242 related articles for article (PubMed ID: 33619228)
1. Comprehensive Mutational Analysis of the BRCA1-Associated DNA Helicase and Tumor-Suppressor FANCJ/BACH1/BRIP1.
Calvo JA; Fritchman B; Hernandez D; Persky NS; Johannessen CM; Piccioni F; Kelch BA; Cantor SB
Mol Cancer Res; 2021 Jun; 19(6):1015-1025. PubMed ID: 33619228
[TBL] [Abstract][Full Text] [Related]
2. FANCJ compensates for RAP80 deficiency and suppresses genomic instability induced by interstrand cross-links.
Awate S; Sommers JA; Datta A; Nayak S; Bellani MA; Yang O; Dunn CA; Nicolae CM; Moldovan GL; Seidman MM; Cantor SB; Brosh RM
Nucleic Acids Res; 2020 Sep; 48(16):9161-9180. PubMed ID: 32797166
[TBL] [Abstract][Full Text] [Related]
3. Mutational analysis of the breast cancer susceptibility gene BRIP1 /BACH1/FANCJ in high-risk non-BRCA1/BRCA2 breast cancer families.
Guénard F; Labrie Y; Ouellette G; Joly Beauparlant C; Simard J; ; Durocher F
J Hum Genet; 2008; 53(7):579. PubMed ID: 18414782
[TBL] [Abstract][Full Text] [Related]
4. A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair.
Bharti SK; Sommers JA; Awate S; Bellani MA; Khan I; Bradley L; King GA; Seol Y; Vidhyasagar V; Wu Y; Abe T; Kobayashi K; Shin-Ya K; Kitao H; Wold MS; Branzei D; Neuman KC; Brosh RM
Nucleic Acids Res; 2018 Jul; 46(12):6238-6256. PubMed ID: 29788478
[TBL] [Abstract][Full Text] [Related]
5. Mutational analysis of FANCJ helicase.
Guo M; Vidhyasagar V; Talwar T; Kariem A; Wu Y
Methods; 2016 Oct; 108():118-29. PubMed ID: 27107905
[TBL] [Abstract][Full Text] [Related]
6. DOG-1 is the Caenorhabditis elegans BRIP1/FANCJ homologue and functions in interstrand cross-link repair.
Youds JL; Barber LJ; Ward JD; Collis SJ; O'Neil NJ; Boulton SJ; Rose AM
Mol Cell Biol; 2008 Mar; 28(5):1470-9. PubMed ID: 18086896
[TBL] [Abstract][Full Text] [Related]
7. Hereditary breast cancer and the BRCA1-associated FANCJ/BACH1/BRIP1.
Cantor SB; Guillemette S
Future Oncol; 2011 Feb; 7(2):253-61. PubMed ID: 21345144
[TBL] [Abstract][Full Text] [Related]
8. Rare
Moyer CL; Ivanovich J; Gillespie JL; Doberstein R; Radke MR; Richardson ME; Kaufmann SH; Swisher EM; Goodfellow PJ
Cancer Res; 2020 Feb; 80(4):857-867. PubMed ID: 31822495
[TBL] [Abstract][Full Text] [Related]
9. A novel breast cancer-associated BRIP1 (FANCJ/BACH1) germ-line mutation impairs protein stability and function.
De Nicolo A; Tancredi M; Lombardi G; Flemma CC; Barbuti S; Di Cristofano C; Sobhian B; Bevilacqua G; Drapkin R; Caligo MA
Clin Cancer Res; 2008 Jul; 14(14):4672-80. PubMed ID: 18628483
[TBL] [Abstract][Full Text] [Related]
10. Insight into the roles of helicase motif Ia by characterizing Fanconi anemia group J protein (FANCJ) patient mutations.
