258 related articles for article (PubMed ID: 34618203)
21. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
Mackenzie IR; Bigio EH; Ince PG; Geser F; Neumann M; Cairns NJ; Kwong LK; Forman MS; Ravits J; Stewart H; Eisen A; McClusky L; Kretzschmar HA; Monoranu CM; Highley JR; Kirby J; Siddique T; Shaw PJ; Lee VM; Trojanowski JQ
Ann Neurol; 2007 May; 61(5):427-34. PubMed ID: 17469116
[TBL] [Abstract][Full Text] [Related]
22. Dysregulation of chromatin remodelling complexes in amyotrophic lateral sclerosis.
Tibshirani M; Zhao B; Gentil BJ; Minotti S; Marques C; Keith J; Rogaeva E; Zinman L; Rouaux C; Robertson J; Durham HD
Hum Mol Genet; 2017 Nov; 26(21):4142-4152. PubMed ID: 28973294
[TBL] [Abstract][Full Text] [Related]
23. Pathological and immunoblot analysis of phosphorylated TDP-43 in sporadic amyotrophic lateral sclerosis with pallido-nigro-luysian degeneration.
Uchino A; Ogino M; Takahashi-Fujigasaki J; Oonuma S; Kanazawa N; Kajita S; Ichinoe M; Hasegawa M; Nishiyama K; Murayama S
Neuropathology; 2018 Apr; 38(2):171-178. PubMed ID: 28906030
[TBL] [Abstract][Full Text] [Related]
24. TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations.
Maekawa S; Leigh PN; King A; Jones E; Steele JC; Bodi I; Shaw CE; Hortobagyi T; Al-Sarraj S
Neuropathology; 2009 Dec; 29(6):672-83. PubMed ID: 19496940
[TBL] [Abstract][Full Text] [Related]
25. TDP-43 redistribution is an early event in sporadic amyotrophic lateral sclerosis.
Giordana MT; Piccinini M; Grifoni S; De Marco G; Vercellino M; Magistrello M; Pellerino A; Buccinnà B; Lupino E; Rinaudo MT
Brain Pathol; 2010 Mar; 20(2):351-60. PubMed ID: 19338576
[TBL] [Abstract][Full Text] [Related]
26. Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects.
Mitra J; Guerrero EN; Hegde PM; Liachko NF; Wang H; Vasquez V; Gao J; Pandey A; Taylor JP; Kraemer BC; Wu P; Boldogh I; Garruto RM; Mitra S; Rao KS; Hegde ML
Proc Natl Acad Sci U S A; 2019 Mar; 116(10):4696-4705. PubMed ID: 30770445
[TBL] [Abstract][Full Text] [Related]
27. TDP-43 levels in the brain tissue of ALS cases with and without
Yang Y; Halliday GM; Kiernan MC; Tan RH
Neurology; 2019 Nov; 93(19):e1748-e1755. PubMed ID: 31619481
[TBL] [Abstract][Full Text] [Related]
28. RNA and Protein Interactors with TDP-43 in Human Spinal-Cord Lysates in Amyotrophic Lateral Sclerosis.
Volkening K; Keller BA; Leystra-Lantz C; Strong MJ
J Proteome Res; 2018 Apr; 17(4):1712-1729. PubMed ID: 29513014
[TBL] [Abstract][Full Text] [Related]
29. The long non-coding RNA nuclear-enriched abundant transcript 1_2 induces paraspeckle formation in the motor neuron during the early phase of amyotrophic lateral sclerosis.
Nishimoto Y; Nakagawa S; Hirose T; Okano HJ; Takao M; Shibata S; Suyama S; Kuwako K; Imai T; Murayama S; Suzuki N; Okano H
Mol Brain; 2013 Jul; 6():31. PubMed ID: 23835137
[TBL] [Abstract][Full Text] [Related]
30. Oligogenic inheritance of optineurin (OPTN) and C9ORF72 mutations in ALS highlights localisation of OPTN in the TDP-43-negative inclusions of C9ORF72-ALS.
Bury JJ; Highley JR; Cooper-Knock J; Goodall EF; Higginbottom A; McDermott CJ; Ince PG; Shaw PJ; Kirby J
Neuropathology; 2016 Apr; 36(2):125-34. PubMed ID: 26303227
[TBL] [Abstract][Full Text] [Related]
31. The most prevalent genetic cause of ALS-FTD, C9orf72 synergizes the toxicity of ATXN2 intermediate polyglutamine repeats through the autophagy pathway.
