These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
209 related articles for article (PubMed ID: 34405719)
1. LL-37 and HMGB1 induce alveolar damage and reduce lung tissue regeneration via RAGE. Pouwels SD; Hesse L; Wu X; Allam VSRR; van Oldeniel D; Bhiekharie LJ; Phipps S; Oliver BG; Gosens R; Sukkar MB; Heijink IH Am J Physiol Lung Cell Mol Physiol; 2021 Oct; 321(4):L641-L652. PubMed ID: 34405719 [TBL] [Abstract][Full Text] [Related]
2. The receptor for advanced glycation end-products enhances lung epithelial wound repair: An in vitro study. Zhai R; Blondonnet R; Ebrahimi E; Belville C; Audard J; Gross C; Choltus H; Henrioux F; Constantin JM; Pereira B; Blanchon L; Sapin V; Jabaudon M Exp Cell Res; 2020 Jun; 391(2):112030. PubMed ID: 32330509 [TBL] [Abstract][Full Text] [Related]
3. Anti-high-mobility group box-1 (HMGB1) mediates the apoptosis of alveolar epithelial cells (AEC) by receptor of advanced glycation end-products (RAGE)/c-Jun N-terminal kinase (JNK) pathway in the rats of crush injuries. Zhang BF; Song W; Wang J; Wen PF; Zhang YM J Orthop Surg Res; 2022 Jan; 17(1):20. PubMed ID: 35033142 [TBL] [Abstract][Full Text] [Related]
4. The receptor for advanced glycation end products mediates dysfunction of airway epithelial barrier in a lipopolysaccharides-induced murine acute lung injury model. Li J; Wang K; Huang B; Li R; Wang X; Zhang H; Tang H; Chen X Int Immunopharmacol; 2021 Apr; 93():107419. PubMed ID: 33548580 [TBL] [Abstract][Full Text] [Related]
5. Depression of lncRNA NEAT1 Antagonizes LPS-Evoked Acute Injury and Inflammatory Response in Alveolar Epithelial Cells via HMGB1-RAGE Signaling. Zhou H; Wang X; Zhang B Mediators Inflamm; 2020; 2020():8019467. PubMed ID: 32089649 [TBL] [Abstract][Full Text] [Related]
6. Blockade of RAGE ameliorates elastase-induced emphysema development and progression Lee H; Park JR; Kim WJ; Sundar IK; Rahman I; Park SM; Yang SR FASEB J; 2017 May; 31(5):2076-2089. PubMed ID: 28148566 [TBL] [Abstract][Full Text] [Related]
7. Paracrine Regulation of Alveolar Epithelial Damage and Repair Responses by Human Lung-Resident Mesenchymal Stromal Cells. Kruk DMLW; Wisman M; Noordhoek JA; Nizamoglu M; Jonker MR; de Bruin HG; Arevalo Gomez K; Ten Hacken NHT; Pouwels SD; Heijink IH Cells; 2021 Oct; 10(11):. PubMed ID: 34831082 [TBL] [Abstract][Full Text] [Related]
8. High-mobility group box 1 promotes extracellular matrix synthesis and wound repair in human bronchial epithelial cells. Ojo OO; Ryu MH; Jha A; Unruh H; Halayko AJ Am J Physiol Lung Cell Mol Physiol; 2015 Dec; 309(11):L1354-66. PubMed ID: 26432865 [TBL] [Abstract][Full Text] [Related]
9. RAGE inhibition alleviates lipopolysaccharides-induced lung injury via directly suppressing autophagic apoptosis of type II alveolar epithelial cells. Xiong X; Dou J; Shi J; Ren Y; Wang C; Zhang Y; Cui Y Respir Res; 2023 Jan; 24(1):24. PubMed ID: 36691012 [TBL] [Abstract][Full Text] [Related]
10. Emphysema requires the receptor for advanced glycation end-products triggering on structural cells. Waseda K; Miyahara N; Taniguchi A; Kurimoto E; Ikeda G; Koga H; Fujii U; Yamamoto Y; Gelfand EW; Yamamoto H; Tanimoto M; Kanehiro A Am J Respir Cell Mol Biol; 2015 Apr; 52(4):482-91. PubMed ID: 25188021 [TBL] [Abstract][Full Text] [Related]