Guo M; Vidhyasagar V; Ding H; Wu Y
J Biol Chem; 2014 Apr; 289(15):10551-10565. PubMed ID: 24573678
[TBL] [Abstract][Full Text] [Related]
11. Cancer-associated mutations in the iron-sulfur domain of FANCJ affect G-quadruplex metabolism.
Odermatt DC; Lee WTC; Wild S; Jozwiakowski SK; Rothenberg E; Gari K
PLoS Genet; 2020 Jun; 16(6):e1008740. PubMed ID: 32542039
[TBL] [Abstract][Full Text] [Related]
12. Mutation analysis of BRIP1/BACH1 in BRCA1/BRCA2 negative Chinese women with early onset breast cancer or affected relatives.
Cao AY; Huang J; Hu Z; Li WF; Ma ZL; Tang LL; Zhang B; Su FX; Zhou J; Di GH; Shen KW; Wu J; Lu JS; Luo JM; Yuan WT; Shen ZZ; Huang W; Shao ZM
Breast Cancer Res Treat; 2009 May; 115(1):51-5. PubMed ID: 18483852
[TBL] [Abstract][Full Text] [Related]
13. Landscape of BRIP1 molecular lesions in gastrointestinal cancers from published genomic studies.
Voutsadakis IA
World J Gastroenterol; 2020 Mar; 26(11):1197-1207. PubMed ID: 32231423
[TBL] [Abstract][Full Text] [Related]
14. The BRIP1 helicase functions independently of BRCA1 in the Fanconi anemia pathway for DNA crosslink repair.
Bridge WL; Vandenberg CJ; Franklin RJ; Hiom K
Nat Genet; 2005 Sep; 37(9):953-7. PubMed ID: 16116421
[TBL] [Abstract][Full Text] [Related]
15. Screening for BRCA1, BRCA2, CHEK2, PALB2, BRIP1, RAD50, and CDH1 mutations in high-risk Finnish BRCA1/2-founder mutation-negative breast and/or ovarian cancer individuals.
Kuusisto KM; Bebel A; Vihinen M; Schleutker J; Sallinen SL
Breast Cancer Res; 2011 Feb; 13(1):R20. PubMed ID: 21356067
[TBL] [Abstract][Full Text] [Related]
16. Assessing the link between BACH1/FANCJ and MLH1 in DNA crosslink repair.
Cantor SB; Xie J
Environ Mol Mutagen; 2010 Jul; 51(6):500-7. PubMed ID: 20658644
[TBL] [Abstract][Full Text] [Related]
17. Analysis of BRIP1 Variants among Korean Patients with BRCA1/2 Mutation-Negative High-Risk Breast Cancer.
Kim H; Cho DY; Choi DH; Jung GH; Shin I; Park W; Huh SJ; Nam SJ; Lee JE; Gil WH; Kim SW
Cancer Res Treat; 2016 Jul; 48(3):955-61. PubMed ID: 26790966
[TBL] [Abstract][Full Text] [Related]
18. The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells.
Peng M; Litman R; Xie J; Sharma S; Brosh RM; Cantor SB
EMBO J; 2007 Jul; 26(13):3238-49. PubMed ID: 17581638
[TBL] [Abstract][Full Text] [Related]
19. FANCJ helicase promotes DNA end resection by facilitating CtIP recruitment to DNA double-strand breaks.
Nath S; Nagaraju G
PLoS Genet; 2020 Apr; 16(4):e1008701. PubMed ID: 32251466
[TBL] [Abstract][Full Text] [Related]
20. BRIP1 loss-of-function mutations confer high risk for familial ovarian cancer, but not familial breast cancer.
Weber-Lassalle N; Hauke J; Ramser J; Richters L; Groß E; Blümcke B; Gehrig A; Kahlert AK; Müller CR; Hackmann K; Honisch E; Weber-Lassalle K; Niederacher D; Borde J; Thiele H; Ernst C; Altmüller J; Neidhardt G; Nürnberg P; Klaschik K; Schroeder C; Platzer K; Volk AE; Wang-Gohrke S; Just W; Auber B; Kubisch C; Schmidt G; Horvath J; Wappenschmidt B; Engel C; Arnold N; Dworniczak B; Rhiem K; Meindl A; Schmutzler RK; Hahnen E
Breast Cancer Res; 2018 Jan; 20(1):7. PubMed ID: 29368626
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
