202 related articles for article (PubMed ID: 35409182)
1. Melatonin-Induced Postconditioning Suppresses NMDA Receptor through Opening of the Mitochondrial Permeability Transition Pore via Melatonin Receptor in Mouse Neurons.
Furuta T; Nakagawa I; Yokoyama S; Morisaki Y; Saito Y; Nakase H
Int J Mol Sci; 2022 Mar; 23(7):. PubMed ID: 35409182
[TBL] [Abstract][Full Text] [Related]
2. Ischemic Postconditioning Reduces NMDA Receptor Currents Through the Opening of the Mitochondrial Permeability Transition Pore and K
Morisaki Y; Nakagawa I; Ogawa Y; Yokoyama S; Furuta T; Saito Y; Nakase H
Cell Mol Neurobiol; 2022 May; 42(4):1079-1089. PubMed ID: 33159622
[TBL] [Abstract][Full Text] [Related]
3. Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice.
Sasaki H; Nakagawa I; Furuta T; Yokoyama S; Morisaki Y; Saito Y; Nakase H
Cell Mol Neurobiol; 2024 Apr; 44(1):32. PubMed ID: 38568450
[TBL] [Abstract][Full Text] [Related]
4. Ischemic postconditioning prevents surge of presynaptic glutamate release by activating mitochondrial ATP-dependent potassium channels in the mouse hippocampus.
Yokoyama S; Nakagawa I; Ogawa Y; Morisaki Y; Motoyama Y; Park YS; Saito Y; Nakase H
PLoS One; 2019; 14(4):e0215104. PubMed ID: 30978206
[TBL] [Abstract][Full Text] [Related]
5. Melatonin improves cryopreservation of ram sperm by inhibiting mitochondrial permeability transition pore opening.
Fang Y; Zhao C; Xiang H; Jia G; Zhong R
Reprod Domest Anim; 2020 Sep; 55(9):1240-1249. PubMed ID: 32649783
[TBL] [Abstract][Full Text] [Related]
6. Anoxia-mediated calcium release through the mitochondrial permeability transition pore silences NMDA receptor currents in turtle neurons.
Hawrysh PJ; Buck LT
J Exp Biol; 2013 Dec; 216(Pt 23):4375-87. PubMed ID: 24259257
[TBL] [Abstract][Full Text] [Related]
7. Alleviation by GABA
Kinjo T; Ashida Y; Higashi H; Sugimura S; Washida M; Niihara H; Ogita K; Yoneda Y; Kuramoto N
Neurochem Res; 2018 Jan; 43(1):79-88. PubMed ID: 28608233
[TBL] [Abstract][Full Text] [Related]
8. PI 3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning.
Bopassa JC; Ferrera R; Gateau-Roesch O; Couture-Lepetit E; Ovize M
Cardiovasc Res; 2006 Jan; 69(1):178-85. PubMed ID: 16216231
[TBL] [Abstract][Full Text] [Related]
9. Melatonin protects against heart ischemia-reperfusion injury by inhibiting mitochondrial permeability transition pore opening.
Petrosillo G; Colantuono G; Moro N; Ruggiero FM; Tiravanti E; Di Venosa N; Fiore T; Paradies G
Am J Physiol Heart Circ Physiol; 2009 Oct; 297(4):H1487-93. PubMed ID: 19684190
[TBL] [Abstract][Full Text] [Related]
10. Postconditioning inhibits mPTP opening independent of oxidative phosphorylation and membrane potential.
Paillard M; Gomez L; Augeul L; Loufouat J; Lesnefsky EJ; Ovize M
J Mol Cell Cardiol; 2009 Jun; 46(6):902-9. PubMed ID: 19254723
[TBL] [Abstract][Full Text] [Related]
11. Postconditioning protects against human endothelial ischaemia-reperfusion injury via subtype-specific KATP channel activation and is mimicked by inhibition of the mitochondrial permeability transition pore.
Okorie MI; Bhavsar DD; Ridout D; Charakida M; Deanfield JE; Loukogeorgakis SP; MacAllister RJ
Eur Heart J; 2011 May; 32(10):1266-74. PubMed ID: 21362704
[TBL] [Abstract][Full Text] [Related]
12. Hydroxysafflor Yellow A Protects Neurons From Excitotoxic Death through Inhibition of NMDARs.
Wang X; Ma Z; Fu Z; Gao S; Yang L; Jin Y; Sun H; Wang C; Fan W; Chen L; Zheng QY; Bi G; Ma CL
ASN Neuro; 2016; 8(2):. PubMed ID: 27067428
[TBL] [Abstract][Full Text] [Related]
13. Direct inhibition of the mitochondrial permeability transition pore: a possible mechanism responsible for anti-apoptotic effects of melatonin.
Andrabi SA; Sayeed I; Siemen D; Wolf G; Horn TF
FASEB J; 2004 May; 18(7):869-71. PubMed ID: 15033929
[TBL] [Abstract][Full Text] [Related]
14. Postconditioning for salvage of ischemic skeletal muscle from reperfusion injury: efficacy and mechanism.
McAllister SE; Ashrafpour H; Cahoon N; Huang N; Moses MA; Neligan PC; Forrest CR; Lipa JE; Pang CY
Am J Physiol Regul Integr Comp Physiol; 2008 Aug; 295(2):R681-9. PubMed ID: 18509099
[TBL] [Abstract][Full Text] [Related]
15. Sevoflurane postconditioning improves spatial learning and memory ability involving mitochondrial permeability transition pore in hemorrhagic shock and resuscitation rats.
Zhang L; Huang L; Wang J; Zhang M; Zhang Y; Hu X
Brain Behav; 2020 Jan; 10(1):e01501. PubMed ID: 31833229
[TBL] [Abstract][Full Text] [Related]
16. Sevoflurane postconditioning improves long-term learning and memory of neonatal hypoxia-ischemia brain damage rats via the PI3K/Akt-mPTP pathway.
Lai Z; Zhang L; Su J; Cai D; Xu Q
Brain Res; 2016 Jan; 1630():25-37. PubMed ID: 26541582
[TBL] [Abstract][Full Text] [Related]
17. Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion.
Gomez L; Paillard M; Thibault H; Derumeaux G; Ovize M
Circulation; 2008 May; 117(21):2761-8. PubMed ID: 18490522
[TBL] [Abstract][Full Text] [Related]
18. Melatonin protects cardiac microvasculature against ischemia/reperfusion injury via suppression of mitochondrial fission-VDAC1-HK2-mPTP-mitophagy axis.
Zhou H; Zhang Y; Hu S; Shi C; Zhu P; Ma Q; Jin Q; Cao F; Tian F; Chen Y
J Pineal Res; 2017 Aug; 63(1):. PubMed ID: 28398674
[TBL] [Abstract][Full Text] [Related]
19. Inhibition of mitochondrial permeability transition pore opening contributes to the neuroprotective effects of ischemic postconditioning in rats.
Sun J; Luan Q; Dong H; Song W; Xie K; Hou L; Xiong L
Brain Res; 2012 Feb; 1436():101-10. PubMed ID: 22197697
[TBL] [Abstract][Full Text] [Related]
20. The Melatonin Receptor Agonist Ramelteon Induces Cardioprotection that Requires MT2 Receptor Activation and Release of Reactive Oxygen Species.
Stroethoff M; Goetze L; Torregroza C; Bunte S; Raupach A; Heinen A; Mathes A; Hollmann MW; Huhn R
Cardiovasc Drugs Ther; 2020 Jun; 34(3):303-310. PubMed ID: 32236860
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]