200 related articles for article (PubMed ID: 34504569)
1. Identification of HIF-1α/VEGFA signaling pathway and transcription factors in Kashin-Beck disease by integrated bioinformatics analysis.
Xiao B; Wang G; Huo H; Li W
Exp Ther Med; 2021 Oct; 22(4):1115. PubMed ID: 34504569
[TBL] [Abstract][Full Text] [Related]
2. Identified molecular mechanism of interaction between environmental risk factors and differential expression genes in cartilage of Kashin-Beck disease.
Yu FF; Zhang YX; Zhang LH; Li WR; Guo X; Lammi MJ
Medicine (Baltimore); 2016 Dec; 95(52):e5669. PubMed ID: 28033256
[TBL] [Abstract][Full Text] [Related]
3. Low selenium and T-2 toxin may be involved in the pathogenesis of Kashin-Beck disease by affecting AMPK/mTOR/ULK1 pathway mediated autophagy.
Deng H; Lin X; Xiang R; Bao M; Qiao L; Liu H; He H; Wen X; Han J
Ecotoxicol Environ Saf; 2024 Jul; 279():116503. PubMed ID: 38810288
[TBL] [Abstract][Full Text] [Related]
4. Expression Profiles of Selenium-Related Genes in Human Chondrocytes Exposed to T-2 Toxin and Deoxynivalenol.
Yang L; Zhang J; Li X; Xu C; Wang X; Guo X
Biol Trace Elem Res; 2019 Aug; 190(2):295-302. PubMed ID: 30406490
[TBL] [Abstract][Full Text] [Related]
5. The Importance of Se-Related Genes in the Chondrocyte of Kashin-Beck Disease Revealed by Whole Genomic Microarray and Network Analysis.
Wang S; Zhao G; Shao W; Liu H; Wang W; Wu C; Lammi MJ; Guo X
Biol Trace Elem Res; 2019 Feb; 187(2):367-375. PubMed ID: 29948913
[TBL] [Abstract][Full Text] [Related]
6. WISP1 Is Involved in the Pathogenesis of Kashin-Beck Disease via the Autophagy Pathway.
Li P; Cheng B; Yao Y; Yu W; Liu L; Cheng S; Zhang L; Ma M; Qi X; Liang C; Chu X; Ye J; Sun S; Jia Y; Guo X; Wen Y; Zhang F
Int J Mol Sci; 2023 Nov; 24(22):. PubMed ID: 38003226
[TBL] [Abstract][Full Text] [Related]
7. Comparative Analysis of Differentially Expressed Genes in Chondrocytes from Rats Exposed to Low Selenium and T-2 Toxin.
Wu Y; Gong Y; Liu Y; Chen F; Chen S; Zhang F; Wang C; Li S; Hu M; Huang R; Guo X; Wang X; Ning Y; Yang L
Biol Trace Elem Res; 2024 Mar; 202(3):1020-1030. PubMed ID: 37326932
[TBL] [Abstract][Full Text] [Related]
8. Death of chondrocytes in Kashin-Beck disease: Apoptosis, necrosis or necroptosis?
Zhang Y; He Y; Zhang D; Zhang M; Wang M; Zhang Y; Ma T; Chen J
Int J Exp Pathol; 2018 Dec; 99(6):312-322. PubMed ID: 30680829
[TBL] [Abstract][Full Text] [Related]
9. Pathway-based network analyses and candidate genes associated with Kashin-Beck disease.
Zhang R; Guo H; Yang X; Zhang D; Li B; Li Z; Xiong Y
Medicine (Baltimore); 2019 May; 98(18):e15498. PubMed ID: 31045836
[TBL] [Abstract][Full Text] [Related]
10. The Roles of the Interaction of BCL2-Antagonist/Killer 1, Apoptotic Peptidase Activating Factor 1 and Selenium in the Pathogenesis of Kashin-Beck Disease.
Wang S; Duan C; Zhang F; Wang X; Guo X
Biol Trace Elem Res; 2016 Mar; 170(1):17-24. PubMed ID: 26179084
[TBL] [Abstract][Full Text] [Related]
11. Trans-omics pathway analysis suggests that eQTLs contribute to chondrocyte apoptosis of Kashin-Beck disease through regulating apoptosis pathway expression.
