635 related articles for article (PubMed ID: 34753506)
1. The role of pathological tau in synaptic dysfunction in Alzheimer's diseases.
Wu M; Zhang M; Yin X; Chen K; Hu Z; Zhou Q; Cao X; Chen Z; Liu D
Transl Neurodegener; 2021 Nov; 10(1):45. PubMed ID: 34753506
[TBL] [Abstract][Full Text] [Related]
2. Synaptic Dysfunction in Alzheimer's Disease: Aβ, Tau, and Epigenetic Alterations.
Li K; Wei Q; Liu FF; Hu F; Xie AJ; Zhu LQ; Liu D
Mol Neurobiol; 2018 Apr; 55(4):3021-3032. PubMed ID: 28456942
[TBL] [Abstract][Full Text] [Related]
3. Synaptic Mitochondria: An Early Target of Amyloid-β and Tau in Alzheimer's Disease.
Torres AK; Jara C; Park-Kang HS; Polanco CM; Tapia D; Alarcón F; de la Peña A; Llanquinao J; Vargas-Mardones G; Indo JA; Inestrosa NC; Tapia-Rojas C
J Alzheimers Dis; 2021; 84(4):1391-1414. PubMed ID: 34719499
[TBL] [Abstract][Full Text] [Related]
4. Circulating small extracellular vesicles in Alzheimer's disease: a case-control study of neuro-inflammation and synaptic dysfunction.
Singh R; Rai S; Bharti PS; Zehra S; Gorai PK; Modi GP; Rani N; Dev K; Inampudi KK; Y VV; Chatterjee P; Nikolajeff F; Kumar S
BMC Med; 2024 Jun; 22(1):254. PubMed ID: 38902659
[TBL] [Abstract][Full Text] [Related]
5. The Role of Amyloid-Beta and Tau in the Early Pathogenesis of Alzheimer's Disease.
Yin X; Qiu Y; Zhao C; Zhou Z; Bao J; Qian W
Med Sci Monit; 2021 Sep; 27():e933084. PubMed ID: 34471085
[TBL] [Abstract][Full Text] [Related]
6. Pre-synaptic C-terminal truncated tau is released from cortical synapses in Alzheimer's disease.
Sokolow S; Henkins KM; Bilousova T; Gonzalez B; Vinters HV; Miller CA; Cornwell L; Poon WW; Gylys KH
J Neurochem; 2015 May; 133(3):368-79. PubMed ID: 25393609
[TBL] [Abstract][Full Text] [Related]
7. Abnormal tau, mitochondrial dysfunction, impaired axonal transport of mitochondria, and synaptic deprivation in Alzheimer's disease.
Reddy PH
Brain Res; 2011 Sep; 1415():136-48. PubMed ID: 21872849
[TBL] [Abstract][Full Text] [Related]
8. Amyloid-Beta and Phosphorylated Tau Accumulations Cause Abnormalities at Synapses of Alzheimer's disease Neurons.
Rajmohan R; Reddy PH
J Alzheimers Dis; 2017; 57(4):975-999. PubMed ID: 27567878
[TBL] [Abstract][Full Text] [Related]
9. A role for tau at the synapse in Alzheimer's disease pathogenesis.
Pooler AM; Noble W; Hanger DP
Neuropharmacology; 2014 Jan; 76 Pt A():1-8. PubMed ID: 24076336
[TBL] [Abstract][Full Text] [Related]
10. Abeta exacerbates the neuronal dysfunction caused by human tau expression in a Drosophila model of Alzheimer's disease.
Folwell J; Cowan CM; Ubhi KK; Shiabh H; Newman TA; Shepherd D; Mudher A
Exp Neurol; 2010 Jun; 223(2):401-9. PubMed ID: 19782075
[TBL] [Abstract][Full Text] [Related]
11. Synaptic Impairment in Alzheimer's Disease: A Dysregulated Symphony.
Forner S; Baglietto-Vargas D; Martini AC; Trujillo-Estrada L; LaFerla FM
Trends Neurosci; 2017 Jun; 40(6):347-357. PubMed ID: 28494972
[TBL] [Abstract][Full Text] [Related]
12. Synaptic changes in Alzheimer's disease and its models.
Pozueta J; Lefort R; Shelanski ML
Neuroscience; 2013 Oct; 251():51-65. PubMed ID: 22687952
[TBL] [Abstract][Full Text] [Related]
13. Accumulation of intraneuronal β-amyloid 42 peptides is associated with early changes in microtubule-associated protein 2 in neurites and synapses.
Takahashi RH; Capetillo-Zarate E; Lin MT; Milner TA; Gouras GK
PLoS One; 2013; 8(1):e51965. PubMed ID: 23372648
[TBL] [Abstract][Full Text] [Related]
14. Synaptic tau: A pathological or physiological phenomenon?
Robbins M; Clayton E; Kaminski Schierle GS
Acta Neuropathol Commun; 2021 Sep; 9(1):149. PubMed ID: 34503576
[TBL] [Abstract][Full Text] [Related]
15. The intersection of amyloid β and tau in glutamatergic synaptic dysfunction and collapse in Alzheimer's disease.
Crimins JL; Pooler A; Polydoro M; Luebke JI; Spires-Jones TL
Ageing Res Rev; 2013 Jun; 12(3):757-63. PubMed ID: 23528367
[TBL] [Abstract][Full Text] [Related]
16. Linking amyloid-β and tau: amyloid-β induced synaptic dysfunction via local wreckage of the neuronal cytoskeleton.
Zempel H; Mandelkow EM
Neurodegener Dis; 2012; 10(1-4):64-72. PubMed ID: 22156588
[TBL] [Abstract][Full Text] [Related]
17. The synaptic accumulation of hyperphosphorylated tau oligomers in Alzheimer disease is associated with dysfunction of the ubiquitin-proteasome system.
Tai HC; Serrano-Pozo A; Hashimoto T; Frosch MP; Spires-Jones TL; Hyman BT
Am J Pathol; 2012 Oct; 181(4):1426-35. PubMed ID: 22867711
[TBL] [Abstract][Full Text] [Related]
18. Mechanism of Oxidative Stress and Synapse Dysfunction in the Pathogenesis of Alzheimer's Disease: Understanding the Therapeutics Strategies.
Kamat PK; Kalani A; Rai S; Swarnkar S; Tota S; Nath C; Tyagi N
Mol Neurobiol; 2016 Jan; 53(1):648-661. PubMed ID: 25511446
[TBL] [Abstract][Full Text] [Related]
19. Synapses and dendritic spines as pathogenic targets in Alzheimer's disease.
Yu W; Lu B
Neural Plast; 2012; 2012():247150. PubMed ID: 22474602
[TBL] [Abstract][Full Text] [Related]
20. SPG302 Reverses Synaptic and Cognitive Deficits Without Altering Amyloid or Tau Pathology in a Transgenic Model of Alzheimer's Disease.
Trujillo-Estrada L; Vanderklish PW; Nguyen MMT; Kuang RR; Nguyen C; Huynh E; da Cunha C; Javonillo DI; Forner S; Martini AC; Sarraf ST; Simmon VF; Baglietto-Vargas D; LaFerla FM
Neurotherapeutics; 2021 Oct; 18(4):2468-2483. PubMed ID: 34738197
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
