263 related articles for article (PubMed ID: 35766275)
1. Inhibiting Succinate Release Worsens Cardiac Reperfusion Injury by Enhancing Mitochondrial Reactive Oxygen Species Generation.
Milliken AS; Nadtochiy SM; Brookes PS
J Am Heart Assoc; 2022 Jul; 11(13):e026135. PubMed ID: 35766275
[TBL] [Abstract][Full Text] [Related]
2. The role of succinate and ROS in reperfusion injury - A critical appraisal.
Andrienko TN; Pasdois P; Pereira GC; Ovens MJ; Halestrap AP
J Mol Cell Cardiol; 2017 Sep; 110():1-14. PubMed ID: 28689004
[TBL] [Abstract][Full Text] [Related]
3. Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.
Chouchani ET; Pell VR; Gaude E; Aksentijević D; Sundier SY; Robb EL; Logan A; Nadtochiy SM; Ord ENJ; Smith AC; Eyassu F; Shirley R; Hu CH; Dare AJ; James AM; Rogatti S; Hartley RC; Eaton S; Costa ASH; Brookes PS; Davidson SM; Duchen MR; Saeb-Parsy K; Shattock MJ; Robinson AJ; Work LM; Frezza C; Krieg T; Murphy MP
Nature; 2014 Nov; 515(7527):431-435. PubMed ID: 25383517
[TBL] [Abstract][Full Text] [Related]
4. Inhibition of pyruvate dehydrogenase kinase 4 ameliorates kidney ischemia-reperfusion injury by reducing succinate accumulation during ischemia and preserving mitochondrial function during reperfusion.
Oh CJ; Kim MJ; Lee JM; Kim DH; Kim IY; Park S; Kim Y; Lee KB; Lee SH; Lim CW; Kim M; Lee JY; Pagire HS; Pagire SH; Bae MA; Chanda D; Thoudam T; Khang AR; Harris RA; Ahn JH; Jeon JH; Lee IK
Kidney Int; 2023 Oct; 104(4):724-739. PubMed ID: 37399974
[TBL] [Abstract][Full Text] [Related]
5. Reactive oxygen species production induced by pore opening in cardiac mitochondria: The role of complex II.
Korge P; John SA; Calmettes G; Weiss JN
J Biol Chem; 2017 Jun; 292(24):9896-9905. PubMed ID: 28450394
[TBL] [Abstract][Full Text] [Related]
6. Increased Succinate Accumulation Induces ROS Generation in
Kamarauskaite J; Baniene R; Trumbeckas D; Strazdauskas A; Trumbeckaite S
Biomed Res Int; 2020; 2020():8855585. PubMed ID: 33102598
[TBL] [Abstract][Full Text] [Related]
7. The cardioprotectant 3',4'-dihydroxyflavonol inhibits opening of the mitochondrial permeability transition pore after myocardial ischemia and reperfusion in rats.
Woodman OL; Long R; Pons S; Eychenne N; Berdeaux A; Morin D
Pharmacol Res; 2014 Mar; 81():26-33. PubMed ID: 24521796
[TBL] [Abstract][Full Text] [Related]
8. Ischemic preconditioning protects against cardiac ischemia reperfusion injury without affecting succinate accumulation or oxidation.
Pell VR; Spiroski AM; Mulvey J; Burger N; Costa ASH; Logan A; Gruszczyk AV; Rosa T; James AM; Frezza C; Murphy MP; Krieg T
J Mol Cell Cardiol; 2018 Oct; 123():88-91. PubMed ID: 30118790
[TBL] [Abstract][Full Text] [Related]
9. Aldose reductase mediates myocardial ischemia-reperfusion injury in part by opening mitochondrial permeability transition pore.
Ananthakrishnan R; Kaneko M; Hwang YC; Quadri N; Gomez T; Li Q; Caspersen C; Ramasamy R
Am J Physiol Heart Circ Physiol; 2009 Feb; 296(2):H333-41. PubMed ID: 19060123
[TBL] [Abstract][Full Text] [Related]
10. Elucidating Mitochondrial Electron Transport Chain Supercomplexes in the Heart During Ischemia-Reperfusion.
Jang S; Lewis TS; Powers C; Khuchua Z; Baines CP; Wipf P; Javadov S
Antioxid Redox Signal; 2017 Jul; 27(1):57-69. PubMed ID: 27604998
[TBL] [Abstract][Full Text] [Related]
11. Cyclosporine A normalizes mitochondrial coupling, reactive oxygen species production, and inflammation and partially restores skeletal muscle maximal oxidative capacity in experimental aortic cross-clamping.