Zhang F; Wen Y; Guo X; Yang T; Shen H; Chen X; Tan L; Tian Q; Deng HW
Gene; 2014 Dec; 553(2):166-9. PubMed ID: 25307768
[TBL] [Abstract][Full Text] [Related]
12. Expression and localization of the small proteoglycans decorin and biglycan in articular cartilage of Kashin-Beck disease and rats induced by T-2 toxin and selenium deficiency.
Wang M; Xue S; Fang Q; Zhang M; He Y; Zhang Y; Lammi MJ; Cao J; Chen J
Glycoconj J; 2019 Dec; 36(6):451-459. PubMed ID: 31478096
[TBL] [Abstract][Full Text] [Related]
13. Proteomic analysis of knee cartilage reveals potential signaling pathways in pathological mechanism of Kashin-Beck disease compared with osteoarthritis.
Lei J; Amhare AF; Wang L; Lv Y; Deng H; Gao H; Guo X; Han J; Lammi MJ
Sci Rep; 2020 Apr; 10(1):6824. PubMed ID: 32322000
[TBL] [Abstract][Full Text] [Related]
14. The integrative analysis of DNA methylation and mRNA expression profiles confirmed the role of selenocompound metabolism pathway in Kashin-Beck disease.
Li P; Ning Y; Wang W; Guo X; Poulet B; Wang X; Wen Y; Han J; Hao J; Liang X; Liu L; Du Y; Cheng B; Cheng S; Zhang L; Ma M; Qi X; Liang C; Wu C; Wang S; Zhao H; Zhao G; Goldring MB; Zhang F; Xu P
Cell Cycle; 2020 Sep; 19(18):2351-2366. PubMed ID: 32816579
[TBL] [Abstract][Full Text] [Related]
15. Expression profiles of genes involved in apoptosis and selenium metabolism in articular cartilage of patients with Kashin-Beck osteoarthritis.
Wu SX; Wang WZ; Zhang F; Wu CY; Dennis BS; Qu CJ; Bai YD; Guo X
Gene; 2014 Feb; 535(2):124-30. PubMed ID: 24316489
[TBL] [Abstract][Full Text] [Related]
16. RNA-seq analysis reveals different gene ontologies and pathways in rheumatoid arthritis and Kashin-Beck disease.
Yu F; Duan C; Zhang X; Yao D; Si G; Gao Y; Gao Z; Umer F; Guo X
Int J Rheum Dis; 2018 Sep; 21(9):1686-1694. PubMed ID: 30256536
[TBL] [Abstract][Full Text] [Related]
17. Mechanism of DNA methylation-mediated downregulation of O6-methylguanine-DNA methyltransferase in cartilage injury of Kashin-Beck disease.
Zhang D; Zhang D; Wang C; Zhang R; Li Q; Xiong Y
Rheumatology (Oxford); 2022 Aug; 61(8):3471-3480. PubMed ID: 34888649
[TBL] [Abstract][Full Text] [Related]
18. Increased Chondrocyte Apoptosis in Kashin-Beck Disease and Rats Induced by T-2 Toxin and Selenium Deficiency.
Yang HJ; Zhang Y; Wang ZL; Xue SH; Li SY; Zhou XR; Zhang M; Fang Q; Wang WJ; Chen C; Deng XH; Chen JH
Biomed Environ Sci; 2017 May; 30(5):351-362. PubMed ID: 28549491
[TBL] [Abstract][Full Text] [Related]
19. The Role of Selenium-Mediated Notch/Hes1 Signaling Pathway in Kashin-Beck Disease Patients and Cartilage Injury Models.
Zhang D; Zhang D; Yang X; Li Q; Zhang R; Xiong Y
Biol Trace Elem Res; 2023 Jun; 201(6):2765-2774. PubMed ID: 36083571
[TBL] [Abstract][Full Text] [Related]
20. Identification of differentially expressed genes and pathways between primary osteoarthritis and endemic osteoarthritis (Kashin-Beck disease).
Zhang F; Guo X; Duan C; Wu S; Yu H; Lammi M
Scand J Rheumatol; 2013; 42(1):71-9. PubMed ID: 23157206
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]