11. Multi-walled carbon nanotube induces nitrative DNA damage in human lung epithelial cells via HMGB1-RAGE interaction and Toll-like receptor 9 activation. Hiraku Y; Guo F; Ma N; Yamada T; Wang S; Kawanishi S; Murata M Part Fibre Toxicol; 2016 Mar; 13():16. PubMed ID: 27026438 [TBL] [Abstract][Full Text] [Related]
12. Inhibition of the HMGB1/RAGE axis protects against cisplatin-induced ototoxicity via suppression of inflammation and oxidative stress. Qiao X; Li W; Zheng Z; Liu C; Zhao L; He Y; Li H Int J Biol Sci; 2024; 20(2):784-800. PubMed ID: 38169643 [TBL] [Abstract][Full Text] [Related]
13. Receptor for advanced glycation end products expressed on alveolar epithelial cells is the main target for hyperoxia-induced lung injury. Ota C; Ishizawa K; Yamada M; Tando Y; He M; Takahashi T; Yamaya M; Yamamoto Y; Yamamoto H; Kure S; Kubo H Respir Investig; 2016 Mar; 54(2):98-108. PubMed ID: 26879479 [TBL] [Abstract][Full Text] [Related]
14. The myocardial infarct-exacerbating effect of cell-free DNA is mediated by the high-mobility group box 1-receptor for advanced glycation end products-Toll-like receptor 9 pathway. Tian Y; Charles EJ; Yan Z; Wu D; French BA; Kron IL; Yang Z J Thorac Cardiovasc Surg; 2019 Jun; 157(6):2256-2269.e3. PubMed ID: 30401529 [TBL] [Abstract][Full Text] [Related]
15. HMGB1 may act via RAGE to promote angiogenesis in the later phase after intracerebral hemorrhage. Lei C; Zhang S; Cao T; Tao W; Liu M; Wu B Neuroscience; 2015 Jun; 295():39-47. PubMed ID: 25813710 [TBL] [Abstract][Full Text] [Related]
16. Arsenic-induced lung inflammation and fibrosis in a rat model: Contribution of the HMGB1/RAGE, PI3K/AKT, and TGF-β1/SMAD pathways. Wang W; Zheng F; Zhang A Toxicol Appl Pharmacol; 2021 Dec; 432():115757. PubMed ID: 34673086 [TBL] [Abstract][Full Text] [Related]
17. HMGB1 contributes to the irradiation-induced endothelial barrier injury through receptor for advanced glycation endproducts (RAGE). Zhou H; Jin C; Cui L; Xing H; Liu J; Liao W; Liao H; Yu Y J Cell Physiol; 2018 Sep; 233(9):6714-6721. PubMed ID: 29215715 [TBL] [Abstract][Full Text] [Related]
18. The role of the receptor for advanced glycation end-products in lung fibrosis. He M; Kubo H; Ishizawa K; Hegab AE; Yamamoto Y; Yamamoto H; Yamaya M Am J Physiol Lung Cell Mol Physiol; 2007 Dec; 293(6):L1427-36. PubMed ID: 17951314 [TBL] [Abstract][Full Text] [Related]
19. Expression of high-mobility group box 1 and of receptor for advanced glycation end products in chronic obstructive pulmonary disease. Ferhani N; Letuve S; Kozhich A; Thibaudeau O; Grandsaigne M; Maret M; Dombret MC; Sims GP; Kolbeck R; Coyle AJ; Aubier M; Pretolani M Am J Respir Crit Care Med; 2010 May; 181(9):917-27. PubMed ID: 20133931 [TBL] [Abstract][Full Text] [Related]
20. The role of HMGB1/RAGE/TLR4 signaling pathways in cigarette smoke-induced inflammation in chronic obstructive pulmonary disease. Lin L; Li J; Song Q; Cheng W; Chen P Immun Inflamm Dis; 2022 Nov; 10(11):e711. PubMed ID: 36301039 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]