215 related articles for article (PubMed ID: 33468709)
1. A pro-diabetogenic mtDNA polymorphism in the mitochondrial-derived peptide, MOTS-c.
Zempo H; Kim SJ; Fuku N; Nishida Y; Higaki Y; Wan J; Yen K; Miller B; Vicinanza R; Miyamoto-Mikami E; Kumagai H; Naito H; Xiao J; Mehta HH; Lee C; Hara M; Patel YM; Setiawan VW; Moore TM; Hevener AL; Sutoh Y; Shimizu A; Kojima K; Kinoshita K; Arai Y; Hirose N; Maeda S; Tanaka K; Cohen P
Aging (Albany NY); 2021 Jan; 13(2):1692-1717. PubMed ID: 33468709
[TBL] [Abstract][Full Text] [Related]
2. The MOTS-c K14Q polymorphism in the mtDNA is associated with muscle fiber composition and muscular performance.
Kumagai H; Natsume T; Kim SJ; Tobina T; Miyamoto-Mikami E; Shiose K; Ichinoseki-Sekine N; Kakigi R; Tsuzuki T; Miller B; Yen K; Murakami H; Miyachi M; Zempo H; Dobashi S; Machida S; Kobayashi H; Naito H; Cohen P; Fuku N
Biochim Biophys Acta Gen Subj; 2022 Feb; 1866(2):130048. PubMed ID: 34728329
[TBL] [Abstract][Full Text] [Related]
3. The mitochondrial-derived peptide MOTS-c is a regulator of plasma metabolites and enhances insulin sensitivity.
Kim SJ; Miller B; Mehta HH; Xiao J; Wan J; Arpawong TE; Yen K; Cohen P
Physiol Rep; 2019 Jul; 7(13):e14171. PubMed ID: 31293078
[TBL] [Abstract][Full Text] [Related]
4. MOTS-c: A novel mitochondrial-derived peptide regulating muscle and fat metabolism.
Lee C; Kim KH; Cohen P
Free Radic Biol Med; 2016 Nov; 100():182-187. PubMed ID: 27216708
[TBL] [Abstract][Full Text] [Related]
5. Adiponectin treatment improves insulin resistance in mice by regulating the expression of the mitochondrial-derived peptide MOTS-c and its response to exercise via APPL1-SIRT1-PGC-1α.
Guo Q; Chang B; Yu QL; Xu ST; Yi XJ; Cao SC
Diabetologia; 2020 Dec; 63(12):2675-2688. PubMed ID: 32880686
[TBL] [Abstract][Full Text] [Related]
6. MOTS-c interacts synergistically with exercise intervention to regulate PGC-1α expression, attenuate insulin resistance and enhance glucose metabolism in mice via AMPK signaling pathway.
Yang B; Yu Q; Chang B; Guo Q; Xu S; Yi X; Cao S
Biochim Biophys Acta Mol Basis Dis; 2021 Jun; 1867(6):166126. PubMed ID: 33722744
[TBL] [Abstract][Full Text] [Related]
7. The mitochondrial-derived peptide MOTS-c: a player in exceptional longevity?
Fuku N; Pareja-Galeano H; Zempo H; Alis R; Arai Y; Lucia A; Hirose N
Aging Cell; 2015 Dec; 14(6):921-3. PubMed ID: 26289118
[TBL] [Abstract][Full Text] [Related]
8. Plasma mitochondrial derived peptides MOTS-c and SHLP2 positively associate with android and liver fat in people without diabetes.
Sequeira IR; Woodhead JST; Chan A; D'Souza RF; Wan J; Hollingsworth KG; Plank LD; Cohen P; Poppitt SD; Merry TL
Biochim Biophys Acta Gen Subj; 2021 Nov; 1865(11):129991. PubMed ID: 34419510
[TBL] [Abstract][Full Text] [Related]
9. mtDNA Haplogroup N9a Increases the Risk of Type 2 Diabetes by Altering Mitochondrial Function and Intracellular Mitochondrial Signals.
