275 related articles for article (PubMed ID: 34903620)
1. Phase Separation Mediates NUP98 Fusion Oncoprotein Leukemic Transformation.
Chandra B; Michmerhuizen NL; Shirnekhi HK; Tripathi S; Pioso BJ; Baggett DW; Mitrea DM; Iacobucci I; White MR; Chen J; Park CG; Wu H; Pounds S; Medyukhina A; Khairy K; Gao Q; Qu C; Abdelhamed S; Gorman SD; Bawa S; Maslanka C; Kinger S; Dogra P; Ferrolino MC; Di Giacomo D; Mecucci C; Klco JM; Mullighan CG; Kriwacki RW
Cancer Discov; 2022 Apr; 12(4):1152-1169. PubMed ID: 34903620
[TBL] [Abstract][Full Text] [Related]
2. Phase separation drives aberrant chromatin looping and cancer development.
Ahn JH; Davis ES; Daugird TA; Zhao S; Quiroga IY; Uryu H; Li J; Storey AJ; Tsai YH; Keeley DP; Mackintosh SG; Edmondson RD; Byrum SD; Cai L; Tackett AJ; Zheng D; Legant WR; Phanstiel DH; Wang GG
Nature; 2021 Jul; 595(7868):591-595. PubMed ID: 34163069
[TBL] [Abstract][Full Text] [Related]
3. Mechanistic insights into chromatin targeting by leukemic NUP98-PHF23 fusion.
Zhang Y; Guo Y; Gough SM; Zhang J; Vann KR; Li K; Cai L; Shi X; Aplan PD; Wang GG; Kutateladze TG
Nat Commun; 2020 Jul; 11(1):3339. PubMed ID: 32620764
[TBL] [Abstract][Full Text] [Related]
4. NUP98 Fusion Proteins Interact with the NSL and MLL1 Complexes to Drive Leukemogenesis.
Xu H; Valerio DG; Eisold ME; Sinha A; Koche RP; Hu W; Chen CW; Chu SH; Brien GL; Park CY; Hsieh JJ; Ernst P; Armstrong SA
Cancer Cell; 2016 Dec; 30(6):863-878. PubMed ID: 27889185
[TBL] [Abstract][Full Text] [Related]
5. Amino-terminal enhancer of split (AES) interacts with the oncoprotein NUP98-HOXA9 and enhances its transforming ability.
Sarma NJ; Yaseen NR
J Biol Chem; 2011 Nov; 286(45):38989-9001. PubMed ID: 21937451
[TBL] [Abstract][Full Text] [Related]
6. Inhibition of CRM1-mediated nuclear export of transcription factors by leukemogenic NUP98 fusion proteins.
Takeda A; Sarma NJ; Abdul-Nabi AM; Yaseen NR
J Biol Chem; 2010 May; 285(21):16248-57. PubMed ID: 20233715
[TBL] [Abstract][Full Text] [Related]
7. NUP98 fusion in human leukemia: dysregulation of the nuclear pore and homeodomain proteins.
Nakamura T
Int J Hematol; 2005 Jul; 82(1):21-7. PubMed ID: 16105755
[TBL] [Abstract][Full Text] [Related]
8. Trans-repressive effect of NUP98-PMX1 on PMX1-regulated c-FOS gene through recruitment of histone deacetylase 1 by FG repeats.
Bai XT; Gu BW; Yin T; Niu C; Xi XD; Zhang J; Chen Z; Chen SJ
Cancer Res; 2006 May; 66(9):4584-90. PubMed ID: 16651408
[TBL] [Abstract][Full Text] [Related]
9. Dissection of the transformation of primary human hematopoietic cells by the oncogene NUP98-HOXA9.
Yassin ER; Sarma NJ; Abdul-Nabi AM; Dombrowski J; Han Y; Takeda A; Yaseen NR
PLoS One; 2009 Aug; 4(8):e6719. PubMed ID: 19696924
[TBL] [Abstract][Full Text] [Related]
10. Expression of Leukemia-Associated Nup98 Fusion Proteins Generates an Aberrant Nuclear Envelope Phenotype.
Fahrenkrog B; Martinelli V; Nilles N; Fruhmann G; Chatel G; Juge S; Sauder U; Di Giacomo D; Mecucci C; Schwaller J
PLoS One; 2016; 11(3):e0152321. PubMed ID: 27031510
[TBL] [Abstract][Full Text] [Related]
11. The chromosome translocation t(7;11)(p15;p15) in acute myeloid leukemia results in fusion of the NUP98 gene with a HOXA cluster gene, HOXA13, but not HOXA9.
