These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
9. Loss of p16(Ink4a) function rescues cellular senescence induced by telomere dysfunction. Zhang X; Wu X; Tang W; Luo Y Int J Mol Sci; 2012; 13(5):5866-5877. PubMed ID: 22754337 [TBL] [Abstract][Full Text] [Related]
10. [Experimental study of relationship between effect of ginsenoside Rg1 to delay hematopoietic stem cell senescence and expression of p16(INK4a)]. Zhou Y; Yang B; Yao X; Wang Y Zhongguo Zhong Yao Za Zhi; 2011 Mar; 36(5):608-13. PubMed ID: 21657082 [TBL] [Abstract][Full Text] [Related]
11. Transcriptional factor HBP1 targets P16(INK4A), upregulating its expression and consequently is involved in Ras-induced premature senescence. Li H; Wang W; Liu X; Paulson KE; Yee AS; Zhang X Oncogene; 2010 Sep; 29(36):5083-94. PubMed ID: 20581871 [TBL] [Abstract][Full Text] [Related]
12. Intrinsic cooperation between p16INK4a and p21Waf1/Cip1 in the onset of cellular senescence and tumor suppression in vivo. Takeuchi S; Takahashi A; Motoi N; Yoshimoto S; Tajima T; Yamakoshi K; Hirao A; Yanagi S; Fukami K; Ishikawa Y; Sone S; Hara E; Ohtani N Cancer Res; 2010 Nov; 70(22):9381-90. PubMed ID: 21062974 [TBL] [Abstract][Full Text] [Related]
13. Murine mesenchymal cells that express elevated levels of the CDK inhibitor p16(Ink4a) in vivo are not necessarily senescent. Frescas D; Hall BM; Strom E; Virtuoso LP; Gupta M; Gleiberman AS; Rydkina E; Balan V; Vujcic S; Chernova OB; Gudkov AV Cell Cycle; 2017 Aug; 16(16):1526-1533. PubMed ID: 28650766 [TBL] [Abstract][Full Text] [Related]
14. FOXA1 mediates p16(INK4a) activation during cellular senescence. Li Q; Zhang Y; Fu J; Han L; Xue L; Lv C; Wang P; Li G; Tong T EMBO J; 2013 Mar; 32(6):858-73. PubMed ID: 23443045 [TBL] [Abstract][Full Text] [Related]
15. Two mechanisms underlying the loss of p16(Ink4a) function are associated with distinct tumorigenic consequences for WS MEFs escaping from senescence. Wu X; Jia S; Zhang X; Si X; Tang W; Luo Y Mech Ageing Dev; 2012 Aug; 133(8):549-55. PubMed ID: 22813853 [TBL] [Abstract][Full Text] [Related]
16. Aging of mice is associated with p16(Ink4a)- and β-galactosidase-positive macrophage accumulation that can be induced in young mice by senescent cells. Hall BM; Balan V; Gleiberman AS; Strom E; Krasnov P; Virtuoso LP; Rydkina E; Vujcic S; Balan K; Gitlin I; Leonova K; Polinsky A; Chernova OB; Gudkov AV Aging (Albany NY); 2016 Jul; 8(7):1294-315. PubMed ID: 27391570 [TBL] [Abstract][Full Text] [Related]
19. Differential effects of p19(Arf) and p16(Ink4a) loss on senescence of murine bone marrow-derived preB cells and macrophages. Randle DH; Zindy F; Sherr CJ; Roussel MF Proc Natl Acad Sci U S A; 2001 Aug; 98(17):9654-9. PubMed ID: 11481442 [TBL] [Abstract][Full Text] [Related]
20. Real-time in vivo imaging of p16Ink4a reveals cross talk with p53. Yamakoshi K; Takahashi A; Hirota F; Nakayama R; Ishimaru N; Kubo Y; Mann DJ; Ohmura M; Hirao A; Saya H; Arase S; Hayashi Y; Nakao K; Matsumoto M; Ohtani N; Hara E J Cell Biol; 2009 Aug; 186(3):393-407. PubMed ID: 19667129 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]