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5. K27M mutation in histone H3.3 defines clinically and biologically distinct subgroups of pediatric diffuse intrinsic pontine gliomas. Khuong-Quang DA; Buczkowicz P; Rakopoulos P; Liu XY; Fontebasso AM; Bouffet E; Bartels U; Albrecht S; Schwartzentruber J; Letourneau L; Bourgey M; Bourque G; Montpetit A; Bourret G; Lepage P; Fleming A; Lichter P; Kool M; von Deimling A; Sturm D; Korshunov A; Faury D; Jones DT; Majewski J; Pfister SM; Jabado N; Hawkins C Acta Neuropathol; 2012 Sep; 124(3):439-47. PubMed ID: 22661320 [TBL] [Abstract][Full Text] [Related]
6. Transcriptomic and epigenetic profiling of 'diffuse midline gliomas, H3 K27M-mutant' discriminate two subgroups based on the type of histone H3 mutated and not supratentorial or infratentorial location. Castel D; Philippe C; Kergrohen T; Sill M; Merlevede J; Barret E; Puget S; Sainte-Rose C; Kramm CM; Jones C; Varlet P; Pfister SM; Grill J; Jones DTW; Debily MA Acta Neuropathol Commun; 2018 Nov; 6(1):117. PubMed ID: 30396367 [TBL] [Abstract][Full Text] [Related]
7. EZH2 is a potential therapeutic target for H3K27M-mutant pediatric gliomas. Mohammad F; Weissmann S; Leblanc B; Pandey DP; Højfeldt JW; Comet I; Zheng C; Johansen JV; Rapin N; Porse BT; Tvardovskiy A; Jensen ON; Olaciregui NG; Lavarino C; Suñol M; de Torres C; Mora J; Carcaboso AM; Helin K Nat Med; 2017 Apr; 23(4):483-492. PubMed ID: 28263309 [TBL] [Abstract][Full Text] [Related]
8. Histone H3F3A and HIST1H3B K27M mutations define two subgroups of diffuse intrinsic pontine gliomas with different prognosis and phenotypes. Castel D; Philippe C; Calmon R; Le Dret L; Truffaux N; Boddaert N; Pagès M; Taylor KR; Saulnier P; Lacroix L; Mackay A; Jones C; Sainte-Rose C; Blauwblomme T; Andreiuolo F; Puget S; Grill J; Varlet P; Debily MA Acta Neuropathol; 2015 Dec; 130(6):815-27. PubMed ID: 26399631 [TBL] [Abstract][Full Text] [Related]
9. H3 K27M-altered glioma and diffuse intrinsic pontine glioma: Semi-systematic review of treatment landscape and future directions. van den Bent M; Saratsis AM; Geurts M; Franceschi E Neuro Oncol; 2024 May; 26(Supplement_2):S110-S124. PubMed ID: 38102230 [TBL] [Abstract][Full Text] [Related]
10. Potent anti-tumor efficacy of palbociclib in treatment-naïve H3.3K27M-mutant diffuse intrinsic pontine glioma. Sun Y; Sun Y; Yan K; Li Z; Xu C; Geng Y; Pan C; Chen X; Zhang L; Xi Q EBioMedicine; 2019 May; 43():171-179. PubMed ID: 31060906 [TBL] [Abstract][Full Text] [Related]
11. Simultaneous disruption of PRC2 and enhancer function underlies histone H3.3-K27M oncogenic activity in human hindbrain neural stem cells. Brien GL; Bressan RB; Monger C; Gannon D; Lagan E; Doherty AM; Healy E; Neikes H; Fitzpatrick DJ; Deevy O; Grant V; Marqués-Torrejón MA; Alfazema N; Pollard SM; Bracken AP Nat Genet; 2021 Aug; 53(8):1221-1232. PubMed ID: 34294917 [TBL] [Abstract][Full Text] [Related]
12. Histone H3.3 K27M chromatin functions implicate a network of neurodevelopmental factors including ASCL1 and NEUROD1 in DIPG. Lewis NA; Klein RH; Kelly C; Yee J; Knoepfler PS Epigenetics Chromatin; 2022 May; 15(1):18. PubMed ID: 35590427 [TBL] [Abstract][Full Text] [Related]
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19. Use of human embryonic stem cells to model pediatric gliomas with H3.3K27M histone mutation. Funato K; Major T; Lewis PW; Allis CD; Tabar V Science; 2014 Dec; 346(6216):1529-33. PubMed ID: 25525250 [TBL] [Abstract][Full Text] [Related]
20. Pre-Clinical Study of Panobinostat in Xenograft and Genetically Engineered Murine Diffuse Intrinsic Pontine Glioma Models. Hennika T; Hu G; Olaciregui NG; Barton KL; Ehteda A; Chitranjan A; Chang C; Gifford AJ; Tsoli M; Ziegler DS; Carcaboso AM; Becher OJ PLoS One; 2017; 12(1):e0169485. PubMed ID: 28052119 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]