Taketani T; Taki T; Ono R; Kobayashi Y; Ida K; Hayashi Y
Genes Chromosomes Cancer; 2002 Aug; 34(4):437-43. PubMed ID: 12112533
[TBL] [Abstract][Full Text] [Related]
12. Leukemogenic properties of NUP98-PMX1 are linked to NUP98 and homeodomain sequence functions but not to binding properties of PMX1 to serum response factor.
Hirose K; Abramovich C; Argiropoulos B; Humphries RK
Oncogene; 2008 Oct; 27(46):6056-67. PubMed ID: 18604245
[TBL] [Abstract][Full Text] [Related]
13. Dynein Light Chain 1 (DYNLT1) Interacts with Normal and Oncogenic Nucleoporins.
Sarma NJ; Yaseen NR
PLoS One; 2013; 8(6):e67032. PubMed ID: 23840580
[TBL] [Abstract][Full Text] [Related]
14. MLL is essential for NUP98-HOXA9-induced leukemia.
Shima Y; Yumoto M; Katsumoto T; Kitabayashi I
Leukemia; 2017 Oct; 31(10):2200-2210. PubMed ID: 28210005
[TBL] [Abstract][Full Text] [Related]
15. Human models of NUP98-KDM5A megakaryocytic leukemia in mice contribute to uncovering new biomarkers and therapeutic vulnerabilities.
Cardin S; Bilodeau M; Roussy M; Aubert L; Milan T; Jouan L; Rouette A; Laramée L; Gendron P; Duchaine J; Decaluwe H; Spinella JF; Mourad S; Couture F; Sinnett D; Haddad É; Landry JR; Ma J; Humphries RK; Roux PP; Hébert J; Gruber TA; Wilhelm BT; Cellot S
Blood Adv; 2019 Nov; 3(21):3307-3321. PubMed ID: 31698461
[TBL] [Abstract][Full Text] [Related]
16. The Retinoblastoma Tumor Suppressor Is Required for the NUP98-HOXA9-Induced Aberrant Nuclear Envelope Phenotype.
Vaz M; Fahrenkrog B
Cells; 2021 Oct; 10(11):. PubMed ID: 34831074
[TBL] [Abstract][Full Text] [Related]
17. Functional analysis of the NUP98-CCDC28A fusion protein.
Petit A; Ragu C; Soler G; Ottolenghi C; Schluth C; Radford-Weiss I; Schneider-Maunoury S; Callebaut I; Dastugue N; Drabkin HA; Bernard OA; Romana S; Penard-Lacronique V
Haematologica; 2012 Mar; 97(3):379-87. PubMed ID: 22058212
[TBL] [Abstract][Full Text] [Related]
18. The clinical and biological characteristics of NUP98-KDM5A in pediatric acute myeloid leukemia.
Noort S; Wander P; Alonzo TA; Smith J; Ries RE; Gerbing RB; Dolman MEM; Locatelli F; Reinhardt D; Baruchel A; Stary J; Molenaar JJ; Stam RW; van den Heuvel-Eibrink MM; Zwaan MC; Meshinchi S
Haematologica; 2021 Feb; 106(2):630-634. PubMed ID: 32381579
[No Abstract] [Full Text] [Related]
19. [HUP98-HOXA9 transgenic mice are susceptible to N-ethyl-N-nitrosourea stimulation in leukemogenesis].
Lu SY; Gu MM; Wang Y; Tan Y; Sun YP; Wang L; Kong H; Lu ZY; Wang ZG
Zhonghua Xue Ye Xue Za Zhi; 2004 May; 25(5):257-61. PubMed ID: 15182530
[TBL] [Abstract][Full Text] [Related]
20. Biomolecular condensation of NUP98 fusion proteins drives leukemogenic gene expression.
Terlecki-Zaniewicz S; Humer T; Eder T; Schmoellerl J; Heyes E; Manhart G; Kuchynka N; Parapatics K; Liberante FG; Müller AC; Tomazou EM; Grebien F
Nat Struct Mol Biol; 2021 Feb; 28(2):190-201. PubMed ID: 33479542
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