Fang H; Hu N; Zhao Q; Wang B; Zhou H; Fu Q; Shen L; Chen X; Shen F; Lyu J
Diabetes; 2018 Jul; 67(7):1441-1453. PubMed ID: 29735607
[TBL] [Abstract][Full Text] [Related]
10. MOTS-c increases in skeletal muscle following long-term physical activity and improves acute exercise performance after a single dose.
Hyatt JK
Physiol Rep; 2022 Jul; 10(13):e15377. PubMed ID: 35808870
[TBL] [Abstract][Full Text] [Related]
11. MOTS-c and Exercise Restore Cardiac Function by Activating of NRG1-ErbB Signaling in Diabetic Rats.
Li S; Wang M; Ma J; Pang X; Yuan J; Pan Y; Fu Y; Laher I
Front Endocrinol (Lausanne); 2022; 13():812032. PubMed ID: 35370955
[TBL] [Abstract][Full Text] [Related]
12. Mitochondrial DNA association study of type 2 diabetes with or without ischemic stroke in Taiwan.
Loo JH; Trejaut JA; Yen JC; Chen ZS; Ng WM; Huang CY; Hsu KN; Hung KH; Hsiao Y; Wei YH; Lin M
BMC Res Notes; 2014 Apr; 7():223. PubMed ID: 24713204
[TBL] [Abstract][Full Text] [Related]
13. Effects of MOTS-c on the mitochondrial function of cells harboring 3243 A to G mutant mitochondrial DNA.
Ahn CH; Choi EH; Kong BS; Cho YM
Mol Biol Rep; 2020 May; 47(5):4029-4035. PubMed ID: 32279209
[TBL] [Abstract][Full Text] [Related]
14. Association of Omentin rs2274907 and FTO rs9939609 gene polymorphisms with insulin resistance in Iranian individuals with newly diagnosed type 2 diabetes.
Khoshi A; Bajestani MK; Shakeri H; Goodarzi G; Azizi F
Lipids Health Dis; 2019 Jun; 18(1):142. PubMed ID: 31200723
[TBL] [Abstract][Full Text] [Related]
15. MOTS-c reduces myostatin and muscle atrophy signaling.
Kumagai H; Coelho AR; Wan J; Mehta HH; Yen K; Huang A; Zempo H; Fuku N; Maeda S; Oliveira PJ; Cohen P; Kim SJ
Am J Physiol Endocrinol Metab; 2021 Apr; 320(4):E680-E690. PubMed ID: 33554779
[TBL] [Abstract][Full Text] [Related]
16. The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance.
Lee C; Zeng J; Drew BG; Sallam T; Martin-Montalvo A; Wan J; Kim SJ; Mehta H; Hevener AL; de Cabo R; Cohen P
Cell Metab; 2015 Mar; 21(3):443-54. PubMed ID: 25738459
[TBL] [Abstract][Full Text] [Related]
17. Mitochondrial-derived peptides: Antidiabetic functions and evolutionary perspectives.
Kal S; Mahata S; Jati S; Mahata SK
Peptides; 2024 Feb; 172():171147. PubMed ID: 38160808
[TBL] [Abstract][Full Text] [Related]
18. Role of mitochondrial DNA variation in the pathogenesis of diabetes mellitus.
Kwak SH; Park KS
Front Biosci (Landmark Ed); 2016 Jun; 21(6):1151-67. PubMed ID: 27100497
[TBL] [Abstract][Full Text] [Related]
19. MOTS-c peptide regulates adipose homeostasis to prevent ovariectomy-induced metabolic dysfunction.
Lu H; Wei M; Zhai Y; Li Q; Ye Z; Wang L; Luo W; Chen J; Lu Z
J Mol Med (Berl); 2019 Apr; 97(4):473-485. PubMed ID: 30725119
[TBL] [Abstract][Full Text] [Related]
20. Type 2 diabetes risk alleles near BCAR1 and in ANK1 associate with decreased β-cell function whereas risk alleles near ANKRD55 and GRB14 associate with decreased insulin sensitivity in the Danish Inter99 cohort.
Harder MN; Ribel-Madsen R; Justesen JM; Sparsø T; Andersson EA; Grarup N; Jørgensen T; Linneberg A; Hansen T; Pedersen O
J Clin Endocrinol Metab; 2013 Apr; 98(4):E801-6. PubMed ID: 23457408
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]