BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

171 related articles for article (PubMed ID: 35604590)

  • 1. Alternative end-joining originates stable chromosome aberrations induced by etoposide during targeted inhibition of DNA-PKcs in ATM-deficient tumor cells.
    de Campos Nebel M; Palmitelli M; Pérez Maturo J; González-Cid M
    Chromosome Res; 2022 Dec; 30(4):459-476. PubMed ID: 35604590
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Progression of chromosomal damage induced by etoposide in G2 phase in a DNA-PKcs-deficient context.
    Palmitelli M; de Campos-Nebel M; González-Cid M
    Chromosome Res; 2015 Dec; 23(4):719-32. PubMed ID: 26152239
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Genetic interaction between DNA repair factors PAXX, XLF, XRCC4 and DNA-PKcs in human cells.
    Xing M; Oksenych V
    FEBS Open Bio; 2019 Jul; 9(7):1315-1326. PubMed ID: 31141305
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Non-homologous end joining is the responsible pathway for the repair of fludarabine-induced DNA double strand breaks in mammalian cells.
    de Campos-Nebel M; Larripa I; González-Cid M
    Mutat Res; 2008 Nov; 646(1-2):8-16. PubMed ID: 18812179
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The catalytic subunit of DNA-dependent protein kinase is required for cellular resistance to oxidative stress independent of DNA double-strand break repair.
    Li M; Lin YF; Palchik GA; Matsunaga S; Wang D; Chen BP
    Free Radic Biol Med; 2014 Nov; 76():278-85. PubMed ID: 25224041
    [TBL] [Abstract][Full Text] [Related]  

  • 6. DNA-PKcs and ATM co-regulate DNA double-strand break repair.
    Shrivastav M; Miller CA; De Haro LP; Durant ST; Chen BP; Chen DJ; Nickoloff JA
    DNA Repair (Amst); 2009 Aug; 8(8):920-9. PubMed ID: 19535303
    [TBL] [Abstract][Full Text] [Related]  

  • 7. ATM antagonizes NHEJ proteins assembly and DNA-ends synapsis at single-ended DNA double strand breaks.
    Britton S; Chanut P; Delteil C; Barboule N; Frit P; Calsou P
    Nucleic Acids Res; 2020 Sep; 48(17):9710-9723. PubMed ID: 32890395
    [TBL] [Abstract][Full Text] [Related]  

  • 8. DNA-PKcs-dependent NHEJ pathway supports the progression of topoisomerase II poison-induced chromosome aberrant cells.
    Elguero ME; de Campos-Nebel M; González-Cid M
    Environ Mol Mutagen; 2012 Oct; 53(8):608-18. PubMed ID: 22987276
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Correct end use during end joining of multiple chromosomal double strand breaks is influenced by repair protein RAD50, DNA-dependent protein kinase DNA-PKcs, and transcription context.
    Gunn A; Bennardo N; Cheng A; Stark JM
    J Biol Chem; 2011 Dec; 286(49):42470-42482. PubMed ID: 22027841
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Topoisomerase II-mediated DNA damage is differently repaired during the cell cycle by non-homologous end joining and homologous recombination.
    de Campos-Nebel M; Larripa I; González-Cid M
    PLoS One; 2010 Sep; 5(9):. PubMed ID: 20824055
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Repair-independent functions of DNA-PKcs protect irradiated cells from mitotic slippage and accelerated senescence.
    Liu Y; Efimova EV; Ramamurthy A; Kron SJ
    J Cell Sci; 2019 Jul; 132(13):. PubMed ID: 31189537
    [TBL] [Abstract][Full Text] [Related]  

  • 12. DNA-Dependent Protein Kinase Catalytic Subunit: The Sensor for DNA Double-Strand Breaks Structurally and Functionally Related to Ataxia Telangiectasia Mutated.
    Matsumoto Y; Asa ADDC; Modak C; Shimada M
    Genes (Basel); 2021 Jul; 12(8):. PubMed ID: 34440313
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Therapeutic targeting of a robust non-oncogene addiction to PRKDC in ATM-defective tumors.
    Riabinska A; Daheim M; Herter-Sprie GS; Winkler J; Fritz C; Hallek M; Thomas RK; Kreuzer KA; Frenzel LP; Monfared P; Martins-Boucas J; Chen S; Reinhardt HC
    Sci Transl Med; 2013 Jun; 5(189):189ra78. PubMed ID: 23761041
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Regulation of the DNA Damage Response by DNA-PKcs Inhibitory Phosphorylation of ATM.
    Zhou Y; Lee JH; Jiang W; Crowe JL; Zha S; Paull TT
    Mol Cell; 2017 Jan; 65(1):91-104. PubMed ID: 27939942
    [TBL] [Abstract][Full Text] [Related]  

  • 15. ATM phosphorylates the FATC domain of DNA-PKcs at threonine 4102 to promote non-homologous end joining.
    Lu H; Zhang Q; Laverty DJ; Puncheon AC; Augustine MM; Williams GJ; Nagel ZD; Chen BPC; Davis AJ
    Nucleic Acids Res; 2023 Jul; 51(13):6770-6783. PubMed ID: 37309889
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Deciphering the role of distinct DNA-PK phosphorylations at collapsed replication forks.
    Neal JA; Dunger K; Geith K; Meek K
    DNA Repair (Amst); 2020 Oct; 94():102925. PubMed ID: 32674014
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Inhibiting DNA-PKcs in a non-homologous end-joining pathway in response to DNA double-strand breaks.
    Dong J; Zhang T; Ren Y; Wang Z; Ling CC; He F; Li GC; Wang C; Wen B
    Oncotarget; 2017 Apr; 8(14):22662-22673. PubMed ID: 28186989
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Mechanisms of DNA double strand break repair and chromosome aberration formation.
    Iliakis G; Wang H; Perrault AR; Boecker W; Rosidi B; Windhofer F; Wu W; Guan J; Terzoudi G; Pantelias G
    Cytogenet Genome Res; 2004; 104(1-4):14-20. PubMed ID: 15162010
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Differential phosphorylation of DNA-PKcs regulates the interplay between end-processing and end-ligation during nonhomologous end-joining.
    Jiang W; Crowe JL; Liu X; Nakajima S; Wang Y; Li C; Lee BJ; Dubois RL; Liu C; Yu X; Lan L; Zha S
    Mol Cell; 2015 Apr; 58(1):172-85. PubMed ID: 25818648
    [TBL] [Abstract][Full Text] [Related]  

  • 20. DNA repair kinetics in SCID mice Sertoli cells and DNA-PKcs-deficient mouse embryonic fibroblasts.
    Ahmed EA; Vélaz E; Rosemann M; Gilbertz KP; Scherthan H
    Chromosoma; 2017 Mar; 126(2):287-298. PubMed ID: 27136939
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.