Ciura S; Sellier C; Campanari ML; Charlet-Berguerand N; Kabashi E
Autophagy; 2016 Aug; 12(8):1406-8. PubMed ID: 27245636
[TBL] [Abstract][Full Text] [Related]
32. Single-copy expression of an amyotrophic lateral sclerosis-linked TDP-43 mutation (M337V) in BAC transgenic mice leads to altered stress granule dynamics and progressive motor dysfunction.
Gordon D; Dafinca R; Scaber J; Alegre-Abarrategui J; Farrimond L; Scott C; Biggs D; Kent L; Oliver PL; Davies B; Ansorge O; Wade-Martins R; Talbot K
Neurobiol Dis; 2019 Jan; 121():148-162. PubMed ID: 30290270
[TBL] [Abstract][Full Text] [Related]
33. Matrin 3 Is a Component of Neuronal Cytoplasmic Inclusions of Motor Neurons in Sporadic Amyotrophic Lateral Sclerosis.
Tada M; Doi H; Koyano S; Kubota S; Fukai R; Hashiguchi S; Hayashi N; Kawamoto Y; Kunii M; Tanaka K; Takahashi K; Ogawa Y; Iwata R; Yamanaka S; Takeuchi H; Tanaka F
Am J Pathol; 2018 Feb; 188(2):507-514. PubMed ID: 29128563
[TBL] [Abstract][Full Text] [Related]
34. Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration.
Melamed Z; López-Erauskin J; Baughn MW; Zhang O; Drenner K; Sun Y; Freyermuth F; McMahon MA; Beccari MS; Artates JW; Ohkubo T; Rodriguez M; Lin N; Wu D; Bennett CF; Rigo F; Da Cruz S; Ravits J; Lagier-Tourenne C; Cleveland DW
Nat Neurosci; 2019 Feb; 22(2):180-190. PubMed ID: 30643298
[TBL] [Abstract][Full Text] [Related]
35. Splicing repression is a major function of TDP-43 in motor neurons.
Donde A; Sun M; Ling JP; Braunstein KE; Pang B; Wen X; Cheng X; Chen L; Wong PC
Acta Neuropathol; 2019 Nov; 138(5):813-826. PubMed ID: 31332509
[TBL] [Abstract][Full Text] [Related]
36. Nuclear poly(ADP-ribose) activity is a therapeutic target in amyotrophic lateral sclerosis.
McGurk L; Mojsilovic-Petrovic J; Van Deerlin VM; Shorter J; Kalb RG; Lee VM; Trojanowski JQ; Lee EB; Bonini NM
Acta Neuropathol Commun; 2018 Aug; 6(1):84. PubMed ID: 30157956
[TBL] [Abstract][Full Text] [Related]
37. Protein misdirection inside and outside motor neurons in Amyotrophic Lateral Sclerosis (ALS): a possible clue for therapeutic strategies.
Ido A; Fukuyama H; Urushitani M
Int J Mol Sci; 2011; 12(10):6980-7003. PubMed ID: 22072931
[TBL] [Abstract][Full Text] [Related]
38. Activation of AMP-activated protein kinase α1 mediates mislocalization of TDP-43 in amyotrophic lateral sclerosis.
Liu YJ; Ju TC; Chen HM; Jang YS; Lee LM; Lai HL; Tai HC; Fang JM; Lin YL; Tu PH; Chern Y
Hum Mol Genet; 2015 Feb; 24(3):787-801. PubMed ID: 25256353
[TBL] [Abstract][Full Text] [Related]
39. Targeted depletion of TDP-43 expression in the spinal cord motor neurons leads to the development of amyotrophic lateral sclerosis-like phenotypes in mice.
Wu LS; Cheng WC; Shen CK
J Biol Chem; 2012 Aug; 287(33):27335-44. PubMed ID: 22718760
[TBL] [Abstract][Full Text] [Related]
40. Hu antigen R (HuR) is a positive regulator of the RNA-binding proteins TDP-43 and FUS/TLS: implications for amyotrophic lateral sclerosis.
Lu L; Zheng L; Si Y; Luo W; Dujardin G; Kwan T; Potochick NR; Thompson SR; Schneider DA; King PH
J Biol Chem; 2014 Nov; 289(46):31792-31804. PubMed ID: 25239623
[TBL] [Abstract][Full Text] [Related]
[Previous] [Next] [New Search]