Pottecher J; Guillot M; Belaidi E; Charles AL; Lejay A; Gharib A; Diemunsch P; Geny B
J Vasc Surg; 2013 Apr; 57(4):1100-1108.e2. PubMed ID: 23332985
[TBL] [Abstract][Full Text] [Related]
12. Succinate dehydrogenase inhibition with malonate during reperfusion reduces infarct size by preventing mitochondrial permeability transition.
Valls-Lacalle L; Barba I; Miró-Casas E; Alburquerque-Béjar JJ; Ruiz-Meana M; Fuertes-Agudo M; Rodríguez-Sinovas A; García-Dorado D
Cardiovasc Res; 2016 Mar; 109(3):374-84. PubMed ID: 26705364
[TBL] [Abstract][Full Text] [Related]
13. Interplay between Ca2+ cycling and mitochondrial permeability transition pores promotes reperfusion-induced injury of cardiac myocytes.
Abdallah Y; Kasseckert SA; Iraqi W; Said M; Shahzad T; Erdogan A; Neuhof C; Gündüz D; Schlüter KD; Tillmanns H; Piper HM; Reusch HP; Ladilov Y
J Cell Mol Med; 2011 Nov; 15(11):2478-85. PubMed ID: 21199327
[TBL] [Abstract][Full Text] [Related]
14. Real-Time Fluorescence Measurements of ROS and [Ca2+] in Ischemic / Reperfused Rat Hearts: Detectable Increases Occur only after Mitochondrial Pore Opening and Are Attenuated by Ischemic Preconditioning.
Andrienko T; Pasdois P; Rossbach A; Halestrap AP
PLoS One; 2016; 11(12):e0167300. PubMed ID: 27907091
[TBL] [Abstract][Full Text] [Related]
15. Acid enhancement of ROS generation by complex-I reverse electron transport is balanced by acid inhibition of complex-II: Relevance for tissue reperfusion injury.
Milliken AS; Kulkarni CA; Brookes PS
Redox Biol; 2020 Oct; 37():101733. PubMed ID: 33007502
[TBL] [Abstract][Full Text] [Related]
16. The role of mitochondria in protection of the heart by preconditioning.
Halestrap AP; Clarke SJ; Khaliulin I
Biochim Biophys Acta; 2007 Aug; 1767(8):1007-31. PubMed ID: 17631856
[TBL] [Abstract][Full Text] [Related]
17. Propofol Prevents Oxidative Stress by Decreasing the Ischemic Accumulation of Succinate in Focal Cerebral Ischemia-Reperfusion Injury.
Yu W; Gao D; Jin W; Liu S; Qi S
Neurochem Res; 2018 Feb; 43(2):420-429. PubMed ID: 29168092
[TBL] [Abstract][Full Text] [Related]
18. Mff-Dependent Mitochondrial Fission Contributes to the Pathogenesis of Cardiac Microvasculature Ischemia/Reperfusion Injury via Induction of mROS-Mediated Cardiolipin Oxidation and HK2/VDAC1 Disassociation-Involved mPTP Opening.
Zhou H; Hu S; Jin Q; Shi C; Zhang Y; Zhu P; Ma Q; Tian F; Chen Y
J Am Heart Assoc; 2017 Mar; 6(3):. PubMed ID: 28288978
[TBL] [Abstract][Full Text] [Related]
19. A model of mitochondrial superoxide production during ischaemia-reperfusion injury for therapeutic development and mechanistic understanding.
Sorby-Adams A; Prime TA; Miljkovic JL; Prag HA; Krieg T; Murphy MP
Redox Biol; 2024 Jun; 72():103161. PubMed ID: 38677214
[TBL] [Abstract][Full Text] [Related]
20. ROS-mediated PARP activity undermines mitochondrial function after permeability transition pore opening during myocardial ischemia-reperfusion.
Schriewer JM; Peek CB; Bass J; Schumacker PT
J Am Heart Assoc; 2013 Apr; 2(2):e000159. PubMed ID: 23598272
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
