BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

202 related articles for article (PubMed ID: 33556455)

  • 1. Early Hypoexcitability in a Subgroup of Spinal Motoneurons in Superoxide Dismutase 1 Transgenic Mice, a Model of Amyotrophic Lateral Sclerosis.
    Filipchuk A; Pambo-Pambo A; Gaudel F; Liabeuf S; Brocard C; Gueritaud JP; Durand J
    Neuroscience; 2021 May; 463():337-353. PubMed ID: 33556455
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Early excitability changes in lumbar motoneurons of transgenic SOD1G85R and SOD1G(93A-Low) mice.
    Pambo-Pambo A; Durand J; Gueritaud JP
    J Neurophysiol; 2009 Dec; 102(6):3627-42. PubMed ID: 19828728
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Postnatal electrical and morphological abnormalities in lumbar motoneurons from transgenic mouse models of amyotrophic lateral sclerosis.
    Amendola J; Gueritaud JP; Lamotte d'Incamps B; Bories C; Liabeuf S; Allene C; Pambo-Pambo A; Durand J
    Arch Ital Biol; 2007 Nov; 145(3-4):311-23. PubMed ID: 18075124
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Glycine receptor channels in spinal motoneurons are abnormal in a transgenic mouse model of amyotrophic lateral sclerosis.
    Chang Q; Martin LJ
    J Neurosci; 2011 Feb; 31(8):2815-27. PubMed ID: 21414903
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Accumulation of SOD1 mutants in postnatal motoneurons does not cause motoneuron pathology or motoneuron disease.
    Lino MM; Schneider C; Caroni P
    J Neurosci; 2002 Jun; 22(12):4825-32. PubMed ID: 12077179
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Early abnormalities in transgenic mouse models of amyotrophic lateral sclerosis.
    Durand J; Amendola J; Bories C; Lamotte d'Incamps B
    J Physiol Paris; 2006; 99(2-3):211-20. PubMed ID: 16448809
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Altered postnatal maturation of electrical properties in spinal motoneurons in a mouse model of amyotrophic lateral sclerosis.
    Quinlan KA; Schuster JE; Fu R; Siddique T; Heckman CJ
    J Physiol; 2011 May; 589(Pt 9):2245-60. PubMed ID: 21486770
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Early electrophysiological abnormalities in lumbar motoneurons in a transgenic mouse model of amyotrophic lateral sclerosis.
    Bories C; Amendola J; Lamotte d'Incamps B; Durand J
    Eur J Neurosci; 2007 Jan; 25(2):451-9. PubMed ID: 17284186
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Preferential potentiation of AMPA-mediated currents in brainstem hypoglossal motoneurons by subchronic exposure of mice expressing the human superoxide dismutase 1 G93A gene mutation to neurotoxicant methylmercury in vivo.
    Yuan Y; Bailey JM; Rivera-Lopez GM; Atchison WD
    Neurotoxicology; 2024 Jan; 100():72-84. PubMed ID: 38065418
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Preferential motor unit loss in the SOD1 G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Hegedus J; Putman CT; Tyreman N; Gordon T
    J Physiol; 2008 Jul; 586(14):3337-51. PubMed ID: 18467368
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Postnatal dendritic development in lumbar motoneurons in mutant superoxide dismutase 1 mouse model of amyotrophic lateral sclerosis.
    Filipchuk AA; Durand J
    Neuroscience; 2012 May; 209():144-54. PubMed ID: 22387111
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
    Kuo JJ; Schonewille M; Siddique T; Schults AN; Fu R; Bär PR; Anelli R; Heckman CJ; Kroese AB
    J Neurophysiol; 2004 Jan; 91(1):571-5. PubMed ID: 14523070
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
    Li QX; Mok SS; Laughton KM; McLean CA; Volitakis I; Cherny RA; Cheung NS; White AR; Masters CL
    Aging Cell; 2006 Apr; 5(2):153-65. PubMed ID: 16626394
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Stabilization of mutant Cu/Zn superoxide dismutase (SOD1) protein by coexpressed wild SOD1 protein accelerates the disease progression in familial amyotrophic lateral sclerosis mice.
    Fukada K; Nagano S; Satoh M; Tohyama C; Nakanishi T; Shimizu A; Yanagihara T; Sakoda S
    Eur J Neurosci; 2001 Dec; 14(12):2032-6. PubMed ID: 11860498
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Autophagy activation and neuroprotection by progesterone in the G93A-SOD1 transgenic mouse model of amyotrophic lateral sclerosis.
    Kim J; Kim TY; Cho KS; Kim HN; Koh JY
    Neurobiol Dis; 2013 Nov; 59():80-5. PubMed ID: 23891729
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Increase in presynaptic territory of C-terminals on lumbar motoneurons of G93A SOD1 mice during disease progression.
    Pullen AH; Athanasiou D
    Eur J Neurosci; 2009 Feb; 29(3):551-61. PubMed ID: 19187267
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Dysregulation of intracellular copper homeostasis is common to transgenic mice expressing human mutant superoxide dismutase-1s regardless of their copper-binding abilities.
    Tokuda E; Okawa E; Watanabe S; Ono S; Marklund SL
    Neurobiol Dis; 2013 Jun; 54():308-19. PubMed ID: 23321002
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Downregulation of the potassium chloride cotransporter KCC2 in vulnerable motoneurons in the SOD1-G93A mouse model of amyotrophic lateral sclerosis.
    Fuchs A; Ringer C; Bilkei-Gorzo A; Weihe E; Roeper J; Schütz B
    J Neuropathol Exp Neurol; 2010 Oct; 69(10):1057-70. PubMed ID: 20838240
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Motoneuron subtypes show specificity in glycine receptor channel abnormalities in a transgenic mouse model of amyotrophic lateral sclerosis.
    Chang Q; Martin LJ
    Channels (Austin); 2011; 5(4):299-303. PubMed ID: 21558795
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 11.