Ischemic Encephalopathy.
    Li Y; Dammer EB; Zhang-Brotzge X; Chen S; Duong DM; Seyfried NT; Kuan CY; Sun YY
    eNeuro; 2017; 4(1):. PubMed ID: 28101531
    [TBL] [Abstract][Full Text] [Related]  

  • 33. Changes in arginase isoforms in a murine model of neonatal brain hypoxia-ischemia.
    Mike JK; Pathipati P; Sheldon RA; Ferriero DM
    Pediatr Res; 2021 Mar; 89(4):830-837. PubMed ID: 32464635
    [TBL] [Abstract][Full Text] [Related]  

  • 34. Monocytes promote acute neuroinflammation and become pathological microglia in neonatal hypoxic-ischemic brain injury.
    Chen HR; Chen CW; Kuo YM; Chen B; Kuan IS; Huang H; Lee J; Anthony N; Kuan CY; Sun YY
    Theranostics; 2022; 12(2):512-529. PubMed ID: 34976198
    [No Abstract]   [Full Text] [Related]  

  • 35. Hydrogen-rich saline promotes microglia M2 polarization and complement-mediated synapse loss to restore behavioral deficits following hypoxia-ischemic in neonatal mice via AMPK activation.
    Chu X; Cao L; Yu Z; Xin D; Li T; Ma W; Zhou X; Chen W; Liu D; Wang Z
    J Neuroinflammation; 2019 May; 16(1):104. PubMed ID: 31103039
    [TBL] [Abstract][Full Text] [Related]  

  • 36. Early Pro-inflammatory Microglia Activation After Inflammation-Sensitized Hypoxic-Ischemic Brain Injury in Neonatal Rats.
    Serdar M; Kempe K; Rizazad M; Herz J; Bendix I; Felderhoff-Müser U; Sabir H
    Front Cell Neurosci; 2019; 13():237. PubMed ID: 31178702
    [No Abstract]   [Full Text] [Related]  

  • 37. NAAG peptidase inhibitor reduces cellular damage in a model of TBI with secondary hypoxia.
    Feng JF; Gurkoff GG; Van KC; Song M; Lowe DA; Zhou J; Lyeth BG
    Brain Res; 2012 Aug; 1469():144-52. PubMed ID: 22750589
    [TBL] [Abstract][Full Text] [Related]  

  • 38. Peptidylarginine deiminases: novel drug targets for prevention of neuronal damage following hypoxic ischemic insult (HI) in neonates.
    Lange S; Rocha-Ferreira E; Thei L; Mawjee P; Bennett K; Thompson PR; Subramanian V; Nicholas AP; Peebles D; Hristova M; Raivich G
    J Neurochem; 2014 Aug; 130(4):555-62. PubMed ID: 24762056
    [TBL] [Abstract][Full Text] [Related]  

  • 39. Differential Expression and Distinct Roles of Proteinase-Activated Receptor 2 in Microglia and Neurons in Neonatal Mouse Brain After Hypoxia-Ischemic Injury.
    Liu Y; Li H; Hu J; Wu Z; Meng J; Hayashi Y; Nakanishi H; Qing H; Ni J
    Mol Neurobiol; 2022 Jan; 59(1):717-730. PubMed ID: 34762231
    [TBL] [Abstract][Full Text] [Related]  

  • 40. Antioxidant status alters levels of Fas-associated death domain-like IL-1B-converting enzyme inhibitory protein following neonatal hypoxia-ischemia.
    Payton KS; Sheldon RA; Mack DW; Zhu C; Blomgren K; Ferriero DM; Northington FJ
    Dev Neurosci; 2007; 29(4-5):403-11. PubMed ID: 17762208
    [TBL] [Abstract][Full Text] [Related]  

    [Previous]   [Next]    [New Search]
    of 21.